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exo1-Dependent mutator mutations: model system for studying functional interactions in mismatch repair
scientific article
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instance of
scholarly article
1 reference
stated in
PubMed
PubMed ID
11438669
retrieved
1 December 2016
title
exo1-Dependent mutator mutations: model system for studying functional interactions in mismatch repair
(English)
1 reference
stated in
PubMed
PubMed ID
11438669
retrieved
1 December 2016
main subject
cell biology
0 references
Rad2 family nuclease EXO1 YOR033C
1 reference
stated in
GOA release 2020-03-11
DNA mismatch repair
1 reference
based on heuristic
inferred from title
author name string
N S Amin
series ordinal
1
0 references
M N Nguyen
series ordinal
2
0 references
S Oh
series ordinal
3
0 references
R D Kolodner
series ordinal
4
0 references
language of work or name
English
0 references
publication date
August 2001
0 references
published in
Molecular and Cellular Biology
1 reference
stated in
PubMed
PubMed ID
11438669
retrieved
1 December 2016
volume
21
0 references
page(s)
5142-55
0 references
issue
15
0 references
cites work
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The evolutionarily conserved zinc finger motif in the largest subunit of human replication protein A is required for DNA replication and mismatch repair but not for nucleotide excision repair
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Isolation of an hMSH2-p160 heterodimer that restores DNA mismatch repair to tumor cells
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Methylation of the hMLH1 promoter correlates with lack of expression of hMLH1 in sporadic colon tumors and mismatch repair-defective human tumor cell lines
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Redundancy of Saccharomyces cerevisiae MSH3 and MSH6 in MSH2-dependent mismatch repair
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Microsatellite instability in cancer of the proximal colon
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Interactions of Exo1p with components of MutLalpha in Saccharomyces cerevisiae
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Decreased meiotic intergenic recombination and increased meiosis I nondisjunction in exo1 mutants of Saccharomyces cerevisiae
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29 September 2017
EXO1 and MSH6 are high-copy suppressors of conditional mutations in the MSH2 mismatch repair gene of Saccharomyces cerevisiae
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Functional interaction between the Werner Syndrome protein and DNA polymerase delta
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Mismatch repair defects in cancer
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Saccharomyces cerevisiae pol30 (proliferating cell nuclear antigen) mutations impair replication fidelity and mismatch repair
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29 September 2017
Mutator phenotypes conferred by MLH1 overexpression and by heterozygosity for mlh1 mutations.
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29 September 2017
The 3'-->5' exonucleases of DNA polymerases delta and epsilon and the 5'-->3' exonuclease Exo1 have major roles in postreplication mutation avoidance in Saccharomyces cerevisiae
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Restoration of mismatch repair to nuclear extracts of H6 colorectal tumor cells by a heterodimer of human MutL homologs
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29 September 2017
A mutational analysis of the yeast proliferating cell nuclear antigen indicates distinct roles in DNA replication and DNA repair
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2 June 2018
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2 June 2018
Chromosomal rearrangements occur in S. cerevisiae rfa1 mutator mutants due to mutagenic lesions processed by double-strand-break repair
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Methylation of hMLH1 in a population-based series of endometrial carcinomas
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12 December 2020
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Methylation of the hMLH1 promoter but no hMLH1 mutations in sporadic gastric carcinomas with high-level microsatellite instability
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12 December 2020
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Somatic mutations in the hMSH2 gene in microsatellite unstable colorectal carcinomas
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12 December 2020
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The Saccharomyces cerevisiae protein YJR043C (Pol32) interacts with the catalytic subunit of DNA polymerase alpha and is required for cell cycle progression in G2/M
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12 December 2020
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Mutations predisposing to hereditary nonpolyposis colorectal cancer: Database and results of a collaborative study. The International Collaborative Group on Hereditary Nonpolyposis Colorectal Cancer
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12 December 2020
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A novel mutation avoidance mechanism dependent on S. cerevisiae RAD27 is distinct from DNA mismatch repair
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stated in
PubMed
reference URL
https://pubmed.ncbi.nlm.nih.gov/11438669
retrieved
12 December 2020
based on heuristic
inferred from PubMed ID database lookup
POL32, a subunit of the Saccharomyces cerevisiae DNA polymerase delta, defines a link between DNA replication and the mutagenic bypass repair pathway
1 reference
stated in
PubMed
reference URL
https://pubmed.ncbi.nlm.nih.gov/11438669
retrieved
12 December 2020
based on heuristic
inferred from PubMed ID database lookup
Identifiers
DOI
10.1128/MCB.21.15.5142-5155.2001
1 reference
stated in
Consolidated OpenCitations Corpus – April 2017
OpenCitations bibliographic resource ID
123310
OpenCitations bibliographic resource ID
123310
1 reference
stated in
Consolidated OpenCitations Corpus – April 2017
OpenCitations bibliographic resource ID
123310
PMCID
87239
1 reference
stated in
Consolidated OpenCitations Corpus – April 2017
OpenCitations bibliographic resource ID
123310
PubMed ID
11438669
1 reference
stated in
Consolidated OpenCitations Corpus – April 2017
OpenCitations bibliographic resource ID
123310
ResearchGate publication ID
11901865
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