(Q52537703)

2 May 2018

title

Targeted disruption in murine cells reveals variable requirement for Smad4 in transforming growth factor beta-related signaling. (English)

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2 May 2018

author

Pamela A. Hoodless

5

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2 May 2018

author name string

Sirard C

1

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2 May 2018

Kim S

2

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2 May 2018

Mirtsos C

3

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2 May 2018

Tadich P

4

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2 May 2018

Itié A

6

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2 May 2018

Maxson R

7

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2 May 2018

Wrana JL

8

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2 May 2018

Mak TW

9

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2 May 2018

language of work or name

English

0 references

publication date

1 January 2000

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2 May 2018

Journal of Biological Chemistry

number of pages

8

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based on heuristic

inferred from page(s)

published in

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2 May 2018

volume

275

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2 May 2018

page(s)

2063-2070

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2 May 2018

issue

3

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2 May 2018

TGF-beta receptors and actions

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Smad4/DPC4 and Smad3 mediate transforming growth factor-beta (TGF-beta) signaling through direct binding to a novel TGF-beta-responsive element in the human plasminogen activator inhibitor-1 promoter

21 January 2018

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The tumor suppressor SMAD4/DPC4 is essential for epiblast proliferation and mesoderm induction in mice

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Targeted deletion of Smad4 shows it is required for transforming growth factor beta and activin signaling in colorectal cancer cells

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Targeted disruption of Smad3 reveals an essential role in transforming growth factor beta-mediated signal transduction

21 January 2018

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Identification of two Smad4 proteins in Xenopus. Their common and distinct properties.

21 January 2018

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21 January 2018

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21 January 2018

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21 January 2018

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21 January 2018

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21 January 2018

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21 January 2018

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21 January 2018

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21 January 2018

Identifiers

DOI

10.1074/JBC.275.3.2063

1 reference

2 May 2018

PubMed ID

10636910

1 reference

2 May 2018