(Q28186500)

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Differential roles of cyclooxygenase isoforms after kainic acid-induced prostaglandin E(2) production and neurodegeneration in cortical and hippocampal cell cultures

scientific article (publication date: 20 July 2001)

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Differential roles of cyclooxygenase isoforms after kainic acid-induced prostaglandin E(2) production and neurodegeneration in cortical and hippocampal cell cultures (English)

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neurodegeneration

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author name string

E J Kim

1

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J E Lee

2

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K J Kwon

3

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S H Lee

4

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C H Moon

5

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E J Baik

6

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language of work or name

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publication date

20 July 2001

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908

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1

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1-9

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Prostaglandin E2 protects cultured cortical neurons against N-methyl-D-aspartate receptor-mediated glutamate cytotoxicity

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Cyclooxygenase-2 selective inhibitors aggravate kainic acid induced seizure and neuronal cell death in the hippocampus

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Increased prostaglandin formation in rat brain following systemic application of kainic acid

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The cyclooxygenase and lipoxygenase inhibitor BW755C protects rats against kainic acid-induced seizures and neurotoxicity

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Differential regulation of apoptosis-related genes in resistant and vulnerable subfields of the rat epileptic hippocampus.

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Prostaglandins stimulate calcium-dependent glutamate release in astrocytes.

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Sources of reactive oxygen species production in excitotoxin- stimulated cerebellar granule cells.

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Protection by prostaglandins from glutamate toxicity in cortical neurons

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Glutamate neurotoxicity in cortical cell culture.

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The role of glutamate neurotoxicity in hypoxic-ischemic neuronal death

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Single-Step Method of RNA Isolation by Acid Guanidinium Thiocyanate–Phenol–Chloroform Extraction

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Crypt stem cell survival in the mouse intestinal epithelium is regulated by prostaglandins synthesized through cyclooxygenase-1.

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Rat cortical neurons in cell culture: Culture methods, cell morphology, electrophysiology, and synapse formation

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Kainate-induced status epilepticus alters glutamate and GABAA receptor gene expression in adult rat hippocampus: an in situ hybridization study

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Induction of cyclooxygenase (COX)-2 but not COX-1 gene expression in apoptotic cell death.

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Cyclooxygenase-1 behaves as a delayed response gene in PC12 cells differentiated by nerve growth factor

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Potentiation of excitotoxicity in transgenic mice overexpressing neuronal cyclooxygenase-2

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Quantitative determination of glutamate mediated cortical neuronal injury in cell culture by lactate dehydrogenase efflux assay

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Indomethacin inhibits delayed DNA fragmentation of hippocampal CA1 pyramidal neurons after transient forebrain ischemia in gerbils.

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Excitatory amino acid neurotoxicity and neurodegenerative disease

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Mitochondrial Targeting Signal of Rat Liver Monoamine Oxidase B Is Located at Its Carboxy Terminus

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Cyclooxygenase-2 inhibition prevents delayed death of CA1 hippocampal neurons following global ischemia

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Cyclo-oxygenase-2 gene expression in neurons contributes to ischemic brain damage.

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Markers for neuronal degeneration in organotypic slice cultures.

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Prostaglandin E1 induces c-Fos and Myc proteins and protects rat hippocampal cells against hypoxic injury

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Cyclooxygenase-2 expression is increased in frontal cortex of Alzheimer's disease brain.

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The relative contribution of constitutive and inducible cyclooxygenase activity to lipopolysaccharide-induced prostaglandin production by primary cultures of rat hypothalamic astrocytes

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Hundred-fold increase in neuronal vulnerability to glutamate toxicity in astrocyte-poor cultures of rat cerebral cortex

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Differentiation of monocytoid THP-1 cells with phorbol ester induces expression of prostaglandin endoperoxide synthase-1 (COX-1).

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Glutamate increases glycogen content and reduces glucose utilization in primary astrocyte culture.

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High sensitivity of glutamate uptake to extracellular free arachidonic acid levels in rat cortical synaptosomes and astrocytes.

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7 January 2021

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10.1016/S0006-8993(01)02432-5

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