(Q24310559)

extrinsic apoptotic signaling pathway via death domain receptors

GOA release 2020-03-11

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GOA release 2020-03-11

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X Luo

1

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I Budihardjo

2

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H Zou

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C Slaughter

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X Wang

Bcl-x(L) can inhibit apoptosis in cells that have undergone Fas-induced protease activation

5

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language of work or name

English

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publication date

21 August 1998

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published in

Cell

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volume

94

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issue

4

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page(s)

481-90

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cites work

1 reference

https://api.crossref.org/works/10.1016%2FS0092-8674%2800%2981589-5

7 January 2021

Involvement of MACH, a novel MORT1/FADD-interacting protease, in Fas/APO-1- and TNF receptor-induced cell death

1 reference

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7 January 2021

The C. elegans protein EGL-1 is required for programmed cell death and interacts with the Bcl-2-like protein CED-9

1 reference

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7 January 2021

Regulation of apoptosis by BH3 domains in a cell-free system

1 reference

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7 January 2021

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7 January 2021

A caspase-activated DNase that degrades DNA during apoptosis, and its inhibitor ICAD

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7 January 2021

Multiple species of CPP32 and Mch2 are the major active caspases present in apoptotic cells

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7 January 2021

Site-directed mutagenesis by overlap extension using the polymerase chain reaction

1 reference

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7 January 2021

Programmed cell death in animal development

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7 January 2021

Bax directly induces release of cytochrome c from isolated mitochondria

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7 January 2021

The release of cytochrome c from mitochondria: a primary site for Bcl-2 regulation of apoptosis

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7 January 2021

Caspase-3-generated fragment of gelsolin: effector of morphological change in apoptosis

1 reference

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7 January 2021

Loss of function of cytochrome c in Jurkat cells undergoing fas-mediated apoptosis

1 reference

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7 January 2021

Decreased apoptosis in the brain and premature lethality in CPP32-deficient mice

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7 January 2021

Apoptosis induction by caspase-8 is amplified through the mitochondrial release of cytochrome c.

1 reference

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7 January 2021

Studies of the lamin proteinase reveal multiple parallel biochemical pathways during apoptotic execution

1 reference

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7 January 2021

Cytochrome c and dATP-dependent formation of Apaf-1/caspase-9 complex initiates an apoptotic protease cascade

1 reference

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7 January 2021

Induction of apoptotic program in cell-free extracts: requirement for dATP and cytochrome c

1 reference

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7 January 2021

DFF, a heterodimeric protein that functions downstream of caspase-3 to trigger DNA fragmentation during apoptosis

1 reference

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7 January 2021

The 40-kDa subunit of DNA fragmentation factor induces DNA fragmentation and chromatin condensation during apoptosis

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7 January 2021

Proteolysis of fodrin (non-erythroid spectrin) during apoptosis.

1 reference

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7 January 2021

Bcl-xL acts downstream of caspase-8 activation by the CD95 death-inducing signaling complex.

1 reference

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7 January 2021

X-ray and NMR structure of human Bcl-xL, an inhibitor of programmed cell death

1 reference

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7 January 2021

FLICE, a novel FADD-homologous ICE/CED-3-like protease, is recruited to the CD95 (Fas/APO-1) death--inducing signaling complex

1 reference

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7 January 2021

FLICE Induced Apoptosis in a Cell-free System

1 reference

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7 January 2021

Apoptosis by death factor

1 reference

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7 January 2021

Bcl-2 heterodimerizes in vivo with a conserved homolog, Bax, that accelerates programmed cell death

1 reference

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7 January 2021

The overexpression of Bax produces cell death upon induction of the mitochondrial permeability transition

1 reference

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7 January 2021

Cytochrome c: can't live with it--can't live without it.

1 reference

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7 January 2021

Bcl-2 prolongs cell survival after Bax-induced release of cytochrome c

1 reference

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7 January 2021

Membrane and morphological changes in apoptotic cells regulated by caspase-mediated activation of PAK2

1 reference

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7 January 2021

Caspases: intracellular signaling by proteolysis

1 reference

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7 January 2021

Two CD95 (APO-1/Fas) signaling pathways

1 reference

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7 January 2021

Bcl-xL functions downstream of caspase-8 to inhibit Fas- and tumor necrosis factor receptor 1-induced apoptosis of MCF7 breast carcinoma cells

1 reference

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7 January 2021

Molecular ordering of the Fas-apoptotic pathway: the Fas/APO-1 protease Mch5 is a CrmA-inhibitable protease that activates multiple Ced-3/ICE-like cysteine proteases

1 reference

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7 January 2021

Bcl-2 and Fas/APO-1 regulate distinct pathways to lymphocyte apoptosis

1 reference

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7 January 2021

The central executioner of apoptosis: multiple connections between protease activation and mitochondria in Fas/APO-1/CD95- and ceramide-induced apoptosis

1 reference

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7 January 2021

Fas- and tumor necrosis factor-induced apoptosis is inhibited by the poxvirus crmA gene product

1 reference

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7 January 2021

Apoptosis in the pathogenesis and treatment of disease

1 reference

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7 January 2021

Bcl-xL regulates the membrane potential and volume homeostasis of mitochondria

1 reference

https://api.crossref.org/works/10.1016%2FS0092-8674%2800%2981589-5

7 January 2021

BID: a novel BH3 domain-only death agonist

1 reference

https://api.crossref.org/works/10.1016%2FS0092-8674%2800%2981589-5

7 January 2021

Movement of Bax from the cytosol to mitochondria during apoptosis

1 reference

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7 January 2021

Prevention of apoptosis by Bcl-2: release of cytochrome c from mitochondria blocked

1 reference

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7 January 2021

The C. elegans cell death gene ced-3 encodes a protein similar to mammalian interleukin-1 beta-converting enzyme

1 reference

https://api.crossref.org/works/10.1016%2FS0092-8674%2800%2981589-5

7 January 2021

Serine phosphorylation of death agonist BAD in response to survival factor results in binding to 14-3-3 not BCL-X(L)

1 reference

https://api.crossref.org/works/10.1016%2FS0092-8674%2800%2981589-5

7 January 2021

Apaf-1, a human protein homologous to C. elegans CED-4, participates in cytochrome c-dependent activation of caspase-3

1 reference

https://api.crossref.org/works/10.1016%2FS0092-8674%2800%2981589-5

7 January 2021

10.1016/S0092-8674(00)81589-5

Identifiers

DOI

1 reference

Consolidated OpenCitations Corpus – April 2017

974925

974925

1 reference

Consolidated OpenCitations Corpus – April 2017

Consolidated OpenCitations Corpus – April 2017

1 reference