(Q38338332)

English

A proximal element within the human alpha 2(I) collagen (COL1A2) promoter, distinct from the tumor necrosis factor-alpha response element, mediates transcriptional repression by interferon-gamma

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scholarly article

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Europe PubMed Central

2 September 2017

A proximal element within the human alpha 2(I) collagen (COL1A2) promoter, distinct from the tumor necrosis factor-alpha response element, mediates transcriptional repression by interferon-gamma (English)

1 reference

Europe PubMed Central

2 September 2017

author name string

Higashi K

1

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Europe PubMed Central

2 September 2017

Kouba DJ

2

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Europe PubMed Central

2 September 2017

Song YJ

3

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Europe PubMed Central

2 September 2017

Uitto J

4

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2 September 2017

Mauviel A

5

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2 September 2017

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1 March 1998

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2 September 2017

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Europe PubMed Central

2 September 2017

16

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2 September 2017

447-456

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Europe PubMed Central

2 September 2017

8

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Europe PubMed Central

2 September 2017

Regulation of collagen gene expression in cutaneous diseases with dermal fibrosis: evidence for pretranslational control

1 reference

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A rapid micropreparation technique for extraction of DNA-binding proteins from limiting numbers of mammalian cells

1 reference

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A single polypeptide possesses the binding and transcription activities of the adenovirus major late transcription factor

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An AP-1 binding sequence is essential for regulation of the human alpha2(I) collagen (COL1A2) promoter activity by transforming growth factor-beta.

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Eukaryotic gene transcription with purified components

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Gamma interferon is the lymphokine and beta interferon the monokine responsible for inhibition of fibroblast collagen production and late but not early fibroblast proliferation

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A new technique for the assay of infectivity of human adenovirus 5 DNA

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Recombinant genomes which express chloramphenicol acetyltransferase in mammalian cells

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Treatment of systemic sclerosis with gamma-interferon

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Interleukin-1 beta prevents the stimulatory effect of transforming growth factor-beta on collagen gene expression in human skin fibroblasts

1 reference

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Tumor necrosis factor-alpha and interferon-gamma suppress the activation of human type I collagen gene expression by transforming growth factor-beta 1. Evidence for two distinct mechanisms of inhibition at the transcriptional and posttranscriptional

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The transforming growth factor-beta family

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Matrix metalloproteinase gene expression

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Cytokine regulation of metalloproteinase gene expression

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Transcriptional interactions of transforming growth-factor-beta with pro-inflammatory cytokines

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Cell-specific induction of distinct oncogenes of the Jun family is responsible for differential regulation of collagenase gene expression by transforming growth factor-beta in fibroblasts and keratinocytes.

1 reference

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Comparative effects of interleukin-1 and tumor necrosis factor-alpha on collagen production and corresponding procollagen mRNA levels in human dermal fibroblasts

1 reference

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Evaluation of transforming growth factor beta and type I procollagen gene expression in fibrotic skin diseases by in situ hybridization

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Pharmacologic enhancement of wound healing

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Treatment of systemic sclerosis

1 reference

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Collagens: molecular biology, diseases, and potentials for therapy

1 reference

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Inhibition of excessive scleroderma fibroblast collagen production by recombinant gamma-interferon. Association with a coordinate decrease in types I and III procollagen messenger RNA levels

1 reference

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Synergistic effect of tumor necrosis factor-alpha and interferon-gamma on collagen synthesis of human skin fibroblasts in vitro

1 reference

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Identifiers

10.1016/S0945-053X(98)90016-6

1 reference

Europe PubMed Central

2 September 2017

1 reference

Europe PubMed Central

2 September 2017

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