(Q28207618)

2

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S Wilde

3

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W Hiddemann

4

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F Alves

European Journal of Haematology

5

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language of work or name

English

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publication date

August 2001

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published in

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volume

67

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page(s)

63-71

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issue

2

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cites work

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Jaks and STATs: biological implications

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Targeted disruption of the mouse Stat1 gene results in compromised innate immunity to viral disease

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JAK/STAT signaling by cytokine receptors

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The JAK-STAT pathway: signal transduction involved in proliferation, differentiation and transformation.

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Differential constitutive activation between STAT-related proteins and MAP kinase in primary acute myelogenous leukaemia.

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Stat3 activation is required for cellular transformation by v-src

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Stat3 activation by Src induces specific gene regulation and is required for cell transformation

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Constitutive activation of JAK2 confers murine interleukin-3-independent survival and proliferation of BA/F3 cells.

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Proliferation of adult T cell leukemia/lymphoma cells is associated with the constitutive activation of JAK/STAT proteins

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Inhibition of acute lymphoblastic leukaemia by a Jak-2 inhibitor

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Constitutive activation of a slowly migrating isoform of Stat3 in mycosis fungoides: tyrphostin AG490 inhibits Stat3 activation and growth of mycosis fungoides tumor cell lines

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A biological method for the quantitative measurement of tetrodotoxin (TTX): tissue culture bioassay in combination with a water-soluble tetrazolium salt

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Inhibition of constitutively activated Stat3 correlates with altered Bcl-2/Bax expression and induction of apoptosis in mycosis fungoides tumor cells

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Identification and characterization of a constitutively active STAT5 mutant that promotes cell proliferation

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An alternative pathway for STAT activation that is mediated by the direct interaction between JAK and STAT.

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Src kinases and not JAKs activate STATs during IL-3 induced myeloid cell proliferation

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Activation of STAT3 by the c-Fes protein-tyrosine kinase

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Negative regulation of the JAK/STAT pathway

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Liver degeneration and lymphoid deficiencies in mice lacking suppressor of cytokine signaling-1.

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STAT3beta, a splice variant of transcription factor STAT3, is a dominant negative regulator of transcription

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Expression of granulocyte colony-stimulating factor- and granulocyte-macrophage colony-stimulating factor-associated signal transduction proteins of the JAK/STAT pathway in normal granulopoiesis and in blast cells of acute myelogenous leukemia

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Tyrosyl phosphorylation and DNA binding activity of signal transducers and activators of transcription (STAT) proteins in hematopoietic cell lines transformed by Bcr/Abl

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Activation of a Drosophila Janus kinase (JAK) causes hematopoietic neoplasia and developmental defects.

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Tyrphostins. II. Heterocyclic and .alpha.-substituted benzylidenemalononitrile tyrphostins as potent inhibitors of EGF receptor and ErbB2/neu tyrosine kinases

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Tyrphostins I: synthesis and biological activity of protein tyrosine kinase inhibitors

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JAK2 tyrosine kinase inhibitor tyrphostin AG490 downregulates the mitogen-activated protein kinase (MAPK) and signal transducer and activator of transcription (STAT) pathways and induces apoptosis in myeloma cells

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A central role for Stat3 in IL-6-induced regulation of growth and differentiation in M1 leukemia cells

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Stat5 is essential for the myelo- and lymphoproliferative disease induced by TEL/JAK2

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10.1034/J.1600-0609.2001.T01-1-00385.X

Identifiers

DOI

1 reference

10.1034/J.1600-0609.2001.00385.X

4843970

1 reference

3489079

3489079

1 reference

3489079

4843970

1 reference

1 reference