(Q28211116)

1

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Shaohua Li

2

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J Geoffrey Pickering

3

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Stephen M Sims

American Journal of Physiology Heart and Circulatory Physiology

4

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language of work or name

English

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publication date

June 2003

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published in

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volume

284

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issue

6

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page(s)

H2325-34

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cites work

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Kir2.1 encodes the inward rectifier potassium channel in rat arterial smooth muscle cells

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Functional evidence for an inward rectifier potassium current in rat renal afferent arterioles

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Role of inwardly rectifying K(+) channels in K(+)-induced cerebral vasodilatation in vivo

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The molecular composition of K(ATP) channels in human pulmonary artery smooth muscle cells and their modulation by growth

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Nifedipine blocks Ca2+ store refilling through a pathway not involving L-type Ca2+ channels in rabbit arteriolar smooth muscle

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Inward rectification in submucosal arterioles of guinea-pig ileum

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K+ is an endothelium-derived hyperpolarizing factor in rat arteries.

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Human myoblast fusion requires expression of functional inward rectifier Kir2.1 channels

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Use of cultured airway myocytes for study of airway smooth muscle

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The action potential and underlying ionic currents in proximal rat middle cerebral arterioles

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Ionic basis of the resting potential of submucosal arterioles in the ileum of the guinea-pig

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Functional receptor-channel coupling compared in contractile and proliferative human vascular smooth muscle

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Inwardly rectifying potassium current in rabbit osteoclasts: a whole-cell and single-channel study

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Control of rectification and permeation by two distinct sites after the second transmembrane region in Kir2.1 K+ channel

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Innate diversity of adult human arterial smooth muscle cells: cloning of distinct subtypes from the internal thoracic artery

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Evidence from a novel human cell clone that adult vascular smooth muscle cells can convert reversibly between noncontractile and contractile phenotypes.

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Potassium dilates rat cerebral arteries by two independent mechanisms

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Molecular cloning and characterization of the intermediate-conductance Ca(2+)-activated K(+) channel in vascular smooth muscle: relationship between K(Ca) channel diversity and smooth muscle cell function

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Inward rectifier potassium channels

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Regulation of differentiation of vascular smooth muscle cells

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Mutations in Kir2.1 cause the developmental and episodic electrical phenotypes of Andersen's syndrome

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Sustained membrane depolarization and pulmonary artery smooth muscle cell proliferation.

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Characterisation of Kir2.0 proteins in the rat cerebellum and hippocampus by polyclonal antibodies

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Potassium channel inhibition reduces cell proliferation in the GH3 pituitary cell line

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Targeted disruption of Kir2.1 and Kir2.2 genes reveals the essential role of the inwardly rectifying K(+) current in K(+)-mediated vasodilation.

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