(Q28278802)

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Neurobiology: a channel sets the gain on pain

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Neurobiology: a channel sets the gain on pain (English)

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Stephen G Waxman

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language of work or name

English

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publication date

14 December 2006

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published in

Nature

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volume

444

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issue

7121

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page(s)

831-2

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cites work

An SCN9A channelopathy causes congenital inability to experience pain

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Crossref

reference URL

https://api.crossref.org/works/10.1038%2F444831A

retrieved

21 January 2018

Identification of PN1, a predominant voltage-dependent sodium channel expressed principally in peripheral neurons

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Crossref

reference URL

https://api.crossref.org/works/10.1038%2F444831A

retrieved

21 January 2018

Changes in the expression of tetrodotoxin-sensitive sodium channels within dorsal root ganglia neurons in inflammatory pain

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reference URL

https://api.crossref.org/works/10.1038%2F444831A

retrieved

21 January 2018

Nociceptor-specific gene deletion reveals a major role for Nav1.7 (PN1) in acute and inflammatory pain

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Crossref

reference URL

https://api.crossref.org/works/10.1038%2F444831A

retrieved

21 January 2018

The primary erythermalgia-susceptibility gene is located on chromosome 2q31-32.

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https://api.crossref.org/works/10.1038%2F444831A

retrieved

21 January 2018

Mutations in SCN9A, encoding a sodium channel alpha subunit, in patients with primary erythermalgia

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reference URL

https://api.crossref.org/works/10.1038%2F444831A

retrieved

21 January 2018

Erythermalgia: molecular basis for an inherited pain syndrome

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reference URL

https://api.crossref.org/works/10.1038%2F444831A

retrieved

21 January 2018

Electrophysiological properties of mutant Nav1.7 sodium channels in a painful inherited neuropathy.

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reference URL

https://api.crossref.org/works/10.1038%2F444831A

retrieved

21 January 2018

Gain-of-function mutation in Nav1.7 in familial erythromelalgia induces bursting of sensory neurons

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Crossref

reference URL

https://api.crossref.org/works/10.1038%2F444831A

retrieved

21 January 2018

A single sodium channel mutation produces hyper- or hypoexcitability in different types of neurons

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reference URL

https://api.crossref.org/works/10.1038%2F444831A

retrieved

21 January 2018

Impaired firing and cell-specific compensation in neurons lacking nav1.6 sodium channels.

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reference URL

https://api.crossref.org/works/10.1038%2F444831A

retrieved

21 January 2018

Slow closed-state inactivation: a novel mechanism underlying ramp currents in cells expressing the hNE/PN1 sodium channel.

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Crossref

reference URL

https://api.crossref.org/works/10.1038%2F444831A

retrieved

7 January 2021

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inferred from DOI database lookup

Sensory and electrophysiological properties of guinea-pig sensory neurones expressing Nav 1.7 (PN1) Na+ channel alpha subunit protein

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stated in

Crossref

reference URL

https://api.crossref.org/works/10.1038%2F444831A

retrieved

7 January 2021

based on heuristic

inferred from DOI database lookup

(Q28278802)

Neurobiology: a channel sets the gain on pain

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