(Q24309665)

Circulating levels of cytokines that modulate bone resorption: effects of age and menopause in women

GOA release 2020-03-11

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Hui Liu

1

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Kenian Liu

series ordinal

2

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Donald L Bodenner

series ordinal

3

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language of work or name

English

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publication date

21 August 2005

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published in

Cytokine

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volume

31

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issue

4

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page(s)

251-7

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cites work

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17 beta-estradiol inhibits tumor necrosis factor-alpha-induced nuclear factor-kappa B activation by increasing nuclear factor-kappa B p105 level in MCF-7 breast cancer cells

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Interleukin-6 and viral myocarditis: the Yin-Yang of cardiac innate immune responses.

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The nuclear receptor superfamily: the second decade

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Steroid and related receptors

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Repression of interleukin-6 gene expression by 17 beta-estradiol: inhibition of the DNA-binding activity of the transcription factors NF-IL6 and NF-kappa B by the estrogen receptor

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Interleukin-6 inhibits bone formation in vitro

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Genetics of interleukin 6: implications for Alzheimer's disease

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Suppression by estrogen receptor beta of AP-1 mediated transactivation through estrogen receptor alpha

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17beta-Estradiol inhibits cytokine induction of the human E-selectin promoter

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IL-6 in autoimmune disease and chronic inflammatory proliferative disease

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Interleukin-6 modulates trabecular and endochondral bone turnover in the nude mouse by stimulating osteoclast differentiation

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Interleukin-6 and interleukin-11 support human osteoclast formation by a RANKL-independent mechanism.

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Negative interaction between the RelA(p65) subunit of NF-kappaB and the progesterone receptor

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Distinct domains of the RelA NF-kappaB subunit are required for negative cross-talk and direct interaction with the glucocorticoid receptor.

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Mutations in the estrogen receptor ligand binding domain discriminate between hormone-dependent transactivation and transrepression

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Mechanism of kappa B DNA binding by Rel/NF-kappa B dimers

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Estrogen receptor binding to DNA is not required for its activity through the nonclassical AP1 pathway

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Characterization of the physical interaction between estrogen receptor alpha and JUN proteins

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Opposing action of estrogen receptors alpha and beta on cyclin D1 gene expression

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Estrogen receptor impairs interleukin-6 expression by preventing protein binding on the NF-kappaB site

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Molecular pathways of steroid receptor action

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Independent modes of transcriptional activation by the p50 and p65 subunits of NF-kappa B

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Increased osteoclast development after estrogen loss: mediation by interleukin-6

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Differential ligand activation of estrogen receptors ERalpha and ERbeta at AP1 sites

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Characterization of mechanisms involved in transrepression of NF-kappa B by activated glucocorticoid receptors

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Repression of the interleukin-6 promoter by estrogen receptor is mediated by NF-kappa B and C/EBP beta

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AF-2 activity and recruitment of steroid receptor coactivator 1 to the estrogen receptor depend on a lysine residue conserved in nuclear receptors

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The NF-kappa B and I kappa B proteins: new discoveries and insights

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Competition for p300 regulates transcription by estrogen receptors and nuclear factor-kappaB in human coronary smooth muscle cells

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17 beta-estradiol inhibits interleukin-6 production by bone marrow-derived stromal cells and osteoblasts in vitro: a potential mechanism for the antiosteoporotic effect of estrogens

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Transcriptional activities of estrogen and glucocorticoid receptors are functionally integrated at the AP-1 response element

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An activator protein 1-like motif mediates 17beta-estradiol repression of gonadotropin-releasing hormone receptor promoter via an estrogen receptor alpha-dependent mechanism in ovarian and breast cancer cells.

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Negative cross-talk between RelA and the glucocorticoid receptor: a possible mechanism for the antiinflammatory action of glucocorticoids

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Interleukin-6 (IL-6), IL-1, receptor activator of nuclear factor kappaB ligand (RANKL) and osteoprotegerin production by human osteoblastic cells: comparison of the effects of 17-beta oestradiol and raloxifene

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Anti-interleukin-6 therapy for Crohn's disease

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