(Q24673242)

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I-PLA(2) activation during apoptosis promotes the exposure of membrane lysophosphatidylcholine leading to binding by natural immunoglobulin M antibodies and complement activation

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I-PLA(2) activation during apoptosis promotes the exposure of membrane lysophosphatidylcholine leading to binding by natural immunoglobulin M antibodies and complement activation (English)

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apoptotic process

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author name string

Sun Jun Kim

1

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Debra Gershov

2

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Xiaojing Ma

3

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Nathan Brot

4

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Keith B Elkon

5

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language of work or name

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publication date

2 September 2002

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Journal of Experimental Medicine

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196

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5

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655-65

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C1q and mannose binding lectin engagement of cell surface calreticulin and CD91 initiates macropinocytosis and uptake of apoptotic cells

1 reference

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7 April 2017

C-Reactive protein binds to apoptotic cells, protects the cells from assembly of the terminal complement components, and sustains an antiinflammatory innate immune response: implications for systemic autoimmunity

1 reference

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7 April 2017

The induction of tolerance by dendritic cells that have captured apoptotic cells

1 reference

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Dendritic cells and the control of immunity

1 reference

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Natural antibodies with the T15 idiotype may act in atherosclerosis, apoptotic clearance, and protective immunity

1 reference

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7 April 2017

Targeted gene disruption reveals a role for natural secretory IgM in the maturation of the primary immune response

1 reference

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7 April 2017

IgG Fc receptors

1 reference

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7 April 2017

Macrophages that have ingested apoptotic cells in vitro inhibit proinflammatory cytokine production through autocrine/paracrine mechanisms involving TGF-beta, PGE2, and PAF

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Studies of acute phase protein. I. An immunohistochemical method for the localization of Cx-reactive protein in rabbits. Association with necrosis in local inflammatory lesions

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A pyrrolidine-based specific inhibitor of cytosolic phospholipase A(2)alpha blocks arachidonic acid release in a variety of mammalian cells

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Role of natural and immune IgM antibodies in immune responses

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28 September 2017

Phagocytosis and clearance of apoptotic cells is mediated by MER.

1 reference

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28 September 2017

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28 September 2017

Phospholipase A(2)s in cell injury and death

1 reference

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28 September 2017

Deficiency in serum immunoglobulin (Ig)M predisposes to development of IgG autoantibodies.

1 reference

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28 September 2017

Accelerated development of IgG autoantibodies and autoimmune disease in the absence of secreted IgM.

1 reference

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28 September 2017

Natural adjuvants: endogenous activators of dendritic cells

1 reference

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Regulation of cyclooxygenase-2 expression by phosphatidate phosphohydrolase in human amnionic WISH cells

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2194002

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Monoclonal antibodies against oxidized low-density lipoprotein bind to apoptotic cells and inhibit their phagocytosis by elicited macrophages: evidence that oxidation-specific epitopes mediate macrophage recognition

1 reference

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pmc&linkname=pmc_refs_pubmed&retmode=json&id=2194002

28 September 2017

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1 reference

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Complement-dependent clearance of apoptotic cells by human macrophages

1 reference

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28 September 2017

Involvement of caspase-dependent activation of cytosolic phospholipase A2 in tumor necrosis factor-induced apoptosis

1 reference

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28 September 2017

Anti-phospholipid autoantibodies bind to apoptotic, but not viable, thymocytes in a beta 2-glycoprotein I-dependent manner

1 reference

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1 reference

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Structure and function of natural antibodies.

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Oxidative stress as a mediator of apoptosis

1 reference

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1 reference

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1 reference

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1 reference

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https://pubmed.ncbi.nlm.nih.gov/12208880

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1 reference

https://pubmed.ncbi.nlm.nih.gov/12208880

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inferred from PubMed ID database lookup

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Fas-induced arachidonic acid release is mediated by Ca2+-independent phospholipase A2 but not cytosolic phospholipase A2, which undergoes proteolytic inactivation

1 reference

https://pubmed.ncbi.nlm.nih.gov/12208880

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1 reference

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Identifiers

10.1084/JEM.20020542

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Consolidated OpenCitations Corpus – April 2017

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Consolidated OpenCitations Corpus – April 2017

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Consolidated OpenCitations Corpus – April 2017

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