(Q40120151)

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Downregulation of class I major histocompatibility complex surface expression by varicella-zoster virus involves open reading frame 66 protein kinase-dependent and -independent mechanisms.

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Europe PubMed Central

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https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:17567702%20AND%20SRC:MED&resulttype=core&format=json

31 December 2019

Downregulation of class I major histocompatibility complex surface expression by varicella-zoster virus involves open reading frame 66 protein kinase-dependent and -independent mechanisms (English)

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31 December 2019

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31 December 2019

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31 December 2019

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Amie J Eisfeld

1

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31 December 2019

Allison Abendroth

4

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31 December 2019

Paul R Kinchington

5

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13 June 2007

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31 December 2019

Journal of Virology

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31 December 2019

81

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31 December 2019

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31 December 2019

9034-9049

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31 December 2019

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Selective retention of herpes simplex virus-specific T cells in latently infected human trigeminal ganglia

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Varicella-zoster virus gene 66 transcription and translation in latently infected human Ganglia

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Virion association of IE62, the varicella-zoster virus (VZV) major transcriptional regulatory protein, requires expression of the VZV open reading frame 66 protein kinase

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Varicella-zoster virus retains major histocompatibility complex class I proteins in the Golgi compartment of infected cells

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Nuclear accumulation of IE62, the varicella-zoster virus (VZV) major transcriptional regulatory protein, is inhibited by phosphorylation mediated by the VZV open reading frame 66 protein kinase

1 reference

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Infection of human T lymphocytes with varicella-zoster virus: an analysis with viral mutants and clinical isolates

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1 reference

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1 reference

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https://pubmed.ncbi.nlm.nih.gov/17567702

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Identifiers

10.1128/JVI.00711-07

1 reference

Europe PubMed Central

PMC publication ID

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:17567702%20AND%20SRC:MED&resulttype=core&format=json

31 December 2019

PMC publication ID

1 reference

Europe PubMed Central

PMC publication ID

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:17567702%20AND%20SRC:MED&resulttype=core&format=json

31 December 2019

PubMed publication ID

1 reference

Europe PubMed Central

PMC publication ID

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:17567702%20AND%20SRC:MED&resulttype=core&format=json

31 December 2019

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