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Stimulation of intrachromosomal homologous recombination in mammalian cells by an inhibitor of poly(ADP-ribosylation)

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Stimulation of intrachromosomal homologous recombination in mammalian cells by an inhibitor of poly(ADP-ribosylation) (English)

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author name string

A S Waldman

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1

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B C Waldman

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2

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language of work or name

English

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publication date

11 November 1991

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published in

Nucleic Acids Research

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volume

19

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page(s)

5943-7

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issue

21

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cites work

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Novel inhibitors of poly(ADP-ribose) synthetase

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Altering the genome by homologous recombination

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Regulation of DNA ligase activity by poly(ADP-ribose)

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Conservative intrachromosomal recombination between inverted repeats in mouse cells: association between reciprocal exchange and gene conversion

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27 September 2017

ADP-ribosylation is involved in the integration of foreign DNA into the mammalian cell genome

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27 September 2017

Deletion of transfected oncogenes from NIH 3T3 transformants by inhibitors of poly(ADP-ribose) polymerase

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Carcinogens can induce homologous recombination between duplicated chromosomal sequences in mouse L cells

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Differential effects of base-pair mismatch on intrachromosomal versus extrachromosomal recombination in mouse cells

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Homologous recombination in mammalian cells

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The new mouse genetics: altering the genome by gene targeting

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Illegitimate and homologous recombination in mammalian cells: differential sensitivity to an inhibitor of poly(ADP-ribosylation).

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A table for the estimation of the spontaneous mutations rate of cells in culture

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30 May 2018

Poly(ADP-ribose) synthesis in vitro programmed by damaged DNA. A comparison of DNA molecules containing different types of strand breaks

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based on heuristic

inferred from PubMed ID database lookup

Homologous recombination between repeated chromosomal sequences in mouse cells

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reference URL

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retrieved

12 December 2020

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Identifiers

DOI

10.1093/NAR/19.21.5943

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ResearchGate publication ID

21209875

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(Q24629084)

Stimulation of intrachromosomal homologous recombination in mammalian cells by an inhibitor of poly(ADP-ribosylation)

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