(Q40654522)

22 September 2017

title

Postnatal brain development and neural cell differentiation modulate mitochondrial Bax and BH3 peptide-induced cytochrome c release (English)

1 reference

22 September 2017

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Polster BM

1

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22 September 2017

Robertson CL

2

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22 September 2017

Bucci CJ

3

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22 September 2017

Suzuki M

4

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22 September 2017

Fiskum G

5

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22 September 2017

publication date

1 March 2003

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Cell Death & Differentiation

22 September 2017

published in

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22 September 2017

volume

10

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22 September 2017

issue

3

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22 September 2017

page(s)

365-370

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Bcl-2 family members in the development and degenerative pathologies of the nervous system

22 September 2017

cites work

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401158

BAX is required for neuronal death after trophic factor deprivation and during development

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401158

Bax deletion further orders the cell death pathway in cerebellar granule cells and suggests a caspase-independent pathway to cell death.

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401158

The combined functions of proapoptotic Bcl-2 family members bak and bax are essential for normal development of multiple tissues

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401158

Endogenous bax translocation in SH-SY5Y human neuroblastoma cells and cerebellar granule neurons undergoing apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401158

Molecular cloning of a novel polypeptide, DP5, induced during programmed neuronal death

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401158

Dominant-negative c-Jun promotes neuronal survival by reducing BIM expression and inhibiting mitochondrial cytochrome c release

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401158

Induction of BIM, a proapoptotic BH3-only BCL-2 family member, is critical for neuronal apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401158

Establishment of a noradrenergic clonal line of rat adrenal pheochromocytoma cells which respond to nerve growth factor

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401158

Nerve growth factor-induced neuronal differentiation is accompanied by differential induction and localization of the amyloid precursor protein (APP) in PC12 cells and variant PC12S cells

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401158

Bax translocation to mitochondria subsequent to a rapid loss of mitochondrial membrane potential

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401158

Proapoptotic BAX and BAK: a requisite gateway to mitochondrial dysfunction and death

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401158

Intrinsic and extrinsic pathway signaling during neuronal apoptosis: lessons from the analysis of mutant mice

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401158

Mechanisms of delayed cell death following hypoxic-ischemic injury in the immature rat: evidence for apoptosis during selective neuronal loss

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401158

Nuclear condensation and fragmentation following cerebral hypoxia-ischemia occurs more frequently in immature than older rats

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401158

Impairment of mitochondrial respiration after cerebral hypoxia-ischemia in immature rats: relationship to activation of caspase-3 and neuronal injury

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401158

Involvement of caspase-3 in cell death after hypoxia-ischemia declines during brain maturation

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401158

Apoptotic neurodegeneration following trauma is markedly enhanced in the immature brain

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401158

N-Methyl-D-aspartate antagonists and apoptotic cell death triggered by head trauma in developing rat brain

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401158

Cerebral ischemia and reperfusion: prevention of brain mitochondrial injury by lidoflazine

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401158

Isolation of mitochondria from ascites tumor cells permeabilized with digitonin.

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401158

Targeting Peptides Transiently Block a Mitochondrial Channel

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401158

Structure of Bax: coregulation of dimer formation and intracellular localization

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401158

Mitochondrial precursor signal peptide induces a unique permeability transition and release of cytochrome c from liver and brain mitochondria

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401158

Programmed cell death in neurons: focus on the pathway of nerve growth factor deprivation-induced death of sympathetic neurons.

21 January 2018

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401158

7 January 2021

Widespread elimination of naturally occurring neuronal death in Bax-deficient mice

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401158

7 January 2021

BAX translocation is a critical event in neuronal apoptosis: regulation by neuroprotectants, BCL-2, and caspases

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401158

7 January 2021

Apoptosis has a prolonged role in the neurodegeneration after hypoxic ischemia in the newborn rat.

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401158

7 January 2021

Delayed neurodegeneration in neonatal rat thalamus after hypoxia-ischemia is apoptosis.

1 reference

https://api.crossref.org/works/10.1038%2FSJ.CDD.4401158

7 January 2021

Identifiers

DOI

10.1038/SJ.CDD.4401158

1 reference

Dimensions Publication ID

22 September 2017

1009442292

0 references

Fatcat ID

release_7ollcfkyvvcn5orisuswoiv2rq

1 reference

Fatcat

https://api.fatcat.wiki/v0/release/7ollcfkyvvcn5orisuswoiv2rq

24 November 2022

mapped directly with Wikidata item

1 reference