(Q28731292)

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Amyloid-like fibril formation by polyQ proteins: a critical balance between the polyQ length and the constraints imposed by the host protein

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Amyloid-like fibril formation by polyQ proteins: a critical balance between the polyQ length and the constraints imposed by the host protein (English)

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author name string

Natacha Scarafone

1

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Coralie Pain

2

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Anthony Fratamico

3

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Gilles Gaspard

4

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Nursel Yilmaz

5

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Patrice Filée

6

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Moreno Galleni

7

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André Matagne

8

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Mireille Dumoulin

9

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publication date

2012

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7

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3

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e31253

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Creative Commons Attribution 4.0 International

9 March 2012

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April 2022 Public Data File from Crossref

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Secondary Structure of Huntingtin Amino-Terminal Region

1 reference

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17 March 2017

A camelid antibody fragment inhibits the formation of amyloid fibrils by human lysozyme

1 reference

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17 March 2017

Beta-lactamase of Bacillus licheniformis 749/C. Refinement at 2 A resolution and analysis of hydration

1 reference

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17 March 2017

Trinucleotide Repeat Disorders

1 reference

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7 April 2017

Aggregation of huntingtin in neuronal intranuclear inclusions and dystrophic neurites in brain

1 reference

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7 April 2017

Unfolding free energy changes determined by the linear extrapolation method. 1. Unfolding of phenylmethanesulfonyl alpha-chymotrypsin using different denaturants

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3305072

7 April 2017

Flanking domain stability modulates the aggregation kinetics of a polyglutamine disease protein

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3305072

3 June 2018

Location trumps length: polyglutamine-mediated changes in folding and aggregation of a host protein

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3305072

3 June 2018

Towards the treatment of polyglutamine diseases: the modulatory role of protein context

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3305072

3 June 2018

The non-core regions of human lysozyme amyloid fibrils influence cytotoxicity

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3305072

3 June 2018

Tracking mutant huntingtin aggregation kinetics in cells reveals three major populations that include an invariant oligomer pool.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3305072

3 June 2018

Polyglutamine induced misfolding of huntingtin exon1 is modulated by the flanking sequences

1 reference

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3 June 2018

The structural impact of a polyglutamine tract is location-dependent

1 reference

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3 June 2018

Polyglutamine neurodegeneration: protein misfolding revisited.

1 reference

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3 June 2018

A strategy for generating polyglutamine 'length libraries' in model host proteins

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3305072

3 June 2018

Soluble polyglutamine oligomers formed prior to inclusion body formation are cytotoxic

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3305072

3 June 2018

In-cell aggregation of a polyglutamine-containing chimera is a multistep process initiated by the flanking sequence

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3305072

3 June 2018

The Bacillus licheniformis BlaP beta-lactamase as a model protein scaffold to study the insertion of protein fragments

1 reference

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3 June 2018

Quantitative characterization of intrinsic disorder in polyglutamine: insights from analysis based on polymer theories

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3305072

3 June 2018

The interplay between PolyQ and protein context delays aggregation by forming a reservoir of protofibrils

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3305072

3 June 2018

Fluorescence correlation spectroscopy shows that monomeric polyglutamine molecules form collapsed structures in aqueous solutions

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3305072

3 June 2018

Normal and aberrant biological self-assembly: Insights from studies of human lysozyme and its amyloidogenic variants.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3305072

3 June 2018

Extended polyglutamine tracts cause aggregation and structural perturbation of an adjacent beta barrel protein

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3305072

3 June 2018

Native state kinetic stabilization as a strategy to ameliorate protein misfolding diseases: a focus on the transthyretin amyloidoses.

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3305072

3 June 2018

Formation of morphologically similar globular aggregates from diverse aggregation-prone proteins in mammalian cells

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3305072

3 June 2018

A linear lattice model for polyglutamine in CAG-expansion diseases

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3305072

3 June 2018

Single-domain antibody fragments with high conformational stability

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3305072

3 June 2018

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1 reference

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3 June 2018

Aggregation of truncated GST-HD exon 1 fusion proteins containing normal range and expanded glutamine repeats

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3305072

3 June 2018

Truncated N-terminal fragments of huntingtin with expanded glutamine repeats form nuclear and cytoplasmic aggregates in cell culture.

1 reference

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3 June 2018

Length of huntingtin and its polyglutamine tract influences localization and frequency of intracellular aggregates

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3305072

3 June 2018

Intranuclear neuronal inclusions: a common pathogenic mechanism for glutamine-repeat neurodegenerative diseases?

1 reference

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3 June 2018

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1 reference

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The neuropathology of CAG repeat diseases: review and update of genetic and molecular features

1 reference

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3 June 2018

Glutamine repeats as polar zippers: their possible role in inherited neurodegenerative diseases

1 reference

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3 June 2018

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1 reference

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3 June 2018

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1 reference

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3 June 2018

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1 reference

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3 June 2018

Sequential Edman degredation

1 reference

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3 June 2018

Ectopically expressed CAG repeats cause intranuclear inclusions and a progressive late onset neurological phenotype in the mouse

1 reference

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Biochemical, ultrastructural, and reversibility studies on huntingtin filaments isolated from mouse and human brain.

