(Q28298567)

1

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Rhiana Lau

2

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Patrick W Hein

3

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J Michael Shipley

4

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Gerry Weinmaster

Journal of Biological Chemistry

5

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language of work or name

English

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publication date

14 April 2006

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published in

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volume

281

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issue

15

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page(s)

10089-97

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cites work

Notch signaling: cell fate control and signal integration in development

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M600298200

The Notch1 receptor is cleaved constitutively by a furin-like convertase

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M600298200

Calcium depletion dissociates and activates heterodimeric notch receptors

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M600298200

Ligand-induced signaling in the absence of furin processing of Notch1.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M600298200

Notch signaling: from the outside in

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M600298200

Notch signal transduction: a real rip and more

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M600298200

Activating mutations of NOTCH1 in human T cell acute lymphoblastic leukemia

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M600298200

Notch subunit heterodimerization and prevention of ligand-independent proteolytic activation depend, respectively, on a novel domain and the LNR repeats

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M600298200

Oncogenic forms of NOTCH1 lacking either the primary binding site for RBP-Jkappa or nuclear localization sequences retain the ability to associate with RBP-Jkappa and activate transcription

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M600298200

The extracellular matrix protein MAGP-2 interacts with Jagged1 and induces its shedding from the cell surface

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M600298200

Fibrillin, a new 350-kD glycoprotein, is a component of extracellular microfibrils

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M600298200

Further characterization of proteins associated with elastic fiber microfibrils including the molecular cloning of MAGP-2 (MP25)

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M600298200

Identification of a matrix-binding domain in MAGP1 and MAGP2 and intracellular localization of alternative splice forms

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M600298200

MAGP-2 has multiple binding regions on fibrillins and has covalent periodic association with fibrillin-containing microfibrils

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M600298200

Microfibril-associated glycoprotein-2 interacts with fibrillin-1 and fibrillin-2 suggesting a role for MAGP-2 in elastic fiber assembly

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M600298200

The microfibrillar proteins MAGP-1 and fibrillin-1 form a ternary complex with the chondroitin sulfate proteoglycan decorin

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M600298200

Microfibril-associated glycoprotein-2 specifically interacts with a range of bovine and human cell types via alphaVbeta3 integrin

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M600298200

Microfibril-associated glycoprotein-2 (MAGP-2) is specifically associated with fibrillin-containing microfibrils but exhibits more restricted patterns of tissue localization and developmental expression than its structural relative MAGP-1.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M600298200

Increased expression of type I collagen induced by microfibril-associated glycoprotein 2: novel mechanistic insights into the molecular basis of dermal fibrosis in scleroderma.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M600298200

Fringe glycosyltransferases differentially modulate Notch1 proteolysis induced by Delta1 and Jagged1

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M600298200

The divergent DSL ligand Dll3 does not activate Notch signaling but cell autonomously attenuates signaling induced by other DSL ligands

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M600298200

Notch signaling enhances survival and alters differentiation of 32D myeloblasts

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M600298200

Truncated mammalian Notch1 activates CBF1/RBPJk-repressed genes by a mechanism resembling that of Epstein-Barr virus EBNA2

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M600298200

Fibrillin-1 and -2 contain heparin-binding sites important for matrix deposition and that support cell attachment

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M600298200

Antineurogenic phenotypes induced by truncated Notch proteins indicate a role in signal transduction and may point to a novel function for Notch in nuclei

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M600298200

Signal transduction by activated mNotch: importance of proteolytic processing and its regulation by the extracellular domain.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M600298200

Making sense of latent TGFbeta activation

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M600298200

T cell acute lymphoblastic leukemia/lymphoma: a human cancer commonly associated with aberrant NOTCH1 signaling

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M600298200

Latent transforming growth factor-beta: structural features and mechanisms of activation

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M600298200

Activation of latent TGF-beta by thrombospondin-1: mechanisms and physiology

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M600298200

Domain-specific mutations in TGFB1 result in Camurati-Engelmann disease.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M600298200

Domain-specific mutations of a transforming growth factor (TGF)-beta 1 latency-associated peptide cause Camurati-Engelmann disease because of the formation of a constitutively active form of TGF-beta 1.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M600298200

DNER acts as a neuron-specific Notch ligand during Bergmann glial development

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M600298200

F3/contactin acts as a functional ligand for Notch during oligodendrocyte maturation

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M600298200

NB-3/Notch1 pathway via Deltex1 promotes neural progenitor cell differentiation into oligodendrocytes.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M600298200

The nephroblastoma overexpressed gene (NOV/ccn3) protein associates with Notch1 extracellular domain and inhibits myoblast differentiation via Notch signaling pathway

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M600298200

Vascular expression of Notch pathway receptors and ligands is restricted to arterial vessels.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M600298200

Notch function in the vasculature: insights from zebrafish, mouse and man.

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M600298200

Notch signaling in vascular morphogenesis

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M600298200

Mutations in the gene encoding the latency-associated peptide of TGF-β1 cause Camurati-Engelmann disease

21 January 2018

1 reference

https://api.crossref.org/works/10.1074%2FJBC.M600298200