(Q28267110)

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Caspase cleavage of gene products associated with triplet expansion disorders generates truncated fragments containing the polyglutamine tract

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10 November 2019

Caspase cleavage of gene products associated with triplet expansion disorders generates truncated fragments containing the polyglutamine tract (English)

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10 November 2019

Roshni Singaraja

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10 November 2019

Michael R. Hayden

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10 November 2019

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L M Ellerby

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10 November 2019

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C L Wellington

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10 November 2019

A S Hackam

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10 November 2019

R L Margolis

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10 November 2019

M A Trifiro

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10 November 2019

K McCutcheon

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10 November 2019

G S Salvesen

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10 November 2019

S S Propp

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10 November 2019

M Bromm

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10 November 2019

K J Rowland

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10 November 2019

T Zhang

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10 November 2019

D Rasper

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10 November 2019

S Roy

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10 November 2019

N Thornberry

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10 November 2019

L Pinsky

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10 November 2019

A Kakizuka

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10 November 2019

C A Ross

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10 November 2019

D W Nicholson

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10 November 2019

D E Bredesen

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10 November 2019

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1 April 1998

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10 November 2019

Journal of Biological Chemistry

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10 November 2019

273

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10 November 2019

9158-9167

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10 November 2019

15

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10 November 2019

Toward understanding the molecular pathology of Huntington's disease.

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The neuropathology of CAG repeat diseases: review and update of genetic and molecular features

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21 January 2018

The CAG/polyglutamine tract diseases: gene products and molecular pathogenesis

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Huntington's disease and dentatorubral-pallidoluysian atrophy: proteins, pathogenesis and pathology

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Cloning of the SCA7 gene reveals a highly unstable CAG repeat expansion

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21 January 2018

Clinical aspects of CAG repeat diseases

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21 January 2018

Rethinking genotype and phenotype correlations in polyglutamine expansion disorders

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21 January 2018

When more is less: pathogenesis of glutamine repeat neurodegenerative diseases

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21 January 2018

Aggregation of huntingtin in neuronal intranuclear inclusions and dystrophic neurites in brain

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21 January 2018

Exon 1 of the HD gene with an expanded CAG repeat is sufficient to cause a progressive neurological phenotype in transgenic mice

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21 January 2018

Formation of neuronal intranuclear inclusions underlies the neurological dysfunction in mice transgenic for the HD mutation

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21 January 2018

Intranuclear inclusions of expanded polyglutamine protein in spinocerebellar ataxia type 3.

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21 January 2018

Suppression of aggregate formation and apoptosis by transglutaminase inhibitors in cells expressing truncated DRPLA protein with an expanded polyglutamine stretch.

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21 January 2018

Cleavage of huntingtin by apopain, a proapoptotic cysteine protease, is modulated by the polyglutamine tract

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21 January 2018

Protease activation during apoptosis: death by a thousand cuts?

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21 January 2018

Genetic control of programmed cell death in the nematode C. elegans.

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21 January 2018

A combinatorial approach defines specificities of members of the caspase family and granzyme B. Functional relationships established for key mediators of apoptosis

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21 January 2018

Absence of disease phenotype and intergenerational stability of the CAG repeat in transgenic mice expressing the human Huntington disease transcript

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21 January 2018

DRPLA gene (atrophin-1) sequence and mRNA expression in human brain

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Expanded polyglutamine in the Machado-Joseph disease protein induces cell death in vitro and in vivo

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Evidence for a repressive function of the long polyglutamine tract in the human androgen receptor: possible pathogenetic relevance for the (CAG)n-expanded neuronopathies

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21 January 2018

Identification and inhibition of the ICE/CED-3 protease necessary for mammalian apoptosis

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A combinatorial approach for determining protease specificities: application to interleukin-1beta converting enzyme (ICE)

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The three-dimensional structure of apopain/CPP32, a key mediator of apoptosis

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Target protease specificity of the viral serpin CrmA. Analysis of five caspases

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Death substrates come alive

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Functional domains and upstream activation properties of cloned human TATA binding protein

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Alternative cleavage of Alzheimer-associated presenilins during apoptosis by a caspase-3 family protease

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21 January 2018

Presenilins are processed by caspase-type proteases.

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Huntingtin is ubiquitinated and interacts with a specific ubiquitin-conjugating enzyme

1 reference

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The regulation of anoikis: MEKK-1 activation requires cleavage by caspases

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ICE/CED3-like proteases as therapeutic targets for the control of inappropriate apoptosis.

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Of molecular interactions, mice and mechanisms

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Identifiers

10.1074/JBC.273.15.9158

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Europe PubMed Central

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10 November 2019

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Consolidated OpenCitations Corpus – April 2017

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Europe PubMed Central

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10 November 2019

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