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Cardiac hypertrophy: sorting out the circuitry
scientific article
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scholarly article
1 reference
stated in
Europe PubMed Central
PubMed ID
10377279
retrieved
28 July 2017
review article
1 reference
stated in
Europe PubMed Central
title
Cardiac hypertrophy: sorting out the circuitry
(English)
1 reference
stated in
Europe PubMed Central
PubMed ID
10377279
retrieved
28 July 2017
author name string
McKinsey TA
series ordinal
1
1 reference
stated in
Europe PubMed Central
PubMed ID
10377279
retrieved
28 July 2017
Olson EN
series ordinal
2
1 reference
stated in
Europe PubMed Central
PubMed ID
10377279
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28 July 2017
publication date
1 June 1999
1 reference
stated in
Europe PubMed Central
PubMed ID
10377279
retrieved
28 July 2017
published in
Current Opinion in Genetics & Development
1 reference
stated in
Europe PubMed Central
PubMed ID
10377279
retrieved
28 July 2017
volume
9
1 reference
stated in
Europe PubMed Central
PubMed ID
10377279
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28 July 2017
page(s)
267-274
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stated in
Europe PubMed Central
PubMed ID
10377279
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28 July 2017
issue
3
1 reference
stated in
Europe PubMed Central
PubMed ID
10377279
retrieved
28 July 2017
cites work
Cellular mechanisms of cardiac hypertrophy
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Mechanism of action of beta-blocking agents in heart failure
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Angiotensin II type 1a receptor-deficient mice with hypotension and hyperreninemia
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Pressure overload induces cardiac hypertrophy in angiotensin II type 1A receptor knockout mice.
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Targeting the receptor-Gq interface to inhibit in vivo pressure overload myocardial hypertrophy
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https://api.crossref.org/works/10.1016%2FS0959-437X%2899%2980040-9
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7 January 2021
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Cardiac function in mice overexpressing the beta-adrenergic receptor kinase or a beta ARK inhibitor
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https://api.crossref.org/works/10.1016%2FS0959-437X%2899%2980040-9
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MLP-deficient mice exhibit a disruption of cardiac cytoarchitectural organization, dilated cardiomyopathy, and heart failure
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The MEKK-JNK pathway is stimulated by alpha1-adrenergic receptor and ras activation and is associated with in vitro and in vivo cardiac hypertrophy.
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Ventricular expression of a MLC-2v-ras fusion gene induces cardiac hypertrophy and selective diastolic dysfunction in transgenic mice.
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A Ras-dependent pathway regulates RNA polymerase II phosphorylation in cardiac myocytes: implications for cardiac hypertrophy
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MAP kinase- and Rho-dependent signals interact to regulate gene expression but not actin morphology in cardiac muscle cells
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https://api.crossref.org/works/10.1016%2FS0959-437X%2899%2980040-9
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Cardiac hypertrophy induced by mitogen-activated protein kinase kinase 7, a specific activator for c-Jun NH2-terminal kinase in ventricular muscle cells
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https://api.crossref.org/works/10.1016%2FS0959-437X%2899%2980040-9
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7 January 2021
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Role of the stress-activated protein kinases in endothelin-induced cardiomyocyte hypertrophy
1 reference
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https://api.crossref.org/works/10.1016%2FS0959-437X%2899%2980040-9
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7 January 2021
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Opposing effects of Jun kinase and p38 mitogen-activated protein kinases on cardiomyocyte hypertrophy
1 reference
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https://api.crossref.org/works/10.1016%2FS0959-437X%2899%2980040-9
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A role for the p38 mitogen-activated protein kinase pathway in myocardial cell growth, sarcomeric organization, and cardiac-specific gene expression
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https://api.crossref.org/works/10.1016%2FS0959-437X%2899%2980040-9
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7 January 2021
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Stimulation of the p38 mitogen-activated protein kinase pathway in neonatal rat ventricular myocytes by the G protein-coupled receptor agonists, endothelin-1 and phenylephrine: a role in cardiac myocyte hypertrophy?
