(Q24307362)

extrinsic apoptotic signaling pathway via death domain receptors

GOA release 2020-03-11

1 reference

GOA release 2020-03-11

author name string

A M Chinnaiyan

1

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K O'Rourke

2

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M Tewari

3

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V M Dixit

Use of bacteriophage T7 lysozyme to improve an inducible T7 expression system

4

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language of work or name

English

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publication date

19 May 1995

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published in

Cell

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volume

81

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page(s)

505-12

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issue

4

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cites work

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The polypeptide encoded by the cDNA for human cell surface antigen Fas can mediate apoptosis

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A novel domain within the 55 kd TNF receptor signals cell death

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The C. elegans cell death gene ced-3 encodes a protein similar to mammalian interleukin-1 beta-converting enzyme

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Induction of apoptosis in fibroblasts by IL-1 beta-converting enzyme, a mammalian homolog of the C. elegans cell death gene ced-3

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The p21 Cdk-interacting protein Cip1 is a potent inhibitor of G1 cyclin-dependent kinases

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An evolutionary perspective on apoptosis.

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A novel family of putative signal transducers associated with the cytoplasmic domain of the 75 kDa tumor necrosis factor receptor

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Lymphoproliferation disorder in mice explained by defects in Fas antigen that mediates apoptosis

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A novel heterodimeric cysteine protease is required for interleukin-1 beta processing in monocytes

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Autocrine T-cell suicide mediated by APO-1/(Fas/CD95)

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Cell-autonomous Fas (CD95)/Fas-ligand interaction mediates activation-induced apoptosis in T-cell hybridomas.

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Self-association of the Death Domains of the p55 Tumor Necrosis Factor (TNF) Receptor and Fas/APO1 Prompts Signaling for TNF and Fas/APO1 Effects

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Fas- and tumor necrosis factor-induced apoptosis is inhibited by the poxvirus crmA gene product

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The scanning model for translation: an update

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Mechanisms and Functions of Cell Death

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