(Q40876072)

24 September 2017

Growth factors inactivate the cell death promoter BAD by phosphorylation of its BH3 domain on Ser155. (English)

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24 September 2017

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4

Lutz RJ

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24 September 2017

5

Chittenden T

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24 September 2017

Zhou XM

1

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24 September 2017

Liu Y

series ordinal

2

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24 September 2017

Payne G

series ordinal

3

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24 September 2017

language of work or name

English

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publication date

1 August 2000

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Journal of Biological Chemistry

24 September 2017

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24 September 2017

275

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24 September 2017

25046-25051

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24 September 2017

32

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BCL-2 family members and the mitochondria in apoptosis

24 September 2017

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M002526200

The C. elegans protein EGL-1 is required for programmed cell death and interacts with the Bcl-2-like protein CED-9

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M002526200

Cleavage of BID by caspase 8 mediates the mitochondrial damage in the Fas pathway of apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M002526200

Bid, a Bcl2 interacting protein, mediates cytochrome c release from mitochondria in response to activation of cell surface death receptors

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M002526200

Bad, a heterodimeric partner for Bcl-XL and Bcl-2, displaces Bax and promotes cell death

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M002526200

Serine phosphorylation of death agonist BAD in response to survival factor results in binding to 14-3-3 not BCL-X(L)

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M002526200

Akt phosphorylation of BAD couples survival signals to the cell-intrinsic death machinery

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M002526200

Interleukin-3-induced phosphorylation of BAD through the protein kinase Akt

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M002526200

Cell survival promoted by the Ras-MAPK signaling pathway by transcription-dependent and -independent mechanisms

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M002526200

Bad is a BH3 domain-containing protein that forms an inactivating dimer with Bcl-XL.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M002526200

Dimerization properties of human BAD. Identification of a BH-3 domain and analysis of its binding to mutant BCL-2 and BCL-XL proteins

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M002526200

BH3 domain of BAD is required for heterodimerization with BCL-XL and pro-apoptotic activity

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M002526200

Protein kinase recognition sequence motifs

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M002526200

Structure of Bcl-xL-Bak peptide complex: recognition between regulators of apoptosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M002526200

Type I cAMP-dependent protein kinase delays apoptosis in human neutrophils at a site upstream of caspase-3.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M002526200

Fas/APO-1(CD95)-induced apoptosis of primary hepatocytes is inhibited by cAMP.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M002526200

Akt-dependent and -independent survival signaling pathways utilized by insulin-like growth factor I.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M002526200

Differential effects of protein kinase a sub-units on chinese-hamster-ovary cell cycle and proliferation

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M002526200

21 January 2018

Identifiers

DOI

10.1074/JBC.M002526200

1 reference

24 September 2017

1 reference