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EBNA-1, a bifunctional transcriptional activator
scientific article
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instance of
scholarly article
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title
EBNA-1, a bifunctional transcriptional activator
(English)
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main subject
cell biology
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author name string
Gregory Kennedy
series ordinal
1
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Bill Sugden
series ordinal
2
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language of work or name
English
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publication date
October 2003
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published in
Molecular and Cellular Biology
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volume
23
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issue
19
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page(s)
6901-8
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describes a project that uses
ImageQuant
1 reference
stated in
Europe PubMed Central
retrieved
11 June 2022
reference URL
https://www.ebi.ac.uk/europepmc/webservices/rest/PMC193932/fullTextXML
based on heuristic
inferred from PubMed Central ID database lookup
cites work
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Expression of Epstein-Barr virus nuclear antigen-1 induces B cell neoplasia in transgenic mice
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Transcription activator interactions with multiple SWI/SNF subunits.
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Transcriptional activation by Gcn4p involves independent interactions with the SWI/SNF complex and the SRB/mediator
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Separation of the DNA replication, segregation, and transcriptional activation functions of Epstein-Barr nuclear antigen 1.
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Epstein-Barr virus nuclear antigen 1 activates transcription from episomal but not integrated DNA and does not alter lymphocyte growth
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Epstein-Barr nuclear antigen 1 binds and destabilizes nucleosomes at the viral origin of latent DNA replication
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Epstein-Barr virus/human vector provides high-level, long-term expression of alpha1-antitrypsin in mice
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CD40 and LMP-1 both signal from lipid rafts but LMP-1 assembles a distinct, more efficient signaling complex
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The linking regions of EBNA1 are essential for its support of replication and transcription
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Mapping EBNA-1 domains involved in binding to metaphase chromosomes.
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Genetic evidence that EBNA-1 is needed for efficient, stable latent infection by Epstein-Barr virus
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Up to 100-fold increase of apparent gene expression in the presence of Epstein-Barr virus oriP sequences and EBNA1: implications of the nuclear import of plasmids
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The potential of extrachromosomal replicating vectors for gene therapy
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An EBNA-1-dependent enhancer acts from a distance of 10 kilobase pairs to increase expression of the Epstein-Barr virus LMP gene
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Stimulation of NF-kappa B-mediated transcription by mutant derivatives of the latent membrane protein of Epstein-Barr virus
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Multiple regions within EBNA1 can link DNAs.
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trans activation of an Epstein-Barr viral transcriptional enhancer by the Epstein-Barr viral nuclear antigen 1.
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28 September 2017
Multiple EBNA1-binding sites are required to form an EBNA1-dependent enhancer and to activate a minimal replicative origin within oriP of Epstein-Barr virus.
1 reference
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PubMed Central
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28 September 2017
A promoter of Epstein-Barr virus that can function during latent infection can be transactivated by EBNA-1, a viral protein required for viral DNA replication during latent infection.
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EBNA1 can link the enhancer element to the initiator element of the Epstein-Barr virus plasmid origin of DNA replication
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bcl-xL and RAG genes are induced and the response to IL-2 enhanced in EmuEBNA-1 transgenic mouse lymphocytes.
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Transcriptional regulation of mammalian genes in vivo. A tale of two templates
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Chromatin structure of Epstein-Barr virus
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Highly efficient gene transfer into murine liver achieved by intravenous administration of naked Epstein-Barr virus (EBV)-based plasmid vectors.
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Highly efficient suicide gene expression in hepatocellular carcinoma cells by epstein-barr virus-based plasmid vectors combined with polyamidoamine dendrimer
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28 November 2018
The Structure of Herpes Simplex Virus Type 1 DNA as Probed by Micrococcal Nuclease Digestion
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Transcription factor loading on the MMTV promoter: a bimodal mechanism for promoter activation
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based on heuristic
inferred from PubMed ID database lookup
Identifiers
DOI
10.1128/MCB.23.19.6901-6908.2003
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PMC publication ID
193932
0 references
PubMed publication ID
12972608
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