(Q28255924)

1

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author name string

Daniel R Englmann

2

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Karim R Sultan

3

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Markus Sauer

4

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Thomas Eschenhagen

Basic Research in Cardiology

5

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language of work or name

English

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publication date

January 2012

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published in

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volume

107

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issue

1

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page(s)

235

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cites work

Ubiquitin-proteasome system impairment caused by a missense cardiac myosin-binding protein C mutation and associated with cardiac dysfunction in hypertrophic cardiomyopathy

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Distinct sarcomeric substrates are responsible for protein kinase D-mediated regulation of cardiac myofilament Ca2+ sensitivity and cross-bridge cycling

21 January 2018

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Ubiquitin-mediated degradation of the proapoptotic active form of bid. A functional consequence on apoptosis induction

21 January 2018

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Asymmetric septal hypertrophy in heterozygous cMyBP-C null mice

21 January 2018

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The ubiquitin-proteasome system and nonsense-mediated mRNA decay in hypertrophic cardiomyopathy

21 January 2018

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Intrasarcoplasmic amyloidosis impairs proteolytic function of proteasomes in cardiomyocytes by compromising substrate uptake

21 January 2018

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Novel role for p90 ribosomal S6 kinase in the regulation of cardiac myofilament phosphorylation

21 January 2018

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Autophagy in the cardiovascular system

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Linking of autophagy to ubiquitin-proteasome system is important for the regulation of endoplasmic reticulum stress and cell viability

21 January 2018

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Decreased phosphorylation levels of cardiac myosin-binding protein-C in human and experimental heart failure

21 January 2018

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COOH-terminal truncated cardiac myosin-binding protein C mutants resulting from familial hypertrophic cardiomyopathy mutations exhibit altered expression and/or incorporation in fetal rat cardiomyocytes

21 January 2018

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The ubiquitin-proteasome proteolytic pathway: destruction for the sake of construction

21 January 2018

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Proteasome activation during cardiac hypertrophy by the chaperone H11 Kinase/Hsp22.

21 January 2018

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Regulation of cell proliferation by the E2F transcription factors

21 January 2018

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https://api.crossref.org/works/10.1007%2FS00395-011-0235-3

How proteolysis drives the cell cycle

21 January 2018

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https://api.crossref.org/works/10.1007%2FS00395-011-0235-3

Role of ubiquitin- and Ubl-binding proteins in cell signaling

21 January 2018

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A role for ubiquitin in selective autophagy

21 January 2018

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Protein kinase C alpha and epsilon phosphorylation of troponin and myosin binding protein C reduce Ca2+ sensitivity in human myocardium

21 January 2018

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Myocytes die by multiple mechanisms in failing human hearts

21 January 2018

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Autophagy in the Pathogenesis of Disease

21 January 2018

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Bax degradation by the ubiquitin/proteasome-dependent pathway: involvement in tumor survival and progression

21 January 2018

1 reference

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Atrogin-1/muscle atrophy F-box inhibits calcineurin-dependent cardiac hypertrophy by participating in an SCF ubiquitin ligase complex

21 January 2018

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Enhancement of proteasomal function protects against cardiac proteinopathy and ischemia/reperfusion injury in mice

21 January 2018

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A transgenic mouse model of the ubiquitin/proteasome system

21 January 2018

1 reference

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Evidence from human myectomy samples that MYBPC3 mutations cause hypertrophic cardiomyopathy through haploinsufficiency

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00395-011-0235-3

Changes in cardiac contractility related to calcium-mediated changes in phosphorylation of myosin-binding protein C.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00395-011-0235-3

Atrogin-1 and MuRF1 regulate cardiac MyBP-C levels via different mechanisms

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00395-011-0235-3

The ubiquitin-proteasome system in cardiac dysfunction

21 January 2018

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https://api.crossref.org/works/10.1007%2FS00395-011-0235-3

Autophagy fights disease through cellular self-digestion

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00395-011-0235-3

A newly created splice donor site in exon 25 of the MyBP-C gene is responsible for inherited hypertrophic cardiomyopathy with incomplete disease penetrance

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00395-011-0235-3

The ubiquitin-proteasome pathway is required for processing the NF-kappa B1 precursor protein and the activation of NF-kappa B

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00395-011-0235-3

p62/SQSTM1 binds directly to Atg8/LC3 to facilitate degradation of ubiquitinated protein aggregates by autophagy

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00395-011-0235-3

Ubiquitin proteasome dysfunction in human hypertrophic and dilated cardiomyopathies

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00395-011-0235-3

Atrophy, hypertrophy, and hypoxemia induce transcriptional regulators of the ubiquitin proteasome system in the rat heart

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00395-011-0235-3

Hypertrophic cardiomyopathy: distribution of disease genes, spectrum of mutations, and implications for a molecular diagnosis strategy

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00395-011-0235-3

Inhibitors of the proteasome block the degradation of most cell proteins and the generation of peptides presented on MHC class I molecules

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00395-011-0235-3

Novel splice donor site mutation in the cardiac myosin-binding protein-C gene in familial hypertrophic cardiomyopathy. Characterization Of cardiac transcript and protein

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00395-011-0235-3

Impairment of the ubiquitin-proteasome system by truncated cardiac myosin binding protein C mutants

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00395-011-0235-3

The ubiquitin-proteasome system in cardiomyopathies

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00395-011-0235-3

Cardiac myosin-binding protein C in hypertrophic cardiomyopathy: mechanisms and therapeutic opportunities

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00395-011-0235-3

The ubiquitin-proteasome system in cardiac proteinopathy: a quality control perspective

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00395-011-0235-3

Autophagosome formation and molecular mechanism of autophagy.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00395-011-0235-3

Intracellular protein aggregation is a proximal trigger of cardiomyocyte autophagy

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00395-011-0235-3

Cardiac myosin-binding protein C mutations and hypertrophic cardiomyopathy: haploinsufficiency, deranged phosphorylation, and cardiomyocyte dysfunction

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00395-011-0235-3

Nonsense-mediated mRNA decay and ubiquitin-proteasome system regulate cardiac myosin-binding protein C mutant levels in cardiomyopathic mice

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00395-011-0235-3

Humoral anti-proteasomal autoimmunity in dilated cardiomyopathy

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00395-011-0235-3

Lack of evidence for a pathogenic role of proteasome-directed autoimmunity in dilated cardiomyopathy

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00395-011-0235-3

Hyperubiquitination of proteins in dilated cardiomyopathy

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00395-011-0235-3

In vivo modeling of myosin binding protein C familial hypertrophic cardiomyopathy

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00395-011-0235-3

Cardiac autophagy is a maladaptive response to hemodynamic stress

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00395-011-0235-3

The ubiquitin-proteasome system: focus on the heart.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS00395-011-0235-3