(Q58201468)

2

0 references

Ivana Knezevic

3

0 references

Irene K. Klein

4

0 references

Asrar B. Malik

Journal of Biological Chemistry

5

0 references

language of work or name

English

0 references

publication date

3 April 2007

0 references

published in

0 references

volume

282

0 references

issue

23

0 references

page(s)

17166-17178

0 references

cites work

Splice variants of intersectin are components of the endocytic machinery in neurons and nonneuronal cells

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M608996200

Intersectin 2, a new multimodular protein involved in clathrin-mediated endocytosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M608996200

Intersectin regulates fission and internalization of caveolae in endothelial cells.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M608996200

Two isoforms of a human intersectin (ITSN) protein are produced by brain-specific alternative splicing in a stop codon

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M608996200

Neuronal distribution of EHSH1/intersectin: molecular linker between clathrin-mediated endocytosis and signaling pathways.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M608996200

The human intersectin genes and their spliced variants are differentially expressed

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M608996200

EHSH1/intersectin, a protein that contains EH and SH3 domains and binds to dynamin and SNAP-25. A protein connection between exocytosis and endocytosis?

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M608996200

Intersectin can regulate the Ras/MAP kinase pathway independent of its role in endocytosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M608996200

The endocytic protein intersectin is a major binding partner for the Ras exchange factor mSos1 in rat brain

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M608996200

Intersectin, an adaptor protein involved in clathrin-mediated endocytosis, activates mitogenic signaling pathways

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M608996200

Dap160/intersectin scaffolds the periactive zone to achieve high-fidelity endocytosis and normal synaptic growth.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M608996200

Intersectin activates Ras but stimulates transcription through an independent pathway involving JNK.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M608996200

Cdc42 and Ras cooperate to mediate cellular transformation by intersectin-L.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M608996200

Endothelial transcytotic machinery involves supramolecular protein-lipid complexes

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M608996200

Assays for cytochrome c release from mitochondria during apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M608996200

A distinct pathway remodels mitochondrial cristae and mobilizes cytochrome c during apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M608996200

Mechanisms of cytochrome c release by proapoptotic BCL-2 family members

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M608996200

Caspase-mediated loss of mitochondrial function and generation of reactive oxygen species during apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M608996200

Caspase-2 induces apoptosis by releasing proapoptotic proteins from mitochondria

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M608996200

Requirement for caspase-2 in stress-induced apoptosis before mitochondrial permeabilization

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M608996200

The permeability transition pore triggers Bax translocation to mitochondria during neuronal apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M608996200

Inhibition of mitochondrial permeability transition prevents mitochondrial dysfunction, cytochrome c release and apoptosis induced by heart ischemia

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M608996200

Transient and long-lasting openings of the mitochondrial permeability transition pore can be monitored directly in intact cells by changes in mitochondrial calcein fluorescence

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M608996200

DPP10 is an inactivation modulatory protein of Kv4.3 and Kv1.4.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M608996200

JNK suppresses apoptosis via phosphorylation of the proapoptotic Bcl-2 family protein BAD

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M608996200

Characterization of superoxide-producing sites in isolated brain mitochondria

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M608996200

Enforced dimerization of BAX results in its translocation, mitochondrial dysfunction and apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M608996200

Movement of Bax from the cytosol to mitochondria during apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M608996200

Mitochondrial membrane permeabilization: the sine qua non for cell death

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M608996200

Oncogenic tyrosine kinase NPM/ALK induces activation of the MEK/ERK signaling pathway independently of c-Raf.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M608996200

BH3-only proteins that bind pro-survival Bcl-2 family members fail to induce apoptosis in the absence of Bax and Bak

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M608996200

BCL-2, BCL-X(L) sequester BH3 domain-only molecules preventing BAX- and BAK-mediated mitochondrial apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M608996200

Pro-apoptotic cascade activates BID, which oligomerizes BAK or BAX into pores that result in the release of cytochrome c.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M608996200

Mechanisms of cytochrome c release from mitochondria

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M608996200

Multiple pathways of cytochrome c release from mitochondria in apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M608996200

Caspase-2 permeabilizes the outer mitochondrial membrane and disrupts the binding of cytochrome c to anionic phospholipids

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M608996200

Mitochondrial cytochrome c release may occur by volume-dependent mechanisms not involving permeability transition

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M608996200

Mitochondrial outer membrane permeabilization during apoptosis: the innocent bystander scenario

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M608996200

The apoptosome: physiological, developmental, and pathological modes of regulation

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M608996200

Mitochondrial fission in apoptosis, neurodegeneration and aging

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M608996200

Mitochondrial fission in apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M608996200

Multiple Bcl-2 family members demonstrate selective dimerizations with Bax

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M608996200

Cytosol-to-membrane redistribution of Bax and Bcl-X(L) during apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M608996200

Proapoptotic BAX and BAK: a requisite gateway to mitochondrial dysfunction and death

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M608996200

The role of the Bcl-2 family in the regulation of outer mitochondrial membrane permeability

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M608996200

Opposing effects of ERK and JNK-p38 MAP kinases on apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M608996200

Suppression of survival kinases and activation of JNK mediate ethanol-induced cell death in the developing rat brain

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M608996200

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M608996200

Granzyme B proteolyzes receptors important to proliferation and survival, tipping the balance toward apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M608996200

Caveolin-1 null mice are viable but show evidence of hyperproliferative and vascular abnormalities

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M608996200