(Q24300008)

2

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Rodrigo Martinez

3

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Karen Manoutcharian

4

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Goar Gevorkian

Progressive mitochondrial disease resulting from a novel missense mutation in the mitochondrial DNA ND3 gene.

5

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language of work or name

English

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publication date

April 2006

1 reference

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397

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issue

1-2

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page(s)

79-82

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cites work

1 reference

https://api.crossref.org/works/10.1016%2FJ.NEULET.2005.11.061

7 January 2021

Characterization of cerebrospinal fluid antibody specificities in neurocysticercosis using phage display peptide library

1 reference

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7 January 2021

Involvement of microglial receptor for advanced glycation endproducts (RAGE) in Alzheimer's disease: identification of a cellular activation mechanism.

1 reference

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7 January 2021

Review: model peptides and the physicochemical approach to beta-amyloids

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7 January 2021

Beta-amyloid fragment 25-35 causes mitochondrial dysfunction in primary cortical neurons

1 reference

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7 January 2021

Gradual alteration of mitochondrial structure and function by beta-amyloids: importance of membrane viscosity changes, energy deprivation, reactive oxygen species production, and cytochrome c release

1 reference

https://api.crossref.org/works/10.1016%2FJ.NEULET.2005.11.061

7 January 2021

Hydrogen peroxide mediates amyloid beta protein toxicity

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7 January 2021

beta-amyloid fibrils induce tau phosphorylation and loss of microtubule binding.

1 reference

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7 January 2021

Amyloid-beta peptide-specific single chain Fv antibodies isolated from an immune phage display library

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7 January 2021

Intraneuronal Abeta causes the onset of early Alzheimer's disease-related cognitive deficits in transgenic mice

1 reference

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7 January 2021

Intraneuronal Alzheimer abeta42 accumulates in multivesicular bodies and is associated with synaptic pathology

1 reference

https://api.crossref.org/works/10.1016%2FJ.NEULET.2005.11.061

7 January 2021

Intraneuronal Abeta42 accumulation in human brain.

1 reference

https://api.crossref.org/works/10.1016%2FJ.NEULET.2005.11.061

7 January 2021

Microglia, scavenger receptors, and the pathogenesis of Alzheimer's disease

1 reference

https://api.crossref.org/works/10.1016%2FJ.NEULET.2005.11.061

7 January 2021

Triple-transgenic model of Alzheimer's disease with plaques and tangles: intracellular Abeta and synaptic dysfunction

1 reference

https://api.crossref.org/works/10.1016%2FJ.NEULET.2005.11.061

7 January 2021

Self-assembly of Abeta(1-42) into globular neurotoxins.

1 reference

https://api.crossref.org/works/10.1016%2FJ.NEULET.2005.11.061

7 January 2021

Toxicity of amyloid beta peptide: tales of calcium, mitochondria, and oxidative stress

1 reference

https://api.crossref.org/works/10.1016%2FJ.NEULET.2005.11.061

7 January 2021

An intracellular protein that binds amyloid-beta peptide and mediates neurotoxicity in Alzheimer's disease

1 reference

https://api.crossref.org/works/10.1016%2FJ.NEULET.2005.11.061

7 January 2021

Amyloid-beta oligomers: their production, toxicity and therapeutic inhibition

1 reference

https://api.crossref.org/works/10.1016%2FJ.NEULET.2005.11.061

7 January 2021

Beta-amyloid inhibits integrated mitochondrial respiration and key enzyme activities

1 reference

https://api.crossref.org/works/10.1016%2FJ.NEULET.2005.11.061

7 January 2021

A model for beta-amyloid aggregation and neurotoxicity based on free radical generation by the peptide: relevance to Alzheimer disease

1 reference

https://api.crossref.org/works/10.1016%2FJ.NEULET.2005.11.061

7 January 2021

Amyloid-beta peptide-receptor for advanced glycation endproduct interaction elicits neuronal expression of macrophage-colony stimulating factor: a proinflammatory pathway in Alzheimer disease

1 reference

https://api.crossref.org/works/10.1016%2FJ.NEULET.2005.11.061

7 January 2021

A human brain L-3-hydroxyacyl-coenzyme A dehydrogenase is identical to an amyloid beta-peptide-binding protein involved in Alzheimer's disease

1 reference

https://api.crossref.org/works/10.1016%2FJ.NEULET.2005.11.061

7 January 2021

beta-Amyloid(1-42) binds to alpha7 nicotinic acetylcholine receptor with high affinity. Implications for Alzheimer's disease pathology

1 reference

https://api.crossref.org/works/10.1016%2FJ.NEULET.2005.11.061

7 January 2021

Mitochondrial Polymorphisms Significantly Reduce the Risk of Parkinson Disease

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https://api.crossref.org/works/10.1016%2FJ.NEULET.2005.11.061

7 January 2021

ABAD enhances Abeta-induced cell stress via mitochondrial dysfunction

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https://api.crossref.org/works/10.1016%2FJ.NEULET.2005.11.061

7 January 2021

Alzheimer's disease: a central role for amyloid

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https://api.crossref.org/works/10.1016%2FJ.NEULET.2005.11.061

7 January 2021

Mitochondrial dysfunction and Alzheimer's disease: role of amyloid-beta peptide alcohol dehydrogenase (ABAD).

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https://api.crossref.org/works/10.1016%2FJ.NEULET.2005.11.061

7 January 2021

Common structure of soluble amyloid oligomers implies common mechanism of pathogenesis

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https://api.crossref.org/works/10.1016%2FJ.NEULET.2005.11.061

7 January 2021

ABAD directly links Abeta to mitochondrial toxicity in Alzheimer's disease.

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https://api.crossref.org/works/10.1016%2FJ.NEULET.2005.11.061

7 January 2021

Alzheimer's disease: genes, proteins, and therapy

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https://api.crossref.org/works/10.1016%2FJ.NEULET.2005.11.061

7 January 2021

Shotgun phage display cloning

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https://api.crossref.org/works/10.1016%2FJ.NEULET.2005.11.061

7 January 2021