(Q24294541)

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Suppression of NF-kappaB-dependent gene expression by a hexamethylene bisacetamide-inducible protein HEXIM1 in human vascular smooth muscle cells

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scholarly article

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Suppression of NF-kappaB-dependent gene expression by a hexamethylene bisacetamide-inducible protein HEXIM1 in human vascular smooth muscle cells (English)

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HEXIM P-TEFb complex subunit 1

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GOA release 2020-03-11

author name string

Rika Ouchida

1

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Masatoshi Kusuhara

2

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Noriaki Shimizu

3

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Tetsuya Hisada

4

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Yuichi Makino

5

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Chikao Morimoto

6

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Hiroshi Handa

7

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Fumitaka Ohsuzu

8

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Hirotoshi Tanaka

9

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language of work or name

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publication date

February 2003

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8

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95-107

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2

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The p65 (RelA) subunit of NF-kappaB interacts with the histone deacetylase (HDAC) corepressors HDAC1 and HDAC2 to negatively regulate gene expression

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Deficiency of T2K leads to apoptotic liver degeneration and impaired NF-kappaB-dependent gene transcription

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Duration of nuclear NF-kappaB action regulated by reversible acetylation

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Requirement for glycogen synthase kinase-3beta in cell survival and NF-kappaB activation

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Structure, expression, and functional characterization of the mouse CLP-1 gene

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Silencing mediator of retinoic acid and thyroid hormone receptors, as a novel transcriptional corepressor molecule of activating protein-1, nuclear factor-kappaB, and serum response factor

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Steroid receptor coactivator-1 interacts with the p50 subunit and coactivates nuclear factor kappaB-mediated transactivations

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Atherosclerosis — An Inflammatory Disease

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Inhibition of nuclear factor-kappaB-mediated transcription by association with the amino-terminal enhancer of split, a Groucho-related protein lacking WD40 repeats

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The phosphorylation status of nuclear NF-kappa B determines its association with CBP/p300 or HDAC-1

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Transport into and out of the nucleus

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The nuclear factor kappa-B signaling pathway participates in dysregulation of vascular smooth muscle cells in vitro and in human atherosclerosis

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Modulation of cell injury and survival by high glucose and advancing age.

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Enhancement of nuclear factor-kappa B acetylation by coactivator p300 and HIV-1 Tat proteins.

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Missing pieces in the NF-kappaB puzzle

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Hexamethylenebisacetamide selectively inhibits the proliferation of human and rat vascular smooth-muscle cells

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Troglitazone upregulates nitric oxide synthesis in vascular smooth muscle cells

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Glucocorticoid receptor recruitment of histone deacetylase 2 inhibits interleukin-1beta-induced histone H4 acetylation on lysines 8 and 12.

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Recruitment of CBP/p300 by the IFN beta enhanceosome is required for synergistic activation of transcription

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Functional modulation of the glucocorticoid receptor and suppression of NF-kappaB-dependent transcription by ursodeoxycholic acid

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Cytokine receptor common beta subunit-mediated STAT5 activation confers NF-kappa B activation in murine proB cell line Ba/F3 cells

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Regulation of NF-kappaB by cyclin-dependent kinases associated with the p300 coactivator

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The intima. Soil for atherosclerosis and restenosis.

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Transcriptional activation by NF-kappaB requires multiple coactivators

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High-performance affinity beads for identifying drug receptors

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Contribution of sustained Ca2+ elevation for nitric oxide production in endothelial cells and subsequent modulation of Ca2+ transient in vascular smooth muscle cells in coculture

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beta-very low density lipoprotein enhances inducible nitric oxide synthase expression in cytokine-stimulated vascular smooth muscle cells

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The nuclear factor-kappaB engages CBP/p300 and histone acetyltransferase activity for transcriptional activation of the interleukin-6 gene promoter.

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NF-kappaB is required for TNF-alpha-directed smooth muscle cell migration

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Antisense oligonucleotides to the p65 subunit of NF-kB inhibit human vascular smooth muscle cell adherence and proliferation and prevent neointima formation in rat carotid arteries

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HMG-CoA reductase inhibition by atorvastatin reduces neointimal inflammation in a rabbit model of atherosclerosis

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Glycated serum albumin-induced nitric oxide production in vascular smooth muscle cells by nuclear factor kappaB-dependent transcriptional activation of inducible nitric oxide synthase

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Antioxidants differentially affect nuclear factor kappa B-mediated nitric oxide synthase expression in vascular smooth muscle cells

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The NF-kappaB and IkappaB system in injured arteries

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Involvement of NF-kappa B activation in thrombin-induced human vascular smooth muscle cell proliferation

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An IkappaB-alpha mutant inhibits cytokine gene expression and proliferation in human vascular smooth muscle cells

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Phosphorylation of NF-kappa B p65 by PKA stimulates transcriptional activity by promoting a novel bivalent interaction with the coactivator CBP/p300

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Nitric oxide attenuates vascular smooth muscle cell activation by interferon-gamma. The role of constitutive NF-kappa B activity

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Expression of a constitutive NF-kappa B-like activity is essential for proliferation of cultured bovine vascular smooth muscle cells

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