(Q24337999)

Neurochemistry International

GOA release 2020-03-11

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1 reference

Peethambaran Arun

1

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John R Moffett

series ordinal

2

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Aryan M A Namboodiri

series ordinal

3

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language of work or name

English

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publication date

September 2009

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published in

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volume

55

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issue

4

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page(s)

219-25

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cites work

In vivo quantitation of cerebral metabolite concentrations using natural abundance 13C MRS at 1.5 T.

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Differential distribution of N-acetylaspartylglutamate and N-acetylaspartate immunoreactivities in rat forebrain

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Acetyl transport mechanisms. Metabolism of N-acetyl-L-aspartic acid in the non-nervous tissues of the rat

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Biosynthesis of N-acetyl-L-aspartic acid.

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Immunocytochemical localization of N-acetyl-aspartate with monoclonal antibodies

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Neuronal and astrocytic shuttle mechanisms for cytosolic-mitochondrial transfer of reducing equivalents: current evidence and pharmacological tools

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N-acetylaspartate synthesis in the brain: mitochondria vs. microsomes

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N-Acetylaspartate synthase is bimodally expressed in microsomes and mitochondria of brain

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N-Acetylaspartate in the CNS: from neurodiagnostics to neurobiology

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A radiometric assay for aspartoacylase activity in cultured oligodendrocytes

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Canavan disease: from spongy degeneration to molecular analysis

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Mitochondrial malic enzyme activity is much higher in mitochondria from cortical synaptic terminals compared with mitochondria from primary cultures of cortical neurons or cerebellar granule cells

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Synthesis of N-acetyl-l-aspartate by rat brain mitochondria and its involvement in mitochondrial/cytosolic carbon transport

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In vitro expression of N-acetyl aspartate by oligodendrocytes: implications for proton magnetic resonance spectroscopy signal in vivo

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Characterization of the N-acetylaspartate biosynthetic enzyme from rat brain

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Molecular cloning of Aralar, a new member of the mitochondrial carrier superfamily that binds calcium and is present in human muscle and brain

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Reduced N-acetylaspartate levels in mice lacking aralar, a brain- and muscle-type mitochondrial aspartate-glutamate carrier

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N-Acetylaspartate reduction as a measure of injury severity and mitochondrial dysfunction following diffuse traumatic brain injury

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Immunohistochemical localization of N-acetylaspartate in rat brain

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N-Acetylation of L-aspartate in the nervous system: differential distribution of a specific enzyme.

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Influence of the Malate-Aspartate Shuttle on Oxidative Metabolism in Synaptosomes

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Regulation of N-acetylaspartate and N-acetylaspartylglutamate biosynthesis by protein kinase activators

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Antipsychotic drugs increase N-acetylaspartate and N-acetylaspartylglutamate in SH-SY5Y human neuroblastoma cells

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N-acetyl aspartate: a marker for neuronal loss or mitochondrial dysfunction

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Assessment of mitochondrial impairment in traumatic brain injury using high-resolution proton magnetic resonance spectroscopy

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7 January 2021

10.1016/J.NEUINT.2009.03.003

Identifiers

DOI

1 reference

Consolidated OpenCitations Corpus – April 2017

2125577

2125577

1 reference

Consolidated OpenCitations Corpus – April 2017

Consolidated OpenCitations Corpus – April 2017

1 reference