(Q28200036)

English

NO activation of fos promoter elements requires nuclear translocation of G-kinase I and CREB phosphorylation but is independent of MAP kinase activation

scientific article (publication date: 14 December 2000)

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scholarly article

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NO activation of fos promoter elements requires nuclear translocation of G-kinase I and CREB phosphorylation but is independent of MAP kinase activation (English)

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phosphorylation

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inferred from title

author name string

T Gudi

1

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D E Casteel

2

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C Vinson

3

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G R Boss

4

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R B Pilz

5

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language of work or name

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publication date

14 December 2000

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19

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54

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6324-33

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A dominant-negative inhibitor of CREB reveals that it is a general mediator of stimulus-dependent transcription of c-fos

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Activation of cAMP and mitogen responsive genes relies on a common nuclear factor

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Multiple sequence elements of a single functional class are required for cyclic AMP responsiveness of the mouse c-fos promoter

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Constitutive expression of inducible nitric oxide synthase in human bronchial epithelial cells induces c-fos and stimulates the cGMP pathway

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Nitric oxide activation of p38 mitogen-activated protein kinase in 293T fibroblasts requires cGMP-dependent protein kinase

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Adenovirus-mediated gene transfer of cGMP-dependent protein kinase increases the sensitivity of cultured vascular smooth muscle cells to the antiproliferative and pro-apoptotic effects of nitric oxide/cGMP.

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Cyclic AMP- and cyclic GMP-dependent protein kinases differ in their regulation of cyclic AMP response element-dependent gene transcription

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Identification of a novel inhibitor of mitogen-activated protein kinase kinase

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CREB: a major mediator of neuronal neurotrophin responses.

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Multiple sequence elements in the c-fos promoter mediate induction by cAMP.

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Regulation of CREB phosphorylation in the suprachiasmatic nucleus by light and a circadian clock

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Requirement for nitric oxide activation of p21(ras)/extracellular regulated kinase in neuronal ischemic preconditioning

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cAMP stimulates the C/EBP-related transcription factor rNFIL-6 to trans-locate to the nucleus and induce c-fos transcription

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Odorants induce the phosphorylation of the cAMP response element binding protein in olfactory receptor neurons.

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A dominant negative to activation protein-1 (AP1) that abolishes DNA binding and inhibits oncogenesis.

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CREB: a stimulus-induced transcription factor activated by a diverse array of extracellular signals

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Cyclic-GMP-dependent protein kinase inhibits the Ras/Mitogen-activated protein kinase pathway

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Differential activation of CREB by Ca2+/calmodulin-dependent protein kinases type II and type IV involves phosphorylation of a site that negatively regulates activity

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Functional analysis of an isolated fos promoter element with AP-1 site homology reveals cell type-specific transcriptional properties

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Phosphorylation of splicing factor SF1 on Ser20 by cGMP-dependent protein kinase regulates spliceosome assembly

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AP-1: one switch for many signals

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Role of guanylyl cyclase and cGMP-dependent protein kinase in long-term potentiation.

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cAMP-dependent protein kinase, but not the cGMP-dependent enzyme, rapidly phosphorylates delta-CREB, and a synthetic delta-CREB peptide

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Regulation of gene expression by cGMP-dependent protein kinase. Transactivation of the c-fos promoter

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Stimulation of the cyclic GMP pathway by NO induces expression of the immediate early genes c-fos and junB in PC12 cells

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Nitric Oxide Inhibits c-Jun DNA Binding by Specifically TargetedS-Glutathionylation

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Identifiers

10.1038/SJ.ONC.1204007

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release_wcnotpzu6jgknacsuiwxsb66ka

1 reference

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24 November 2022

based on heuristic

mapped directly with Wikidata item

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ResearchGate publication ID

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Springer Nature article ID

10.1038/sj.onc.1204007

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