(Q28373195)

English

Pimobendan inhibits the production of proinflammatory cytokines and gene expression of inducible nitric oxide synthase in a murine model of viral myocarditis

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Pimobendan inhibits the production of proinflammatory cytokines and gene expression of inducible nitric oxide synthase in a murine model of viral myocarditis (English)

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author name string

A Iwasaki

1

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A Matsumori

2

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T Yamada

3

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T Shioi

4

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W Wang

5

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K Ono

6

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R Nishio

7

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M Okada

8

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S Sasayama

9

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language of work or name

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publication date

April 1999

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Journal of the American College of Cardiology

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33

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5

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1400-7

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Pimobendane (UD-CG 115 BS) in the treatment of severe congestive heart failure. An acute haemodynamic cross-over and double-blind study with two different doses

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Cardiovascular effects and plasma level profile of pimobendan (UD-CG 115 BS) and its metabolite UD-CG 212 in patients with congestive heart failure after single and repeated oral dosing

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Long-term oral administration of amrinone for congestive heart failure: lack of efficacy in a multicenter controlled trial

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Long term survival of class IV heart failure patients treated with oral amrinone

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Multicenter trial of oral enoximone in patients with moderate to moderately severe congestive heart failure. Lack of benefit compared with placebo. Enoximone Multicenter Trial Group

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Effect of oral milrinone on mortality in severe chronic heart failure. The PROMISE Study Research Group

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Beneficial effects of pimobendan on exercise tolerance and quality of life in patients with heart failure. Results of a multicenter trial. The Pimobendan Multicenter Research Group

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Clinical effects of long-term administration of pimobendan in patients with moderate congestive heart failure.

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Contribution of cAMP-phosphodiesterase inhibition and sensitization of the contractile proteins for calcium to the inotropic effect of pimobendan in the failing human myocardium

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Immune modulation of cardiac function: a new frontier

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Increased circulating cytokines in patients with myocarditis and cardiomyopathy

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Cytokines in Acute Myocardial Infarction

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Serum interleukin 6 levels become elevated in acute myocardial infarction

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Monocyte-related cytokines in acute myocardial infarction

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Elevated circulating levels of tumor necrosis factor in severe chronic heart failure

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Increased concentrations of tumour necrosis factor in "cachectic" patients with severe chronic heart failure

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An animal model of congestive (dilated) cardiomyopathy: dilatation and hypertrophy of the heart in the chronic stage in DBA/2 mice with myocarditis caused by encephalomyocarditis virus

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Negative inotropic effects of cytokines on the heart mediated by nitric oxide

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Myocardial contractile dysfunction in the systemic inflammatory response syndrome: role of a cytokine-inducible nitric oxide synthase in cardiac myocytes.

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Inotropic agents differentially inhibit the induction of nitric oxide synthase by endotoxin in cultured macrophages

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10.1016/S0735-1097(98)00692-5

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