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GRIM-19 associates with the serine protease HtrA2 for promoting cell death
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instance of
scholarly article
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title
GRIM-19 associates with the serine protease HtrA2 for promoting cell death
(English)
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main subject
suppression by virus of host protein-protein interaction
1 reference
stated in
Gene Ontology release 2020-05-02
Gene Ontology ID
GO:0036472
HtrA serine peptidase 2
1 reference
stated in
GOA release 2020-03-11
NADH:ubiquinone oxidoreductase subunit A13
1 reference
stated in
GOA release 2020-03-11
cellular response to retinoic acid
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stated in
GOA release 2020-03-11
cell death
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based on heuristic
inferred from title
author name string
X Ma
series ordinal
1
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S Kalakonda
series ordinal
2
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S M Srinivasula
series ordinal
3
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S P Reddy
series ordinal
4
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L C Platanias
series ordinal
5
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D V Kalvakolanu
series ordinal
6
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language of work or name
English
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publication date
19 July 2007
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published in
Oncogene
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volume
26
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issue
33
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page(s)
4842-9
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cites work
The promise of retinoids to fight against cancer.
1 reference
stated in
Crossref
reference URL
https://api.crossref.org/works/10.1038%2FSJ.ONC.1210287
retrieved
7 January 2021
based on heuristic
inferred from DOI database lookup
Identification of GRIM-19, a novel cell death-regulatory gene induced by the interferon-beta and retinoic acid combination, using a genetic approach
1 reference
stated in
Crossref
reference URL
https://api.crossref.org/works/10.1038%2FSJ.ONC.1210287
retrieved
7 January 2021
based on heuristic
inferred from DOI database lookup
Rapid activation by interferon alpha of a latent DNA-binding protein present in the cytoplasm of untreated cells
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stated in
Crossref
reference URL
https://api.crossref.org/works/10.1038%2FSJ.ONC.1210287
retrieved
7 January 2021
based on heuristic
inferred from DOI database lookup
GRIM-19, a cell death regulatory gene product, is a subunit of bovine mitochondrial NADH:ubiquinone oxidoreductase (complex I)
1 reference
stated in
Crossref
reference URL
https://api.crossref.org/works/10.1038%2FSJ.ONC.1210287
retrieved
7 January 2021
based on heuristic
inferred from DOI database lookup
KSHV ORF K9 (vIRF) is an oncogene which inhibits the interferon signaling pathway
1 reference
stated in
Crossref
reference URL
https://api.crossref.org/works/10.1038%2FSJ.ONC.1210287
retrieved
7 January 2021
based on heuristic
inferred from DOI database lookup
Endogenous type I interferons as a defense against tumors.
1 reference
stated in
Crossref
reference URL
https://api.crossref.org/works/10.1038%2FSJ.ONC.1210287
retrieved
7 January 2021
based on heuristic
inferred from DOI database lookup
The C-terminal tail of presenilin regulates Omi/HtrA2 protease activity
1 reference
stated in
Crossref
reference URL
https://api.crossref.org/works/10.1038%2FSJ.ONC.1210287
retrieved
7 January 2021
based on heuristic
inferred from DOI database lookup
Identification of Omi/HtrA2 as a mitochondrial apoptotic serine protease that disrupts inhibitor of apoptosis protein-caspase interaction
1 reference
stated in
Crossref
reference URL
https://api.crossref.org/works/10.1038%2FSJ.ONC.1210287
retrieved
7 January 2021
based on heuristic
inferred from DOI database lookup
Characterization of monoclonal antibodies against GRIM-19, a novel IFN-beta and retinoic acid-activated regulator of cell death
1 reference
stated in
Crossref
reference URL
https://api.crossref.org/works/10.1038%2FSJ.ONC.1210287
retrieved
7 January 2021
based on heuristic
inferred from DOI database lookup
The roles of IFN gamma in protection against tumor development and cancer immunoediting
1 reference
stated in
Crossref
reference URL
https://api.crossref.org/works/10.1038%2FSJ.ONC.1210287
retrieved
7 January 2021
based on heuristic
inferred from DOI database lookup
Retracted: Three unrelated viral transforming proteins (vIRF, EBNA2, and E1A) induce the MYC oncogene through the interferon-responsive PRF element by using different transcription coadaptors
1 reference
stated in
Crossref
reference URL
https://api.crossref.org/works/10.1038%2FSJ.ONC.1210287
retrieved
7 January 2021
based on heuristic
inferred from DOI database lookup
CIAP1 and the serine protease HTRA2 are involved in a novel p53-dependent apoptosis pathway in mammals.
