(Q28264600)

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The autoimmune lymphoproliferative syndrome: an experiment of nature involving lymphocyte apoptosis

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title

The autoimmune lymphoproliferative syndrome: an experiment of nature involving lymphocyte apoptosis (English)

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main subject

autoimmune lymphoproliferative syndrome

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author name string

Thomas A Fleisher

series ordinal

1

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language of work or name

English

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publication date

2008

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published in

Immunologic Research

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volume

40

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issue

1

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page(s)

87-92

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cites work

Pathological findings in human autoimmune lymphoproliferative syndrome

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stated in

Crossref

reference URL

https://api.crossref.org/works/10.1007%2FS12026-007-8001-1

retrieved

21 January 2018

Autoimmune lymphoproliferative syndromes: genetic defects of apoptosis pathways

1 reference

stated in

PubMed

reference URL

https://pubmed.ncbi.nlm.nih.gov/18193364

retrieved

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Fas and Fas ligand: lpr and gld mutations

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stated in

PubMed

reference URL

https://pubmed.ncbi.nlm.nih.gov/18193364

retrieved

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Autoimmune lymphoproliferative syndrome.

1 reference

stated in

PubMed

reference URL

https://pubmed.ncbi.nlm.nih.gov/18193364

retrieved

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Correction of autoimmune lymphoproliferative syndrome by bone marrow transplantation

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stated in

PubMed

reference URL

https://pubmed.ncbi.nlm.nih.gov/18193364

retrieved

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Dominant interfering Fas gene mutations impair apoptosis in a human autoimmune lymphoproliferative syndrome

1 reference

stated in

PubMed

reference URL

https://pubmed.ncbi.nlm.nih.gov/18193364

retrieved

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

A composite picture of TcR alpha/beta(+) CD4(-)CD8(-) T Cells (alpha/beta-DNTCs) in humans with autoimmune lymphoproliferative syndrome

1 reference

stated in

PubMed

reference URL

https://pubmed.ncbi.nlm.nih.gov/18193364

retrieved

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Immunophenotypic profiles in families with autoimmune lymphoproliferative syndrome

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stated in

PubMed

reference URL

https://pubmed.ncbi.nlm.nih.gov/18193364

retrieved

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

The development of lymphomas in families with autoimmune lymphoproliferative syndrome with germline Fas mutations and defective lymphocyte apoptosis

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stated in

PubMed

reference URL

https://pubmed.ncbi.nlm.nih.gov/18193364

retrieved

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Fas gene mutations in the Canale-Smith syndrome, an inherited lymphoproliferative disorder associated with autoimmunity

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stated in

PubMed

reference URL

https://pubmed.ncbi.nlm.nih.gov/18193364

retrieved

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Clincal, immunologic, and genetic features of an autoimmune lymphoproliferative syndrome associated with abnormal lymphocyte apoptosis

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stated in

PubMed

reference URL

https://pubmed.ncbi.nlm.nih.gov/18193364

retrieved

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Autoimmune lymphoproliferative syndrome with defective Fas: genotype influences penetrance

1 reference

stated in

PubMed

reference URL

https://pubmed.ncbi.nlm.nih.gov/18193364

retrieved

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Fas ligand mutation in a patient with systemic lupus erythematosus and lymphoproliferative disease

1 reference

stated in

PubMed

reference URL

https://pubmed.ncbi.nlm.nih.gov/18193364

retrieved

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Autoimmune lymphoproliferative syndrome with somatic Fas mutations

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stated in

PubMed

reference URL

https://pubmed.ncbi.nlm.nih.gov/18193364

retrieved

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Mutations in Fas associated with human lymphoproliferative syndrome and autoimmunity

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stated in

PubMed

reference URL

https://pubmed.ncbi.nlm.nih.gov/18193364

retrieved

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Autoimmune lymphoproliferative syndrome (ALPS) in a child from consanguineous parents: a dominant or recessive disease?

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stated in

PubMed

reference URL

https://pubmed.ncbi.nlm.nih.gov/18193364

retrieved

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Increases in circulating and lymphoid tissue interleukin-10 in autoimmune lymphoproliferative syndrome are associated with disease expression

1 reference

stated in

PubMed

reference URL

https://pubmed.ncbi.nlm.nih.gov/18193364

retrieved

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Identifiers

DOI

10.1007/S12026-007-8001-1

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Dimensions Publication ID

1013504800

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PubMed publication ID

18193364

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(Q28264600)

The autoimmune lymphoproliferative syndrome: an experiment of nature involving lymphocyte apoptosis

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