(Q24296721)

English

Vinexin beta interacts with the non-phosphorylated AF-1 domain of retinoid receptor gamma (RARgamma) and represses RARgamma-mediated transcription

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Vinexin beta interacts with the non-phosphorylated AF-1 domain of retinoid receptor gamma (RARgamma) and represses RARgamma-mediated transcription (English)

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phosphorylation

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Sorbin and SH3 domain containing 3

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GOA release 2020-03-11

Retinoic acid receptor, gamma

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GOA release 2020-03-11

author name string

Gaétan Bour

1

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Jean-Luc Plassat

2

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Annie Bauer

3

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Sébastien Lalevée

4

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Cécile Rochette-Egly

5

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language of work or name

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publication date

29 April 2005

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Journal of Biological Chemistry

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280

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17

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17027-37

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Promoter context- and response element-dependent specificity of the transcriptional activation and modulating functions of retinoic acid receptors

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Nuclear retinoid receptors and the transcription of retinoid-target genes

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Stimulation of RAR alpha activation function AF-1 through binding to the general transcription factor TFIIH and phosphorylation by CDK7

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XPD mutations prevent TFIIH-dependent transactivation by nuclear receptors and phosphorylation of RARalpha

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The nuclear receptor corepressor (N-CoR) modulates basal and activated transcription of genes controlled by retinoic acid

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Loss of retinoic acid receptor gamma function in F9 cells by gene disruption results in aberrant Hoxa-1 expression and differentiation upon retinoic acid treatment

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Reexpression of retinoic acid receptor (RAR) gamma or overexpression of RAR alpha or RAR beta in RAR gamma-null F9 cells reveals a partial functional redundancy between the three RAR types

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Down-regulation of the phosphatidylinositol 3-kinase/Akt pathway is involved in retinoic acid-induced phosphorylation, degradation, and transcriptional activity of retinoic acid receptor gamma 2.

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TFIIH interacts with the retinoic acid receptor gamma and phosphorylates its AF-1-activating domain through cdk7

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Identifiers

10.1074/JBC.M501344200

1 reference

Consolidated OpenCitations Corpus – April 2017

OpenCitations bibliographic resource ID

release_wyktrpughvfodg37hsrmyvwkue

1 reference

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24 November 2022

based on heuristic

mapped directly with Wikidata item

OpenCitations bibliographic resource ID

1 reference

Consolidated OpenCitations Corpus – April 2017

OpenCitations bibliographic resource ID

PubMed publication ID

1 reference

Consolidated OpenCitations Corpus – April 2017

OpenCitations bibliographic resource ID

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