(Q28505578)

24 January 2017

title

Loss of Smad3 gives rise to poor soft callus formation and accelerates early fracture healing (English)

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24 January 2017

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GOA release 2020-03-11

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Motohisa Kawakatsu

1

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24 January 2017

Seiji Kanno

2

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24 January 2017

Ting Gui

3

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24 January 2017

Zhibo Gai

4

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24 January 2017

Shunji Itoh

5

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24 January 2017

Hiroyuki Tanishima

6

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24 January 2017

Kosuke Oikawa

7

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24 January 2017

Yasuteru Muragaki

8

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24 January 2017

publication date

1 February 2011

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Experimental and Molecular Pathology

24 January 2017

published in

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24 January 2017

volume

90

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24 January 2017

issue

1

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24 January 2017

page(s)

107–115

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Mice lacking Smad3 show accelerated wound healing and an impaired local inflammatory response

24 January 2017

cites work

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The influence of mechanical conditions on the healing of experimental fractures in the rabbit: a microscopical study

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Smads as transcriptional co-modulators

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TGF-beta regulates the mechanical properties and composition of bone matrix

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TGF-beta 1 induces bone closure of skull defects: temporal dynamics of bone formation in defects exposed to rhTGF-beta 1.

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Role of active and latent transforming growth factor beta in bone formation

7 January 2021

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The loss of Smad3 results in a lower rate of bone formation and osteopenia through dysregulation of osteoblast differentiation and apoptosis

7 January 2021

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Transforming growth factor beta in fracture repair

7 January 2021

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Differential temporal expression of members of the transforming growth factor beta superfamily during murine fracture healing

7 January 2021

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Smad-dependent and Smad-independent pathways in TGF-beta family signalling

7 January 2021

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Smad3 induces chondrogenesis through the activation of SOX9 via CREB-binding protein/p300 recruitment

7 January 2021

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7 January 2021

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7 January 2021

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Transforming growth factor-beta and the initiation of chondrogenesis and osteogenesis in the rat femur

7 January 2021

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Targeted disruption of Cbfa1 results in a complete lack of bone formation owing to maturational arrest of osteoblasts

7 January 2021

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Cell proliferation and apoptosis during fracture healing.

7 January 2021

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Smad3-deficient chondrocytes have enhanced BMP signaling and accelerated differentiation

7 January 2021

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Placental growth factor mediates mesenchymal cell development, cartilage turnover, and bone remodeling during fracture repair

7 January 2021

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Transcriptional control by the TGF-beta/Smad signaling system

7 January 2021

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Local injection of TGF-beta increases the strength of tibial fractures in the rat

7 January 2021

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Smad3: a key player in pathogenetic mechanisms dependent on TGF-beta

7 January 2021

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Activations of ERK1/2 and JNK by transforming growth factor beta negatively regulate Smad3-induced alkaline phosphatase activity and mineralization in mouse osteoblastic cells

7 January 2021

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Menin is required for bone morphogenetic protein 2- and transforming growth factor beta-regulated osteoblastic differentiation through interaction with Smads and Runx2

7 January 2021

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TGF-beta signaling is essential for joint morphogenesis.

7 January 2021

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Opposite effects of bone morphogenetic protein-2 and transforming growth factor-beta1 on osteoblast differentiation

7 January 2021

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Molecular aspects of fracture healing: which are the important molecules?

7 January 2021

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Differential effects of transforming growth factor-beta on the synthesis of extracellular matrix proteins by normal fetal rat calvarial bone cell populations

7 January 2021

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Targeted disruption of SMAD3 results in impaired mucosal immunity and diminished T cell responsiveness to TGF-beta

7 January 2021

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TGF-beta, BMPS, and their signal transducing mediators, Smads, in rat fracture healing

7 January 2021

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10.1016/J.YEXMP.2010.10.011

7 January 2021

Identifiers

DOI

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