1 reference
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Crossref
reference URL
https://api.crossref.org/works/10.1016%2FS0959-437X%2899%2980040-9
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7 January 2021
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MKK6 activates myocardial cell NF-kappaB and inhibits apoptosis in a p38 mitogen-activated protein kinase-dependent manner
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Crossref
reference URL
https://api.crossref.org/works/10.1016%2FS0959-437X%2899%2980040-9
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7 January 2021
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Cardiac muscle cell hypertrophy and apoptosis induced by distinct members of the p38 mitogen-activated protein kinase family
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Cardiotrophin 1 (CT-1) inhibition of cardiac myocyte apoptosis via a mitogen-activated protein kinase-dependent pathway. Divergence from downstream CT-1 signals for myocardial cell hypertrophy
1 reference
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https://api.crossref.org/works/10.1016%2FS0959-437X%2899%2980040-9
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cis-Acting sequences that mediate induction of beta-myosin heavy chain gene expression during left ventricular hypertrophy due to aortic constriction
1 reference
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Crossref
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https://api.crossref.org/works/10.1016%2FS0959-437X%2899%2980040-9
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7 January 2021
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A calcineurin-dependent transcriptional pathway for cardiac hypertrophy
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Crossref
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https://api.crossref.org/works/10.1016%2FS0959-437X%2899%2980040-9
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Transcription factors of the NFAT family: regulation and function
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The mechanism of action of cyclosporin A and FK506
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Crossref
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Dilated cardiomyopathy in transgenic mice expressing a dominant-negative CREB transcription factor in the heart
1 reference
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7 January 2021
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Calcineurin associated with the inositol 1,4,5-trisphosphate receptor-FKBP12 complex modulates Ca2+ flux
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Differential activation of transcription factors induced by Ca2+ response amplitude and duration
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Exchange of beta- for alpha-tropomyosin in hearts of transgenic mice induces changes in thin filament response to Ca2+, strong cross-bridge binding, and protein phosphorylation
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https://api.crossref.org/works/10.1016%2FS0959-437X%2899%2980040-9
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Prevention of Cardiac Hypertrophy in Mice by Calcineurin Inhibition
1 reference
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Crossref
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https://api.crossref.org/works/10.1016%2FS0959-437X%2899%2980040-9
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7 January 2021
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Cardiac compartment-specific overexpression of a modified retinoic acid receptor produces dilated cardiomyopathy and congestive heart failure in transgenic mice
1 reference
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Crossref
reference URL
https://api.crossref.org/works/10.1016%2FS0959-437X%2899%2980040-9
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7 January 2021
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Cyclosporine attenuates pressure-overload hypertrophy in mice while enhancing susceptibility to decompensation and heart failure
1 reference
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Crossref
reference URL
https://api.crossref.org/works/10.1016%2FS0959-437X%2899%2980040-9
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7 January 2021
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Calcineurin inhibitors and cardiac hypertrophy
1 reference
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Crossref
reference URL
https://api.crossref.org/works/10.1016%2FS0959-437X%2899%2980040-9
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7 January 2021
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Failure of calcineurin inhibitors to prevent pressure-overload left ventricular hypertrophy in rats
1 reference
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Crossref
reference URL
https://api.crossref.org/works/10.1016%2FS0959-437X%2899%2980040-9
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based on heuristic
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Expression of protein kinase C beta in the heart causes hypertrophy in adult mice and sudden death in neonates.
1 reference
stated in
Crossref
reference URL
https://api.crossref.org/works/10.1016%2FS0959-437X%2899%2980040-9
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7 January 2021
based on heuristic
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Increased protein kinase C activity and expression of Ca2+-sensitive isoforms in the failing human heart
1 reference
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Crossref
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https://api.crossref.org/works/10.1016%2FS0959-437X%2899%2980040-9
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7 January 2021
based on heuristic
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Leukemia inhibitory factor, a potent cardiac hypertrophic cytokine, activates the JAK/STAT pathway in rat cardiomyocytes
1 reference
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Crossref
reference URL
https://api.crossref.org/works/10.1016%2FS0959-437X%2899%2980040-9
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7 January 2021
based on heuristic
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Tumor necrosis factor-alpha provokes a hypertrophic growth response in adult cardiac myocytes
1 reference
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Crossref
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https://api.crossref.org/works/10.1016%2FS0959-437X%2899%2980040-9
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7 January 2021
based on heuristic
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Pathophysiologically relevant concentrations of tumor necrosis factor-alpha promote progressive left ventricular dysfunction and remodeling in rats
1 reference
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reference URL
https://api.crossref.org/works/10.1016%2FS0959-437X%2899%2980040-9
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7 January 2021
based on heuristic
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Cardiac failure in transgenic mice with myocardial expression of tumor necrosis factor-alpha.
1 reference
stated in
Crossref
reference URL
https://api.crossref.org/works/10.1016%2FS0959-437X%2899%2980040-9
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7 January 2021
based on heuristic
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The nuclear deltaB isoform of Ca2+/calmodulin-dependent protein kinase II regulates atrial natriuretic factor gene expression in ventricular myocytes
1 reference
stated in
Crossref
reference URL
https://api.crossref.org/works/10.1016%2FS0959-437X%2899%2980040-9
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7 January 2021
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Identifiers
DOI
10.1016/S0959-437X(99)80040-9
1 reference
stated in
Europe PubMed Central
PubMed ID
10377279
retrieved
28 July 2017
PubMed ID
10377279
1 reference
stated in
Europe PubMed Central
PubMed ID
10377279
retrieved
28 July 2017
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