(Q40362124)

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Macrophage migration inhibitory factor promotes innate immune responses by suppressing glucocorticoid-induced expression of mitogen-activated protein kinase phosphatase-1.

scientific article published on December 2005

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Europe PubMed Central

PubMed publication ID

19 September 2017

Macrophage migration inhibitory factor promotes innate immune responses by suppressing glucocorticoid-induced expression of mitogen-activated protein kinase phosphatase-1. (English)

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Europe PubMed Central

PubMed publication ID

19 September 2017

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1

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Europe PubMed Central

PubMed publication ID

19 September 2017

Thierry Calandra

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Thierry Calandra

4

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Anne-Laure Chanson

2

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Europe PubMed Central

PubMed publication ID

19 September 2017

Marlies Knaup-Reymond

3

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Europe PubMed Central

PubMed publication ID

19 September 2017

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publication date

1 December 2005

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Europe PubMed Central

PubMed publication ID

19 September 2017

European Journal of Immunology

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Europe PubMed Central

PubMed publication ID

19 September 2017

35

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Europe PubMed Central

PubMed publication ID

19 September 2017

12

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Europe PubMed Central

PubMed publication ID

19 September 2017

3405-3413

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Europe PubMed Central

PubMed publication ID

19 September 2017

Innate immune recognition

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Association between tumour necrosis factor in serum and fatal outcome in patients with meningococcal disease

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Tumor necrosis factor and interleukin-1 in the serum of children with severe infectious purpura

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Neuroendocrine regulation of immunity

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THE INFLUENCE OF THE ADRENAL GLANDS ON RESISTANCE : II. THE TOXIC EFFECT OF KILLED BACTERIA IN ADRENALECTOMIZED RATS.

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Detection of circulating tumor necrosis factor after endotoxin administration.

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Cortisol response to corticotropin and survival in septic shock

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Effect of treatment with low doses of hydrocortisone and fludrocortisone on mortality in patients with septic shock

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MIF as a glucocorticoid-induced modulator of cytokine production

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Macrophage migration inhibitory factor: a regulator of innate immunity

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The macrophage is an important and previously unrecognized source of macrophage migration inhibitory factor

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Macrophage migration inhibitory factor is a critical mediator of the activation of immune cells by exotoxins of Gram-positive bacteria

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MIF regulates innate immune responses through modulation of Toll-like receptor 4

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Macrophage migration inhibitory factor (MIF) sustains macrophage proinflammatory function by inhibiting p53: regulatory role in the innate immune response

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Sustained mitogen-activated protein kinase (MAPK) and cytoplasmic phospholipase A2 activation by macrophage migration inhibitory factor (MIF). Regulatory role in cell proliferation and glucocorticoid action

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Intracellular action of the cytokine MIF to modulate AP-1 activity and the cell cycle through Jab1

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Dual specificity phosphatases: a gene family for control of MAP kinase function.

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Mitogen activated protein kinases as targets for development of novel anti-inflammatory drugs

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MAP kinase phosphatases

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The growth factor-inducible immediate-early gene 3CH134 encodes a protein-tyrosine-phosphatase

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MKP-1 (3CH134), an immediate early gene product, is a dual specificity phosphatase that dephosphorylates MAP kinase in vivo

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Restraint of proinflammatory cytokine biosynthesis by mitogen-activated protein kinase phosphatase-1 in lipopolysaccharide-stimulated macrophages

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Glucocorticoids induce rapid up-regulation of mitogen-activated protein kinase phosphatase-1 and dephosphorylation of extracellular signal-regulated kinase and impair proliferation in human and mouse osteoblast cell lines

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Glucocorticoids inhibit MAP kinase via increased expression and decreased degradation of MKP-1

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Dexamethasone causes sustained expression of mitogen-activated protein kinase (MAPK) phosphatase 1 and phosphatase-mediated inhibition of MAPK p38.

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Macrophage migration inhibitory factor and hypothalamo-pituitary-adrenal function during critical illness

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Protection from septic shock by neutralization of macrophage migration inhibitory factor

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Toll-like receptor signalling

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Inferences, questions and possibilities in Toll-like receptor signalling

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Defective LPS Signaling in C3H/HeJ and C57BL/10ScCr Mice: Mutations in Tlr4 Gene

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NF-kappaB regulation in the immune system

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Molecular mechanisms of glucocorticoid action and resistance

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Post-transcriptional regulation of gene expression by degradation of messenger RNAs

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Regulation of cyclooxygenase 2 mRNA stability by the mitogen-activated protein kinase p38 signaling cascade

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Purification, bioactivity, and secondary structure analysis of mouse and human macrophage migration inhibitory factor (MIF).

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Identifiers

10.1002/EJI.200535413

1 reference

Europe PubMed Central

PubMed publication ID

19 September 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

19 September 2017

ResearchGate publication ID

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