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Inducible growth arrest: new mechanistic insights
scientific article
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instance of
scholarly article
1 reference
stated in
Europe PubMed Central
PubMed ID
8134335
retrieved
6 August 2017
review article
1 reference
stated in
Europe PubMed Central
title
Inducible growth arrest: new mechanistic insights
(English)
1 reference
stated in
Europe PubMed Central
PubMed ID
8134335
retrieved
6 August 2017
author
David Ron
series ordinal
1
1 reference
stated in
Europe PubMed Central
PubMed ID
8134335
retrieved
6 August 2017
language of work or name
English
0 references
publication date
1 March 1994
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stated in
Europe PubMed Central
PubMed ID
8134335
retrieved
6 August 2017
published in
Proceedings of the National Academy of Sciences of the United States of America
1 reference
stated in
Europe PubMed Central
PubMed ID
8134335
retrieved
6 August 2017
volume
91
1 reference
stated in
Europe PubMed Central
PubMed ID
8134335
retrieved
6 August 2017
page(s)
1985-1986
1 reference
stated in
Europe PubMed Central
PubMed ID
8134335
retrieved
6 August 2017
issue
6
1 reference
stated in
Europe PubMed Central
PubMed ID
8134335
retrieved
6 August 2017
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A proteolytic fragment from the central region of p53 has marked sequence-specific DNA-binding activity when generated from wild-type but not from oncogenic mutant p53 protein
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Sequence-specific transcriptional activation is essential for growth suppression by p53.
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Fusion of the dominant negative transcription regulator CHOP with a novel gene FUS by translocation t(12;16) in malignant liposarcoma
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Post-Translational Regulation of the 54K Cellular Tumor Antigen in Normal and Transformed Cells
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Wild-type p53 can inhibit oncogene-mediated focus formation
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Negative growth regulation in a glioblastoma tumor cell line that conditionally expresses human wild-type p53.
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Participation of p53 protein in the cellular response to DNA damage
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Mice deficient for p53 are developmentally normal but susceptible to spontaneous tumours
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8 July 2018
Wild-type p53 restores cell cycle control and inhibits gene amplification in cells with mutant p53 alleles.
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8 July 2018
p53 function and dysfunction
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8 July 2018
Regulation of the specific DNA binding function of p53
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PubMed Central
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8 July 2018
A mammalian cell cycle checkpoint pathway utilizing p53 and GADD45 is defective in ataxia-telangiectasia
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Defects in a cell cycle checkpoint may be responsible for the genomic instability of cancer cells
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Growth arrest induced by wild-type p53 protein blocks cells prior to or near the restriction point in late G1 phase
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p53 domains: identification and characterization of two autonomous DNA-binding regions.
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27 September 2018
The DNA-binding domain of p53 contains the four conserved regions and the major mutation hot spots
1 reference
stated in
PubMed
reference URL
https://pubmed.ncbi.nlm.nih.gov/8134335
retrieved
12 December 2020
based on heuristic
inferred from PubMed ID database lookup
Activation of the gadd153 promoter by genotoxic agents: a rapid and specific response to DNA damage
1 reference
stated in
PubMed
reference URL
https://pubmed.ncbi.nlm.nih.gov/8134335
retrieved
12 December 2020
based on heuristic
inferred from PubMed ID database lookup
Altered cell cycle arrest and gene amplification potential accompany loss of wild-type p53
1 reference
stated in
PubMed
reference URL
https://pubmed.ncbi.nlm.nih.gov/8134335
retrieved
12 December 2020
based on heuristic
inferred from PubMed ID database lookup
Identifiers
DOI
10.1073/PNAS.91.6.1985
1 reference
stated in
Europe PubMed Central
PubMed ID
8134335
retrieved
6 August 2017
ADS bibcode
1994PNAS...91.1985R
0 references
PMCID
43292
1 reference
stated in
Europe PubMed Central
PubMed ID
8134335
retrieved
6 August 2017
PubMed ID
8134335
1 reference
stated in
Europe PubMed Central
PubMed ID
8134335
retrieved
6 August 2017
ResearchGate publication ID
15065589
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