1 reference

https://api.crossref.org/works/10.1371%2FJOURNAL.PONE.0031253

21 January 2018

Pivotal role of oligomerization in expanded polyglutamine neurodegenerative disorders.

1 reference

https://api.crossref.org/works/10.1371%2FJOURNAL.PONE.0031253

21 January 2018

Aggregated polyglutamine peptides delivered to nuclei are toxic to mammalian cells

1 reference

https://api.crossref.org/works/10.1371%2FJOURNAL.PONE.0031253

21 January 2018

Intra- and intermolecular beta-pleated sheet formation in glutamine-repeat inserted myoglobin as a model for polyglutamine diseases

1 reference

https://api.crossref.org/works/10.1371%2FJOURNAL.PONE.0031253

21 January 2018

Polyglutamine disruption of the huntingtin exon 1 N terminus triggers a complex aggregation mechanism

1 reference

https://api.crossref.org/works/10.1371%2FJOURNAL.PONE.0031253

21 January 2018

Oligoproline effects on polyglutamine conformation and aggregation

1 reference

https://api.crossref.org/works/10.1371%2FJOURNAL.PONE.0031253

21 January 2018

Amino acid sequences flanking polyglutamine stretches influence their potential for aggregate formation

1 reference

https://api.crossref.org/works/10.1371%2FJOURNAL.PONE.0031253

21 January 2018

Flanking polyproline sequences inhibit beta-sheet structure in polyglutamine segments by inducing PPII-like helix structure

1 reference

https://api.crossref.org/works/10.1371%2FJOURNAL.PONE.0031253

21 January 2018

The two-stage pathway of ataxin-3 fibrillogenesis involves a polyglutamine-independent step.

1 reference

https://api.crossref.org/works/10.1371%2FJOURNAL.PONE.0031253

21 January 2018

Solution structure of polyglutamine tracts in GST-polyglutamine fusion proteins

1 reference

https://api.crossref.org/works/10.1371%2FJOURNAL.PONE.0031253

21 January 2018

Rapid and easy development of versatile tools to study protein/ligand interactions.

1 reference

https://api.crossref.org/works/10.1371%2FJOURNAL.PONE.0031253

21 January 2018

Huntingtin-encoded polyglutamine expansions form amyloid-like protein aggregates in vitro and in vivo

1 reference

https://api.crossref.org/works/10.1371%2FJOURNAL.PONE.0031253

21 January 2018

Polyglutamine aggregation behavior in vitro supports a recruitment mechanism of cytotoxicity

1 reference

https://api.crossref.org/works/10.1371%2FJOURNAL.PONE.0031253

21 January 2018

Folding of class A beta-lactamases is rate-limited by peptide bond isomerization and occurs via parallel pathways

1 reference

https://api.crossref.org/works/10.1371%2FJOURNAL.PONE.0031253

21 January 2018

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1 reference

https://api.crossref.org/works/10.1371%2FJOURNAL.PONE.0031253

21 January 2018

Pathogenic and Non-pathogenic Polyglutamine Tracts Have Similar Structural Properties: Towards a Length-dependent Toxicity Gradient

1 reference

https://api.crossref.org/works/10.1371%2FJOURNAL.PONE.0031253

21 January 2018

Mechanisms of Ataxin-3 Misfolding and Fibril Formation: Kinetic Analysis of a Disease-associated Polyglutamine Protein

1 reference

https://api.crossref.org/works/10.1371%2FJOURNAL.PONE.0031253

21 January 2018

The Rate of PolyQ-Mediated Aggregation Is Dramatically Affected by the Number and Location of Surrounding Domains

1 reference

https://api.crossref.org/works/10.1371%2FJOURNAL.PONE.0031253

21 January 2018

[34] X-Ray fiber diffraction of amyloid fibrils

1 reference

https://api.crossref.org/works/10.1371%2FJOURNAL.PONE.0031253

21 January 2018

Glutamine, alanine or glycine repeats inserted into the loop of a protein have minimal effects on stability and folding rates

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=3305072

27 November 2018

In vitro synthesis and cloning of long CAG repeats

1 reference

https://pubmed.ncbi.nlm.nih.gov/22438863

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Peptide models for inherited neurodegenerative disorders: conformation and aggregation properties of long polyglutamine peptides with and without interruptions

1 reference

https://pubmed.ncbi.nlm.nih.gov/22438863

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

A toxic monomeric conformer of the polyglutamine protein

1 reference

https://pubmed.ncbi.nlm.nih.gov/22438863

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Identifiers

10.1371/JOURNAL.PONE.0031253

1 reference

Consolidated OpenCitations Corpus – April 2017

OpenCitations bibliographic resource ID

2012PLoSO...731253S

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OpenCitations bibliographic resource ID

1 reference

Consolidated OpenCitations Corpus – April 2017

OpenCitations bibliographic resource ID

PMC publication ID

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