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The Mre11 complex and ATM: collaborating to navigate S phase
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scholarly article
1 reference
stated in
Europe PubMed Central
PubMed ID
10801460
retrieved
30 July 2017
review article
1 reference
stated in
Europe PubMed Central
title
The Mre11 complex and ATM: collaborating to navigate S phase
(English)
1 reference
stated in
Europe PubMed Central
PubMed ID
10801460
retrieved
30 July 2017
main subject
collaboration
1 reference
based on heuristic
inferred from title
author name string
Petrini JH
series ordinal
1
1 reference
stated in
Europe PubMed Central
PubMed ID
10801460
retrieved
30 July 2017
publication date
1 June 2000
1 reference
stated in
Europe PubMed Central
PubMed ID
10801460
retrieved
30 July 2017
published in
Current Opinion in Cell Biology
1 reference
stated in
Europe PubMed Central
PubMed ID
10801460
retrieved
30 July 2017
volume
12
1 reference
stated in
Europe PubMed Central
PubMed ID
10801460
retrieved
30 July 2017
page(s)
293-296
1 reference
stated in
Europe PubMed Central
PubMed ID
10801460
retrieved
30 July 2017
issue
3
1 reference
stated in
Europe PubMed Central
PubMed ID
10801460
retrieved
30 July 2017
cites work
RuvAB acts at arrested replication forks
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The DNA replication protein PriA and the recombination protein RecG bind D-loops.
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DNA double-strand breaks caused by replication arrest
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Holliday junctions accumulate in replication mutants via a RecA homolog-independent mechanism
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The double-strand-break repair model for recombination
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Double-strand break repair in yeast requires both leading and lagging strand DNA polymerases
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Mre11 is essential for the maintenance of chromosomal DNA in vertebrate cells
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A mutation in mouse rad51 results in an early embryonic lethal that is suppressed by a mutation in p53
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Disruption of mRad50 causes embryonic stem cell lethality, abnormal embryonic development, and sensitivity to ionizing radiation
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Rad51-deficient vertebrate cells accumulate chromosomal breaks prior to cell death
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Embryonic lethality and radiation hypersensitivity mediated by Rad51 in mice lacking Brca2
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7 January 2021
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Conditional gene targeted deletion by Cre recombinase demonstrates the requirement for the double-strand break repair Mre11 protein in murine embryonic stem cells
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7 January 2021
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The mammalian Mre11-Rad50-nbs1 protein complex: integration of functions in the cellular DNA-damage response
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7 January 2021
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The many interfaces of Mre11
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7 January 2021
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The Mre11-Rad50-Xrs2 protein complex facilitates homologous recombination-based double-strand break repair in Saccharomyces cerevisiae
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The hMre11/hRad50 protein complex and Nijmegen breakage syndrome: linkage of double-strand break repair to the cellular DNA damage response
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Ataxia-telangiectasia and the Nijmegen breakage syndrome: related disorders but genes apart
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A single ataxia telangiectasia gene with a product similar to PI-3 kinase
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7 January 2021
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Responses to DNA damage and regulation of cell cycle checkpoints by the ATM protein kinase family
1 reference
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reference URL
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7 January 2021
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ATM phosphorylates p95/nbs1 in an S-phase checkpoint pathway
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The DNA double-strand break repair gene hMRE11 is mutated in individuals with an ataxia-telangiectasia-like disorder
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7 January 2021
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A family showing no evidence of linkage between the ataxia telangiectasia gene and chromosome 11q22-23.
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Ataxia without telangiectasia masquerading as benign hereditary chorea
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7 January 2021
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Human Rad50 is physically associated with human Mre11: identification of a conserved multiprotein complex implicated in recombinational DNA repair
1 reference
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7 January 2021
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Characterization of ATM gene mutations in 66 ataxia telangiectasia families.
1 reference
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Ataxia-telangiectasia: founder effect among north African Jews
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7 January 2021
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A new chromosomal instability disorder: the Nijmegen breakage syndrome
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7 January 2021
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A mammalian cell cycle checkpoint pathway utilizing p53 and GADD45 is defective in ataxia-telangiectasia
1 reference
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Crossref
reference URL
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7 January 2021
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Nijmegen breakage syndrome cells fail to induce the p53-mediated DNA damage response following exposure to ionizing radiation
1 reference
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reference URL
https://api.crossref.org/works/10.1016%2FS0955-0674%2800%2900091-0
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7 January 2021
based on heuristic
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p53 and ATM: cell cycle, cell death, and cancer.
1 reference
stated in
Crossref
reference URL
https://api.crossref.org/works/10.1016%2FS0955-0674%2800%2900091-0
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7 January 2021
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p53 in signaling checkpoint arrest or apoptosis
1 reference
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https://api.crossref.org/works/10.1016%2FS0955-0674%2800%2900091-0
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7 January 2021
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In situ visualization of DNA double-strand break repair in human fibroblasts
1 reference
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7 January 2021
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hMre11 and hRad50 nuclear foci are induced during the normal cellular response to DNA double-strand breaks
1 reference
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reference URL
https://api.crossref.org/works/10.1016%2FS0955-0674%2800%2900091-0
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7 January 2021
based on heuristic
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Hypersensitivity of cells from a new chromosomal-breakage syndrome to DNA-damaging agents
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https://api.crossref.org/works/10.1016%2FS0955-0674%2800%2900091-0
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7 January 2021
based on heuristic
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Immortalization and characterization of Nijmegen Breakage syndrome fibroblasts
1 reference
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reference URL
https://api.crossref.org/works/10.1016%2FS0955-0674%2800%2900091-0
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7 January 2021
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Cell cycle regulation in response to DNA damage in mammalian cells: a historical perspective
1 reference
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Crossref
reference URL
https://api.crossref.org/works/10.1016%2FS0955-0674%2800%2900091-0
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7 January 2021
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Identifiers
DOI
10.1016/S0955-0674(00)00091-0
1 reference
stated in
Europe PubMed Central
PubMed ID
10801460
retrieved
30 July 2017
PubMed ID
10801460
1 reference
stated in
Europe PubMed Central
PubMed ID
10801460
retrieved
30 July 2017
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