(Q42816921)

English

Genistein induces oestrogen receptor-α gene expression in osteoblasts through the activation of mitogen-activated protein kinases/NF-κB/activator protein-1 and promotes cell mineralisation

scientific article published on 5 July 2013

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Europe PubMed Central

PubMed publication ID

7 November 2017

Genistein induces oestrogen receptor-α gene expression in osteoblasts through the activation of mitogen-activated protein kinases/NF-κB/activator protein-1 and promotes cell mineralisation (English)

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Europe PubMed Central

PubMed publication ID

7 November 2017

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author name string

Mei-Hsiu Liao

1

1 reference

Europe PubMed Central

PubMed publication ID

7 November 2017

Yu-Ting Tai

2

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Europe PubMed Central

PubMed publication ID

7 November 2017

Yih-Giun Cherng

3

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Europe PubMed Central

PubMed publication ID

7 November 2017

Shing-Hwa Liu

4

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Europe PubMed Central

PubMed publication ID

7 November 2017

Ya-An Chang

5

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7 November 2017

Pei-I Lin

6

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7 November 2017

Ruei-Ming Chen

7

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PubMed publication ID

7 November 2017

publication date

5 July 2013

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7 November 2017

British Journal of Nutrition

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7 November 2017

111

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PubMed publication ID

7 November 2017

1

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PubMed publication ID

7 November 2017

55-63

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Europe PubMed Central

PubMed publication ID

7 November 2017

Estrogen action and cytoplasmic signaling pathways. Part II: the role of growth factors and phosphorylation in estrogen signaling

1 reference

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The protein kinase complement of the human genome

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Drynaria fortunei J. Sm. promotes osteoblast maturation by inducing differentiation-related gene expression and protecting against oxidative stress-induced apoptotic insults.

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Honokiol traverses the blood-brain barrier and induces apoptosis of neuroblastoma cells via an intrinsic bax-mitochondrion-cytochrome c-caspase protease pathway

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Genistein stimulates osteoblastic differentiation via p38 MAPK-Cbfa1 pathway in bone marrow culture

1 reference

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The Phytoestrogen Genistein Enhances Osteogenesis and Represses Adipogenic Differentiation of Human Primary Bone Marrow Stromal Cells

1 reference

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Genistein stimulates the osteoblastic differentiation via NO/cGMP in bone marrow culture

1 reference

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Anatomy of the estrogen response element

1 reference

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Multiple transcription factor elements collaborate with estrogen receptor alpha to activate an inducible estrogen response element in the NKG2E gene

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A functional androgen receptor is not sufficient to allow estradiol to protect bone after gonadectomy in estradiol receptor-deficient mice

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21 January 2018

International Union of Pharmacology. LXIV. Estrogen receptors

1 reference

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Molecular effects of genistein on estrogen receptor mediated pathways

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Soy phytoestrogens: impact on postmenopausal bone loss and mechanisms of action.

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Genistein, daidzein, and their .beta.-glycoside conjugates: antitumor isoflavones in soybean foods from American and Asian diets

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Pharmaceutical prospects of phytoestrogens

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KCNJ5 mutations in the National Institutes of Health cohort of patients with primary hyperaldosteronism: an infrequent genetic cause of Conn's syndrome

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Birth and Death of Bone Cells: Basic Regulatory Mechanisms and Implications for the Pathogenesis and Treatment of Osteoporosis 1

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Direct transcriptional targets of sex steroid hormones in bone.

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Skeletal effects of estrogens

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AP-1--The Jun proteins: Oncogenes or tumor suppressors in disguise?

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Extracellular matrix protein mediated regulation of the osteoblast differentiation of bone marrow derived human mesenchymal stem cells

1 reference

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7 January 2021

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inferred from DOI database lookup

Hormone response in ovarian cancer: time to reconsider as a clinical target?

1 reference

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7 January 2021

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Osteoblast-related gene expression of bone marrow cells during the osteoblastic differentiation induced by type I collagen

1 reference

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7 January 2021

based on heuristic

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Identifiers

10.1017/S0007114513002043

1 reference

Europe PubMed Central

PubMed publication ID

7 November 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

7 November 2017

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