(Q28207830)

2

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Joe Wooters

3

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Yongchang Qiu

4

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Jack Wang

5

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Lih-Ling Lin

Journal of Biological Chemistry

6

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language of work or name

English

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publication date

18 April 2003

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published in

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volume

278

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issue

16

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page(s)

14363-9

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cites work

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Expression of the lymphotoxin beta receptor on follicular stromal cells in human lymphoid tissues

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Abnormal development of peripheral lymphoid organs in mice deficient in lymphotoxin

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Distinct roles in lymphoid organogenesis for lymphotoxins alpha and beta revealed in lymphotoxin beta-deficient mice

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Elimination of colonic patches with lymphotoxin beta receptor-Ig prevents Th2 cell-type colitis

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A critical role for lymphotoxin-beta receptor in the development of diabetes in nonobese diabetic mice

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Reversal of spontaneous autoimmune insulitis in nonobese diabetic mice by soluble lymphotoxin receptor

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Lymphotoxin beta receptor triggering induces activation of the nuclear factor kappaB transcription factor in some cell types

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Signaling through the lymphotoxin beta receptor induces the death of some adenocarcinoma tumor lines

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The cytoplasmic domain of the lymphotoxin-beta receptor mediates cell death in HeLa cells

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A novel domain within the 55 kd TNF receptor signals cell death

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A novel family of putative signal transducers associated with the cytoplasmic domain of the 75 kDa tumor necrosis factor receptor

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TRADD-TRAF2 and TRADD-FADD interactions define two distinct TNF receptor 1 signal transduction pathways

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TRAF family proteins interact with the common neurotrophin receptor and modulate apoptosis induction

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TRAF5, an activator of NF-kappaB and putative signal transducer for the lymphotoxin-beta receptor

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Lymphotoxin-beta receptor signaling complex: role of tumor necrosis factor receptor-associated factor 3 recruitment in cell death and activation of nuclear factor kappaB

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Cleavage of BID by caspase 8 mediates the mitochondrial damage in the Fas pathway of apoptosis

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Bid, a Bcl2 interacting protein, mediates cytochrome c release from mitochondria in response to activation of cell surface death receptors

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TRAIL-induced apoptosis requires Bax-dependent mitochondrial release of Smac/DIABLO.

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Rapid kinetics of tBid-induced cytochrome c and Smac/DIABLO release and mitochondrial depolarization

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Smac, a mitochondrial protein that promotes cytochrome c-dependent caspase activation by eliminating IAP inhibition

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Identification of DIABLO, a mammalian protein that promotes apoptosis by binding to and antagonizing IAP proteins

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Dominant negative mutants of TRAF3 reveal an important role for the coiled coil domains in cell death signaling by the lymphotoxin-beta receptor

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Automated protein preparation techniques using a digest robot

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A newly identified member of the tumor necrosis factor receptor superfamily with a wide tissue distribution and involvement in lymphocyte activation

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The TNFR2-TRAF signaling complex contains two novel proteins related to baculoviral inhibitor of apoptosis proteins

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Structural and biochemical basis of apoptotic activation by Smac/DIABLO

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Structural basis for binding of Smac/DIABLO to the XIAP BIR3 domain

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Structural basis of IAP recognition by Smac/DIABLO

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NF-kappaB-inducing kinase is dispensable for activation of NF-kappaB in inflammatory settings but essential for lymphotoxin beta receptor activation of NF-kappaB in primary human fibroblasts

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The tumor necrosis factor receptor 2 signal transducers TRAF2 and c-IAP1 are components of the tumor necrosis factor receptor 1 signaling complex

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cIAP-1, but not XIAP, is cleaved by caspases during the apoptosis induced by TGF-beta in fetal rat hepatocytes

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c-IAP1 is cleaved by caspases to produce a proapoptotic C-terminal fragment

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The inhibitor of apoptosis protein-binding domain of Smac is not essential for its proapoptotic activity

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IFN-gamma induces the apoptosis of WEHI 279 and normal pre-B cell lines by expressing direct inhibitor of apoptosis protein binding protein with low pI.

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Contributions of mass spectrometry to peptide and protein structure

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