(Q24302381)

Mixed lineage kinase domain like pseudokinase

ChEBI release 2020-05-28

ChEBI ID

63770

1 reference

Receptor interacting serine/threonine kinase 3

1 reference

Receptor interacting serine/threonine kinase 1

1 reference

Tumor necrosis factor

1 reference

necroptotic signaling pathway

1 reference

Receptor-interacting serine-threonine kinase 3

1 reference

Mixed lineage kinase domain-like

1 reference

author name string

Liming Sun

1

0 references

Huayi Wang

2

0 references

Zhigao Wang

3

0 references

Sudan He

4

0 references

She Chen

5

0 references

Daohong Liao

6

0 references

Lai Wang

7

0 references

Jiacong Yan

8

0 references

Weilong Liu

9

0 references

Xiaoguang Lei

10

0 references

Xiaodong Wang

Receptor interacting protein kinase-3 determines cellular necrotic response to TNF-alpha

11

0 references

language of work or name

English

0 references

publication date

20 January 2012

0 references

published in

Cell

0 references

volume

148

0 references

issue

1-2

0 references

page(s)

213-27

0 references

cites work

1 reference

https://api.crossref.org/works/10.1016%2FJ.CELL.2011.11.031

7 January 2021

Phosphorylation-driven assembly of the RIP1-RIP3 complex regulates programmed necrosis and virus-induced inflammation

1 reference

https://api.crossref.org/works/10.1016%2FJ.CELL.2011.11.031

7 January 2021

RIP kinases at the crossroads of cell death and survival

1 reference

https://api.crossref.org/works/10.1016%2FJ.CELL.2011.11.031

7 January 2021

The mitochondrial phosphatase PGAM5 functions at the convergence point of multiple necrotic death pathways

1 reference

https://api.crossref.org/works/10.1016%2FJ.CELL.2011.11.031

7 January 2021

Cleavage of RIP3 inactivates its caspase-independent apoptosis pathway by removal of kinase domain.

1 reference

https://api.crossref.org/works/10.1016%2FJ.CELL.2011.11.031

7 January 2021

Virus inhibition of RIP3-dependent necrosis

1 reference

https://api.crossref.org/works/10.1016%2FJ.CELL.2011.11.031

7 January 2021

Smac, a mitochondrial protein that promotes cytochrome c-dependent caspase activation by eliminating IAP inhibition

1 reference

https://api.crossref.org/works/10.1016%2FJ.CELL.2011.11.031

7 January 2021

Identification of DIABLO, a mammalian protein that promotes apoptosis by binding to and antagonizing IAP proteins

1 reference

https://api.crossref.org/works/10.1016%2FJ.CELL.2011.11.031

7 January 2021

Cytochrome c and dATP-dependent formation of Apaf-1/caspase-9 complex initiates an apoptotic protease cascade

1 reference

https://api.crossref.org/works/10.1016%2FJ.CELL.2011.11.031

7 January 2021

Apoptosis control by death and decoy receptors

1 reference

https://api.crossref.org/works/10.1016%2FJ.CELL.2011.11.031

7 January 2021

Polyproline-rod approach to isolating protein targets of bioactive small molecules: isolation of a new target of indomethacin

1 reference

https://api.crossref.org/works/10.1016%2FJ.CELL.2011.11.031

7 January 2021

Apoptosis: A Basic Biological Phenomenon with Wideranging Implications in Tissue Kinetics

1 reference

https://api.crossref.org/works/10.1016%2FJ.CELL.2011.11.031

7 January 2021

Catalytic activity of the caspase-8-FLIP(L) complex inhibits RIPK3-dependent necrosis

1 reference

https://api.crossref.org/works/10.1016%2FJ.CELL.2011.11.031

7 January 2021

RIP3 mediates the embryonic lethality of caspase-8-deficient mice

1 reference

https://api.crossref.org/works/10.1016%2FJ.CELL.2011.11.031

7 January 2021

Chemical inhibitor of nonapoptotic cell death with therapeutic potential for ischemic brain injury

1 reference

https://api.crossref.org/works/10.1016%2FJ.CELL.2011.11.031

7 January 2021

Identification of RIP1 kinase as a specific cellular target of necrostatins

1 reference

https://api.crossref.org/works/10.1016%2FJ.CELL.2011.11.031

7 January 2021

Caspase inhibition causes hyperacute tumor necrosis factor-induced shock via oxidative stress and phospholipase A2.

1 reference

https://api.crossref.org/works/10.1016%2FJ.CELL.2011.11.031

7 January 2021

Caspase-independent cell death

1 reference

https://api.crossref.org/works/10.1016%2FJ.CELL.2011.11.031

7 January 2021

Mixed-lineage kinase control of JNK and p38 MAPK pathways

1 reference

https://api.crossref.org/works/10.1016%2FJ.CELL.2011.11.031

7 January 2021

Receptor interacting protein kinases mediate retinal detachment-induced photoreceptor necrosis and compensate for inhibition of apoptosis

1 reference

https://api.crossref.org/works/10.1016%2FJ.CELL.2011.11.031

7 January 2021

An APAF-1.cytochrome c multimeric complex is a functional apoptosome that activates procaspase-9

1 reference

https://api.crossref.org/works/10.1016%2FJ.CELL.2011.11.031

7 January 2021

Smac/DIABLO selectively reduces the levels of c-IAP1 and c-IAP2 but not that of XIAP and livin in HeLa cells

1 reference

https://api.crossref.org/works/10.1016%2FJ.CELL.2011.11.031

7 January 2021

Inhibition of caspases increases the sensitivity of L929 cells to necrosis mediated by tumor necrosis factor.

1 reference

https://api.crossref.org/works/10.1016%2FJ.CELL.2011.11.031

7 January 2021

Two CD95 (APO-1/Fas) signaling pathways

1 reference

https://api.crossref.org/works/10.1016%2FJ.CELL.2011.11.031

7 January 2021

Cleavage of the death domain kinase RIP by caspase-8 prompts TNF-induced apoptosis

1 reference

https://api.crossref.org/works/10.1016%2FJ.CELL.2011.11.031

7 January 2021

A small molecule Smac mimic potentiates TRAIL- and TNFalpha-mediated cell death

1 reference

https://api.crossref.org/works/10.1016%2FJ.CELL.2011.11.031

7 January 2021

RIP3, an energy metabolism regulator that switches TNF-induced cell death from apoptosis to necrosis

1 reference

https://api.crossref.org/works/10.1016%2FJ.CELL.2011.11.031

7 January 2021

Caspases: enemies within

1 reference

https://api.crossref.org/works/10.1016%2FJ.CELL.2011.11.031

7 January 2021

Mechanisms and Functions of Cell Death

1 reference

https://api.crossref.org/works/10.1016%2FJ.CELL.2011.11.031

7 January 2021

The role of mitochondria in apoptosis*.

1 reference

https://api.crossref.org/works/10.1016%2FJ.CELL.2011.11.031

7 January 2021