(Q44020107)

29 November 2017

Mitochondrial death protein Nix is induced in cardiac hypertrophy and triggers apoptotic cardiomyopathy (English)

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29 November 2017

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7

Bruce J Aronow

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29 November 2017

Martin G Yussman

1

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29 November 2017

Tsuyoshi Toyokawa

2

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29 November 2017

Amy Odley

3

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29 November 2017

Roy A Lynch

4

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29 November 2017

Guangyu Wu

5

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29 November 2017

Melissa C Colbert

6

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29 November 2017

John N Lorenz

8

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29 November 2017

Gerald W Dorn

9

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29 November 2017

language of work or name

English

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publication date

10 June 2002

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29 November 2017

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29 November 2017

8

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29 November 2017

725-730

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29 November 2017

7

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https://scigraph.springernature.com/pub.10.1038/nm719

29 November 2017

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cites work

Death by design. Programmed cell death in cardiovascular biology and disease

1 reference

Enhanced Galphaq signaling: a common pathway mediates cardiac hypertrophy and apoptotic heart failure

21 January 2018

1 reference

Loss of a gp130 cardiac muscle cell survival pathway is a critical event in the onset of heart failure during biomechanical stress

21 January 2018

1 reference

Prognostic implications of echocardiographically determined left ventricular mass in the Framingham Heart Study

21 January 2018

1 reference

Molecular advances in cardiovascular biology

21 January 2018

1 reference

Apoptosis and heart failure

21 January 2018

1 reference

21 January 2018

Caspases: enemies within

1 reference

Genetic dissection of cardiac growth control pathways

21 January 2018

1 reference

Transgenic Galphaq overexpression induces cardiac contractile failure in mice

21 January 2018

1 reference

Embryonic cardiomyocyte hypoplasia and craniofacial defects in G alpha q/G alpha 11-mutant mice

21 January 2018

1 reference

Calcineurin-mediated hypertrophy protects cardiomyocytes from apoptosis in vitro and in vivo: An apoptosis-independent model of dilated heart failure

21 January 2018

1 reference

Nix and Nip3 form a subfamily of pro-apoptotic mitochondrial proteins

21 January 2018

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Cardiomyocyte apoptosis induced by Galphaq signaling is mediated by permeability transition pore formation and activation of the mitochondrial death pathway.

21 January 2018

1 reference

A structural view of mitochondria-mediated apoptosis

21 January 2018

1 reference

BNIP3 heterodimerizes with Bcl-2/Bcl-X(L) and induces cell death independent of a Bcl-2 homology 3 (BH3) domain at both mitochondrial and nonmitochondrial sites

21 January 2018

1 reference

The E1B 19K/Bcl-2-binding protein Nip3 is a dimeric mitochondrial protein that activates apoptosis

21 January 2018

1 reference

Left Ventricular Stresses in the Intact Human Heart

21 January 2018

1 reference

Cardiomyopathy of overload. A major determinant of prognosis in congestive heart failure

21 January 2018

1 reference

Stretch-mediated release of angiotensin II induces myocyte apoptosis by activating p53 that enhances the local renin-angiotensin system and decreases the Bcl-2-to-Bax protein ratio in the cell

21 January 2018

1 reference

A mouse model of familial hypertrophic cardiomyopathy

21 January 2018

1 reference

Neonatal cardiomyopathy in mice homozygous for the Arg403Gln mutation in the alpha cardiac myosin heavy chain gene

21 January 2018

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Expression of the gene encoding the proapoptotic Nip3 protein is induced by hypoxia

21 January 2018

1 reference

Evidence of Apoptosis in Arrhythmogenic Right Ventricular Dysplasia

21 January 2018

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Apoptosis in myocytes in end-stage heart failure

21 January 2018

1 reference

7 January 2021

Apoptosis in the failing human heart

1 reference

7 January 2021

Chips ahoy: gene expression in failing hearts surveyed by high-density microarrays

1 reference

7 January 2021

Targeting the receptor-Gq interface to inhibit in vivo pressure overload myocardial hypertrophy

1 reference

7 January 2021

Absence of pressure overload induced myocardial hypertrophy after conditional inactivation of Gαq/Gα11 in cardiomyocytes

1 reference

7 January 2021

Decompensation of pressure-overload hypertrophy in G alpha q-overexpressing mice

1 reference

7 January 2021

Divergent transcriptional responses to independent genetic causes of cardiac hypertrophy

1 reference

7 January 2021

TAK1 is activated in the myocardium after pressure overload and is sufficient to provoke heart failure in transgenic mice.

1 reference

7 January 2021

Regulation of bcl-2 family proteins during development and in response to oxidative stress in cardiac myocytes: association with changes in mitochondrial membrane potential

1 reference

7 January 2021

Expression of bcl-2 protein, an inhibitor of apoptosis, and Bax, an accelerator of apoptosis, in ventricular myocytes of human hearts with myocardial infarction

1 reference

7 January 2021

Dimensions Publication ID

Identifiers

DOI

10.1038/NM719

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