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Cytidine deamination induced HIV-1 drug resistance
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scholarly article
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title
Cytidine deamination induced HIV-1 drug resistance
(English)
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author
Viviana Simon
object named as
Viviana Simon
series ordinal
3
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author name string
Lubbertus C F Mulder
series ordinal
1
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Ariana Harari
series ordinal
2
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language of work or name
English
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publication date
8 April 2008
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published in
Proceedings of the National Academy of Sciences of the United States of America
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volume
105
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page(s)
5501-6
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issue
14
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cites work
7SL RNA mediates virion packaging of the antiviral cytidine deaminase APOBEC3G
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Natural variation in Vif: differential impact on APOBEC3G/3F and a potential role in HIV-1 diversification
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PubMed Central
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A second human antiretroviral factor, APOBEC3F, is suppressed by the HIV-1 and HIV-2 Vif proteins
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Human APOBEC3F is another host factor that blocks human immunodeficiency virus type 1 replication
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APOBEC3F properties and hypermutation preferences indicate activity against HIV-1 in vivo
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Virology. Weapons of mutational destruction
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DNA deamination: not just a trigger for antibody diversification but also a mechanism for defense against retroviruses
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Enzymatically active APOBEC3G is required for efficient inhibition of human immunodeficiency virus type 1.
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Cytidine deaminases APOBEC3G and APOBEC3F interact with human immunodeficiency virus type 1 integrase and inhibit proviral DNA formation
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G-->A hypermutation in protease and reverse transcriptase regions of human immunodeficiency virus type 1 residing in resting CD4+ T cells in vivo
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Antiretroviral drug resistance mutations in human immunodeficiency virus type 1 reverse transcriptase increase template-switching frequency
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The human immunodeficiency virus type 1 Vif protein reduces intracellular expression and inhibits packaging of APOBEC3G (CEM15), a cellular inhibitor of virus infectivity
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Human immunodeficiency virus type 1 DNA sequences genetically damaged by hypermutation are often abundant in patient peripheral blood mononuclear cells and may be generated during near-simultaneous infection and activation of CD4(+) T cells
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28 September 2017
HIV-1 sequence variation: drift, shift, and attenuation
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Biased G-to-A hypermutation in HIV-1 proviral DNA from a long-term non-progressor.
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28 November 2018
Recombination: Multiply infected spleen cells in HIV patients
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Detecting hypermutations in viral sequences with an emphasis on G --> A hypermutation
1 reference
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PubMed
reference URL
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retrieved
12 December 2020
based on heuristic
inferred from PubMed ID database lookup
Identifiers
DOI
10.1073/PNAS.0710190105
1 reference
stated in
Consolidated OpenCitations Corpus – April 2017
OpenCitations bibliographic resource ID
1596956
ADS bibcode
2008PNAS..105.5501M
0 references
OpenCitations bibliographic resource ID
1596956
1 reference
stated in
Consolidated OpenCitations Corpus – April 2017
OpenCitations bibliographic resource ID
1596956
PMCID
2291111
1 reference
stated in
Consolidated OpenCitations Corpus – April 2017
OpenCitations bibliographic resource ID
1596956
PubMed ID
18391217
1 reference
stated in
Consolidated OpenCitations Corpus – April 2017
OpenCitations bibliographic resource ID
1596956
ResearchGate publication ID
5461779
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