(Q53670078)

17 May 2018

title

Role of mitogen-activated protein kinases in inducible nitric oxide synthase and TNFalpha expression in human fetal astrocytes. (English)

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17 May 2018

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6

Herbert B Tanowitz

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17 May 2018

Celia F. Brosnan

7

object named as

Celia F Brosnan

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17 May 2018

author name string

Liwei L Hua

1

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17 May 2018

Meng-Liang Zhao

2

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17 May 2018

Melissa Cosenza

3

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17 May 2018

Mee-Ohk Kim

4

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17 May 2018

Huan Huang

5

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17 May 2018

Sunhee C Lee

8

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17 May 2018

publication date

1 May 2002

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17 May 2018

Journal of Neuroimmunology

published in

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17 May 2018

volume

126

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17 May 2018

page(s)

180-189

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17 May 2018

issue

1-2

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17 May 2018

Cytokine actions in the central nervous system

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Extracellular signal-regulated kinase and p38 subgroups of mitogen-activated protein kinases regulate inducible nitric oxide synthase and tumor necrosis factor-alpha gene expression in endotoxin-stimulated primary glial cultures

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Analysis of the cytokine-stimulated human inducible nitric oxide synthase (iNOS) gene: characterization of differences between human and mouse iNOS promoters.

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How MAP kinases are regulated

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Blockade of p38 mitogen-activated protein kinase pathway inhibits inducible nitric-oxide synthase expression in mouse astrocytes

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MAPKs: new JNK expands the group

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Both p38alpha(MAPK) and JNK/SAPK pathways are important for induction of nitric-oxide synthase by interleukin-1beta in rat glomerular mesangial cells

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Selective inhibition of human glial inducible nitric oxide synthase by interferon-beta: implications for multiple sclerosis

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IL-1beta differentially regulates calcium wave propagation between primary human fetal astrocytes via pathways involving P2 receptors and gap junction channels.

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p38 map kinase regulates TNF-alpha production in human astrocytes and microglia by multiple mechanisms

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7 January 2021

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7 January 2021

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Identifiers

DOI

1 reference

17 May 2018

PubMed ID

12020969

1 reference

17 May 2018