(Q41146738)

27 September 2017

Release of preformed Fas ligand in soluble form is the major factor for activation-induced death of Jurkat T cells (English)

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27 September 2017

Martínez-Lorenzo MJ

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27 September 2017

Alava MA

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27 September 2017

Anel A

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27 September 2017

Piñeiro A

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27 September 2017

Naval J

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27 September 2017

language of work or name

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1 December 1996

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27 September 2017

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27 September 2017

89

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27 September 2017

511-517

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27 September 2017

4

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Transferrin receptors recycle to cis and middle as well as trans Golgi cisternae in Ig-secreting myeloma cells

27 September 2017

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12 December 2020

Role of oxidative damage and IL-1 beta-converting enzyme-like proteases in Fas-based cytotoxicity exerted by effector T cells

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12 December 2020

Cytolysis by Ca-permeable transmembrane channels. Pore formation causes extensive DNA degradation and cell lysis

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12 December 2020

A cell-killing monoclonal antibody (anti-Fas) to a cell surface antigen co-downregulated with the receptor of tumor necrosis factor

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12 December 2020

Lymphoproliferation disorder in mice explained by defects in Fas antigen that mediates apoptosis

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12 December 2020

Increased intracellular cyclic AMP inhibits inositol phospholipid hydrolysis induced by perturbation of the T cell receptor/CD3 complex but not by G-protein stimulation. Association with protein kinase A-mediated phosphorylation of phospholipase C-g

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12 December 2020

Induction of apoptosis by monoclonal antibody anti-APO-1 class switch variants is dependent on cross-linking of APO-1 cell surface antigens

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12 December 2020

Fas involvement in Ca(2+)-independent T cell-mediated cytotoxicity

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12 December 2020

Mature T cells of autoimmune lpr/lpr mice have a defect in antigen-stimulated suicide

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12 December 2020

Molecular cloning and expression of the Fas ligand, a novel member of the tumor necrosis factor family

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12 December 2020

Purification and characterization of the Fas-ligand that induces apoptosis

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12 December 2020

Generalized lymphoproliferative disease in mice, caused by a point mutation in the Fas ligand

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12 December 2020

Fas-based lymphocyte-mediated cytotoxicity against syngeneic activated lymphocytes: a regulatory pathway?

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12 December 2020

T cell receptor-induced Fas ligand expression in cytotoxic T lymphocyte clones is blocked by protein tyrosine kinase inhibitors and cyclosporin A

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12 December 2020

Fas ligand mediates activation-induced cell death in human T lymphocytes

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12 December 2020

Autocrine T-cell suicide mediated by APO-1/(Fas/CD95)

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12 December 2020

Cell-autonomous Fas (CD95)/Fas-ligand interaction mediates activation-induced apoptosis in T-cell hybridomas.

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12 December 2020

Fas(CD95)/FasL interactions required for programmed cell death after T-cell activation.

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12 December 2020

TCR/CD3 coupling to Fas-based cytotoxicity

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12 December 2020

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12 December 2020

The fas antigen is involved in peripheral but not thymic deletion of T lymphocytes in T cell receptor transgenic mice

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12 December 2020

The role of APO-1-mediated apoptosis in the immune system

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12 December 2020

Expression of the Fas ligand in cells of T cell lineage.

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12 December 2020

Expression of the functional soluble form of human fas ligand in activated lymphocytes.

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12 December 2020

A soluble form of TRAP (CD40 ligand) is rapidly released after T cell activation

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12 December 2020

Regulation of cell surface APO-1/Fas (CD95) ligand expression by metalloproteases

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12 December 2020

Interaction of peptides derived from the Fas ligand with the Fyn-SH3 domain

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12 December 2020

Activation-induced death of mature T cells in the regulation of immune responses

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12 December 2020

Fas Ligand-Induced Apoptosis as a Mechanism of Immune Privilege

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12 December 2020

Metalloproteinase-mediated release of human Fas ligand

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12 December 2020

Fas and FasL in the homeostatic regulation of immune responses

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12 December 2020

Fas ligand in human serum

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12 December 2020

Feasibility of drug screening with panels of human tumor cell lines using a microculture tetrazolium assay

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12 December 2020

10.1046/J.1365-2567.1996.D01-782.X

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DOI

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27 September 2017

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