(Q24300967)

English

Targeted cardiac overexpression of A20 improves left ventricular performance and reduces compensatory hypertrophy after myocardial infarction

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scholarly article

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Targeted cardiac overexpression of A20 improves left ventricular performance and reduces compensatory hypertrophy after myocardial infarction (English)

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TNF superfamily member 12

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GOA release 2020-03-11

positive regulation of protein catabolic process

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GOA release 2020-03-11

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based on heuristic

inferred from title

myocardial infarction

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author name string

Hong-Liang Li

1

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Ming-Lei Zhuo

2

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Dong Wang

3

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Ai-Bing Wang

4

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Hua Cai

5

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Li-Hong Sun

6

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Qinglin Yang

7

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Yue Huang

8

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Yu-Sheng Wei

9

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Peter P Liu

10

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De-Pei Liu

11

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Chih-Chuan Liang

12

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language of work or name

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publication date

10 April 2007

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115

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14

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1885-94

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A20 inhibits oxidized low-density lipoprotein-induced apoptosis through negative Fas/Fas ligand-dependent activation of caspase-8 and mitochondrial pathways in murine RAW264.7 macrophages.

1 reference

https://api.crossref.org/works/10.1161%2FCIRCULATIONAHA.106.656835

7 January 2021

based on heuristic

inferred from DOI database lookup

Inhibition of the activation of nuclear factor kappa B to reduce myocardial reperfusion injury and infarct size

1 reference

https://api.crossref.org/works/10.1161%2FCIRCULATIONAHA.106.656835

7 January 2021

based on heuristic

inferred from DOI database lookup

Targeting survival signaling in heart failure

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https://api.crossref.org/works/10.1161%2FCIRCULATIONAHA.106.656835

7 January 2021

based on heuristic

inferred from DOI database lookup

A20 inhibits NF-kappaB activation by dual ubiquitin-editing functions

1 reference

https://api.crossref.org/works/10.1161%2FCIRCULATIONAHA.106.656835

7 January 2021

based on heuristic

inferred from DOI database lookup

Apoptosis of ventricular myocytes: a means to an end.

1 reference

https://api.crossref.org/works/10.1161%2FCIRCULATIONAHA.106.656835

7 January 2021

based on heuristic

inferred from DOI database lookup

Targeted cardiac expression of soluble Fas prevents the development of heart failure in mice with cardiac-specific expression of MCP-1.

1 reference

https://api.crossref.org/works/10.1161%2FCIRCULATIONAHA.106.656835

7 January 2021

based on heuristic

inferred from DOI database lookup

A20 and A20-binding proteins as cellular inhibitors of nuclear factor-kappa B-dependent gene expression and apoptosis

1 reference

https://api.crossref.org/works/10.1161%2FCIRCULATIONAHA.106.656835

7 January 2021

based on heuristic

inferred from DOI database lookup

Signal transduction by tumor necrosis factor and its relatives

1 reference

https://api.crossref.org/works/10.1161%2FCIRCULATIONAHA.106.656835

7 January 2021

based on heuristic

inferred from DOI database lookup

Protein kinase C isoform-selective signals that lead to cardiac hypertrophy and the progression of heart failure

1 reference

https://api.crossref.org/works/10.1161%2FCIRCULATIONAHA.106.656835

7 January 2021

based on heuristic

inferred from DOI database lookup

In vivo transfection of cis element "decoy" against nuclear factor-kappaB binding site prevents myocardial infarction

1 reference

https://api.crossref.org/works/10.1161%2FCIRCULATIONAHA.106.656835

7 January 2021

based on heuristic

inferred from DOI database lookup

Absence of NF-kappaB subunit p50 improves heart failure after myocardial infarction

1 reference

https://api.crossref.org/works/10.1161%2FCIRCULATIONAHA.106.656835

7 January 2021

based on heuristic

inferred from DOI database lookup

Blockade of NF-kappaB improves cardiac function and survival after myocardial infarction

1 reference

https://api.crossref.org/works/10.1161%2FCIRCULATIONAHA.106.656835

7 January 2021

based on heuristic

inferred from DOI database lookup

A direct requirement of nuclear factor-kappa B for suppression of apoptosis in ventricular myocytes

1 reference

https://api.crossref.org/works/10.1161%2FCIRCULATIONAHA.106.656835

7 January 2021

based on heuristic

inferred from DOI database lookup

A20 is dynamically regulated in the heart and inhibits the hypertrophic response

1 reference

https://api.crossref.org/works/10.1161%2FCIRCULATIONAHA.106.656835

7 January 2021

based on heuristic

inferred from DOI database lookup

Nuclear factor-kappaB protects the adult cardiac myocyte against ischemia-induced apoptosis in a murine model of acute myocardial infarction.

1 reference

https://api.crossref.org/works/10.1161%2FCIRCULATIONAHA.106.656835

7 January 2021

based on heuristic

inferred from DOI database lookup

Excessive Tumor Necrosis Factor Activation After Infarction Contributes to Susceptibility of Myocardial Rupture and Left Ventricular Dysfunction

1 reference

https://api.crossref.org/works/10.1161%2FCIRCULATIONAHA.106.656835

7 January 2021

based on heuristic

inferred from DOI database lookup

Role of the mitochondrial permeability transition in myocardial disease

1 reference

https://api.crossref.org/works/10.1161%2FCIRCULATIONAHA.106.656835

7 January 2021

based on heuristic

inferred from DOI database lookup

Inflammatory cytokines and postmyocardial infarction remodeling

1 reference

https://api.crossref.org/works/10.1161%2FCIRCULATIONAHA.106.656835

7 January 2021

based on heuristic

inferred from DOI database lookup

Chronic production of angiotensin IV in the brain leads to hypertension that is reversible with an angiotensin II AT1 receptor antagonist

1 reference

https://api.crossref.org/works/10.1161%2FCIRCULATIONAHA.106.656835

7 January 2021

based on heuristic

inferred from DOI database lookup

Blockade of the natriuretic peptide receptor guanylyl cyclase-A inhibits NF-kappaB activation and alleviates myocardial ischemia/reperfusion injury

1 reference

https://api.crossref.org/works/10.1161%2FCIRCULATIONAHA.106.656835

7 January 2021

based on heuristic

inferred from DOI database lookup

Mitochondria and ischemia/reperfusion injury

1 reference

https://api.crossref.org/works/10.1161%2FCIRCULATIONAHA.106.656835

7 January 2021

based on heuristic

inferred from DOI database lookup

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