(Q28141602)

6

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William C Hahn

1

stated as

W C Hahn

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author name string

C M Counter

2

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A S Lundberg

3

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R L Beijersbergen

4

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M W Brooks

https://scigraph.springernature.com/pub.10.1038/22780

5

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language of work or name

English

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publication date

29 July 1999

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published in

Nature

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volume

400

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page(s)

464-8

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issue

6743

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exact match

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cites work

Tumorigenic conversion of primary embryo fibroblasts requires at least two cooperating oncogenes

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Adenovirus early region 1A enables viral and cellular transforming genes to transform primary cells in culture.

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Senescence as a mode of tumor suppression

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Suppression of tumor growth by senescence in virally transformed human fibroblasts.

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Activated v-myc and v-ras oncogenes do not transform normal human lymphocytes.

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Transformation of human bronchial epithelial cells transfected by Harvey ras oncogene.

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Neoplastic transformation of human epidermal keratinocytes by AD12-SV40 and Kirsten sarcoma viruses.

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Malignant transformation of human fibroblasts caused by expression of a transfected T24 HRAS oncogene.

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Transformation of primary human endothelial cells by Kaposi's sarcoma-associated herpesvirus.

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Reversing time: origin of telomerase

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Telomere structure and telomerase expression during mouse development and tumorigenesis.

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Specific association of human telomerase activity with immortal cells and cancer

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A survey of telomerase activity in human cancer

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Extension of life-span by introduction of telomerase into normal human cells

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Both Rb/p16INK4a inactivation and telomerase activity are required to immortalize human epithelial cells.

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Crisis intervention: the role of telomerase

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Oncogenic ras provokes premature cell senescence associated with accumulation of p53 and p16INK4a

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Dissociation among in vitro telomerase activity, telomere maintenance, and cellular immortalization.

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Absence of cancer-associated changes in human fibroblasts immortalized with telomerase.

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The J domain of simian virus 40 large T antigen is required to functionally inactivate RB family proteins.

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Simian virus 40 large T antigen interacts with human TFIIB-related factor and small nuclear RNA-activating protein complex for transcriptional activation of TATA-containing polymerase III promoters.

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Telomerase expression in human somatic cells does not induce changes associated with a transformed phenotype.

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Recombinant retroviruses encoding simian virus 40 large T antigen and polyomavirus large and middle T antigens.

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Correlation of patterns of anchorage-independent growth with in vivo behavior of cells from a murine fibrosarcoma.

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