(Q64885031)

30 June 2019

title

Correlation between CD11b/CD18 and increase of aggregability of granulocytes in coronary artery disease. (English)

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30 June 2019

author name string

A Mazzone

1

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30 June 2019

D Pasotti

2

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30 June 2019

S De Servi

3

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30 June 2019

G Fossati

4

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30 June 2019

I Hazzucchelli

5

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30 June 2019

P Cavigliano

6

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30 June 2019

G Ricevuti

7

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30 June 2019

publication date

1 August 1992

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30 June 2019

published in

Inflammation

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30 June 2019

volume

16

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30 June 2019

issue

4

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30 June 2019

page(s)

315-323

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30 June 2019

Increased neutrophil aggregability in coronary artery disease

cites work

1 reference

12 December 2020

Activated neutrophils release mediators that may contribute to myocardial injury and dysfunction associated with ischemia and reperfusion

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12 December 2020

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12 December 2020

New trends in coronary artery disease: the role of granulocyte activation

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Granulocyte activation after coronary angioplasty in humans

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Neutrophil function in ischemic heart disease

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Granulocyte aggregation as a manifestation of membrane interactions with complement: possible role in leukocyte margination, microvascular occlusion, and endothelial damage

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Neutrophil adherence receptors (CD 18) in ischemia. Dissociation between quantitative cell surface expression and diapedesis mediated by leukotriene B4

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Tissue destruction by neutrophils

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Endothelial and myocardial injury during ischemia and reperfusion: pathogenesis and therapeutic implications

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Cholesterol and atheroma lipids activate complement and stimulate granulocytes. A possible mechanism for amplification of ischemic injury in atherosclerotic states

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Interaction between morphine and granulocyte aggregation in myocardial ischaemia

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Complement-induced granulocyte aggregation in vivo.

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Identifiers

DOI

10.1007/BF00917624

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30 June 2019

Dimensions Publication ID

1031952538

0 references

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30 June 2019