(Q28213876)

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Mitochondrial K(ATP) channel activation reduces anoxic injury by restoring mitochondrial membrane potential

scientific article (publication date: September 2001)

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title

Mitochondrial K(ATP) channel activation reduces anoxic injury by restoring mitochondrial membrane potential (English)

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1 reference

M Xu

1

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Y Wang

series ordinal

2

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A Ayub

series ordinal

3

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M Ashraf

series ordinal

4

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language of work or name

English

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publication date

September 2001

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published in

American Journal of Physiology Heart and Circulatory Physiology

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volume

281

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page(s)

H1295-303

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issue

3

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cites work

Cardiomyocyte apoptosis induced by Galphaq signaling is mediated by permeability transition pore formation and activation of the mitochondrial death pathway.

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reference URL

https://api.crossref.org/works/10.1152%2FAJPHEART.2001.281.3.H1295

retrieved

21 January 2018

Mitochondria and calcium: from cell signalling to cell death

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Crossref

reference URL

https://api.crossref.org/works/10.1152%2FAJPHEART.2001.281.3.H1295

retrieved

21 January 2018

Cardioprotective effect of diazoxide and its interaction with mitochondrial ATP-sensitive K+ channels. Possible mechanism of cardioprotection.

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Crossref

reference URL

https://api.crossref.org/works/10.1152%2FAJPHEART.2001.281.3.H1295

retrieved

21 January 2018

Direct effects of diazoxide on mitochondria in pancreatic B-cells and on isolated liver mitochondria

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reference URL

https://api.crossref.org/works/10.1152%2FAJPHEART.2001.281.3.H1295

retrieved

21 January 2018

Contribution of cytosolic ionic and energetic milieu change to ischemia- and reperfusion-induced injury in guinea pig heart: fluorometry and nuclear magnetic resonance studies

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reference URL

https://api.crossref.org/works/10.1152%2FAJPHEART.2001.281.3.H1295

retrieved

21 January 2018

Preservation of mitochondrial function by diazoxide during sustained ischaemia in the rat heart

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reference URL

https://api.crossref.org/works/10.1152%2FAJPHEART.2001.281.3.H1295

retrieved

21 January 2018

Effect of reperfusion late in the phase of reversible ischemic injury. Changes in cell volume, electrolytes, metabolites, and ultrastructure.

1 reference

stated in

Crossref

reference URL

https://api.crossref.org/works/10.1152%2FAJPHEART.2001.281.3.H1295

retrieved

21 January 2018

Mitochondrial electron transport chain activity, but not ATP synthesis, is required for drug-induced apoptosis in human leukaemic cells: a possible novel mechanism of regulating drug resistance.

1 reference

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reference URL

https://api.crossref.org/works/10.1152%2FAJPHEART.2001.281.3.H1295

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21 January 2018

The release of cytochrome c from mitochondria: a primary site for Bcl-2 regulation of apoptosis

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https://api.crossref.org/works/10.1152%2FAJPHEART.2001.281.3.H1295

retrieved

21 January 2018

Mitochondrial dysfunction in the pathogenesis of necrotic and apoptotic cell death

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reference URL

https://api.crossref.org/works/10.1152%2FAJPHEART.2001.281.3.H1295

retrieved

21 January 2018

Mitochondrial ATP-dependent potassium channels: novel effectors of cardioprotection?

1 reference

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reference URL

https://api.crossref.org/works/10.1152%2FAJPHEART.2001.281.3.H1295

retrieved

21 January 2018

Morphine mimics preconditioning via free radical signals and mitochondrial K(ATP) channels in myocytes

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Crossref

reference URL

https://api.crossref.org/works/10.1152%2FAJPHEART.2001.281.3.H1295

retrieved

21 January 2018

J-aggregate formation of a carbocyanine as a quantitative fluorescent indicator of membrane potential

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https://api.crossref.org/works/10.1152%2FAJPHEART.2001.281.3.H1295

retrieved

21 January 2018

On the voltage dependence of the mitochondrial permeability transition pore. A critical appraisal.

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reference URL

https://api.crossref.org/works/10.1152%2FAJPHEART.2001.281.3.H1295

retrieved

21 January 2018

Mitochondrial membrane potential changes in osteoblasts treated with parathyroid hormone and estradiol.

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https://api.crossref.org/works/10.1152%2FAJPHEART.2001.281.3.H1295

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21 January 2018

Dual roles of mitochondrial K(ATP) channels in diazoxide-mediated protection in isolated rabbit hearts

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reference URL

https://api.crossref.org/works/10.1152%2FAJPHEART.2001.281.3.H1295

retrieved

21 January 2018

Activation of mitochondrial ATP-sensitive K(+) channel for cardiac protection against ischemic injury is dependent on protein kinase C activity.

1 reference

stated in

Crossref

reference URL

https://api.crossref.org/works/10.1152%2FAJPHEART.2001.281.3.H1295

retrieved

21 January 2018

Calcium preconditioning inhibits mitochondrial permeability transition and apoptosis

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reference URL

https://api.crossref.org/works/10.1152%2FAJPHEART.2001.281.3.H1295

retrieved

21 January 2018

Mitochondrial control of nuclear apoptosis.

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Crossref

reference URL

https://api.crossref.org/works/10.1152%2FAJPHEART.2001.281.3.H1295

retrieved

21 January 2018

Identifiers

DOI

10.1152/AJPHEART.2001.281.3.H1295

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PubMed ID

11514300

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(Q28213876)

Mitochondrial K(ATP) channel activation reduces anoxic injury by restoring mitochondrial membrane potential

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