1 reference
stated in
Crossref
reference URL
https://api.crossref.org/works/10.1038%2FSJ.ONC.1210287
retrieved
7 January 2021
based on heuristic
inferred from DOI database lookup
The GRIMs: a new interface between cell death regulation and interferon/retinoid induced growth suppression
1 reference
stated in
Crossref
reference URL
https://api.crossref.org/works/10.1038%2FSJ.ONC.1210287
retrieved
7 January 2021
based on heuristic
inferred from DOI database lookup
The tumor suppressor WARTS activates the Omi / HtrA2-dependent pathway of cell death
1 reference
stated in
Crossref
reference URL
https://api.crossref.org/works/10.1038%2FSJ.ONC.1210287
retrieved
7 January 2021
based on heuristic
inferred from DOI database lookup
Structural insights into the pro-apoptotic function of mitochondrial serine protease HtrA2/Omi
1 reference
stated in
Crossref
reference URL
https://api.crossref.org/works/10.1038%2FSJ.ONC.1210287
retrieved
7 January 2021
based on heuristic
inferred from DOI database lookup
The serine protease Omi/HtrA2 regulates apoptosis by binding XIAP through a reaper-like motif
1 reference
stated in
Crossref
reference URL
https://api.crossref.org/works/10.1038%2FSJ.ONC.1210287
retrieved
7 January 2021
based on heuristic
inferred from DOI database lookup
Viral interferon regulatory factor 1 of Kaposi's sarcoma-associated herpesvirus interacts with a cell death regulator, GRIM19, and inhibits interferon/retinoic acid-induced cell death.
1 reference
stated in
Crossref
reference URL
https://api.crossref.org/works/10.1038%2FSJ.ONC.1210287
retrieved
7 January 2021
based on heuristic
inferred from DOI database lookup
IFNgamma and lymphocytes prevent primary tumour development and shape tumour immunogenicity
1 reference
stated in
Crossref
reference URL
https://api.crossref.org/works/10.1038%2FSJ.ONC.1210287
retrieved
7 January 2021
based on heuristic
inferred from DOI database lookup
Caspase-9 can be activated without proteolytic processing
1 reference
stated in
Crossref
reference URL
https://api.crossref.org/works/10.1038%2FSJ.ONC.1210287
retrieved
7 January 2021
based on heuristic
inferred from DOI database lookup
A serine protease, HtrA2, is released from the mitochondria and interacts with XIAP, inducing cell death
1 reference
stated in
Crossref
reference URL
https://api.crossref.org/works/10.1038%2FSJ.ONC.1210287
retrieved
7 January 2021
based on heuristic
inferred from DOI database lookup
The serine protease Omi/HtrA2 is released from mitochondria during apoptosis. Omi interacts with caspase-inhibitor XIAP and induces enhanced caspase activity
1 reference
stated in
Crossref
reference URL
https://api.crossref.org/works/10.1038%2FSJ.ONC.1210287
retrieved
7 January 2021
based on heuristic
inferred from DOI database lookup
Bcl-xL regulates the membrane potential and volume homeostasis of mitochondria
1 reference
stated in
Crossref
reference URL
https://api.crossref.org/works/10.1038%2FSJ.ONC.1210287
retrieved
7 January 2021
based on heuristic
inferred from DOI database lookup
Mammalian mitochondrial IAP binding proteins
1 reference
stated in
Crossref
reference URL
https://api.crossref.org/works/10.1038%2FSJ.ONC.1210287
retrieved
7 January 2021
based on heuristic
inferred from DOI database lookup
Omi/HtrA2 catalytic cleavage of inhibitor of apoptosis (IAP) irreversibly inactivates IAPs and facilitates caspase activity in apoptosis
1 reference
stated in
Crossref
reference URL
https://api.crossref.org/works/10.1038%2FSJ.ONC.1210287
retrieved
7 January 2021
based on heuristic
inferred from DOI database lookup
The cell death regulator GRIM-19 is an inhibitor of signal transducer and activator of transcription 3
1 reference
stated in
Crossref
reference URL
https://api.crossref.org/works/10.1038%2FSJ.ONC.1210287
retrieved
7 January 2021
based on heuristic
inferred from DOI database lookup
Identifiers
DOI
10.1038/SJ.ONC.1210287
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Dimensions Publication ID
1008987423
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PubMed publication ID
17297443
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ResearchGate publication ID
6508780
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