(Q28565373)

English

Activation of TGF-beta1-TAK1-p38 MAPK pathway in spared cardiomyocytes is involved in left ventricular remodeling after myocardial infarction in rats

scientific journal article

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scholarly article

1 reference

31 January 2017

Activation of TGF-beta1-TAK1-p38 MAPK pathway in spared cardiomyocytes is involved in left ventricular remodeling after myocardial infarction in rats (English)

1 reference

31 January 2017

myocardial infarction

1 reference

based on heuristic

inferred from title

ventricular remodeling

1 reference

based on heuristic

inferred from title

author name string

Madoka Matsumoto-Ida

1

1 reference

31 January 2017

Yoshihito Takimoto

2

1 reference

31 January 2017

Takeshi Aoyama

3

1 reference

31 January 2017

Masaharu Akao

4

1 reference

31 January 2017

Toshihiro Takeda

5

1 reference

31 January 2017

Toru Kita

6

1 reference

31 January 2017

publication date

1 February 2006

1 reference

31 January 2017

American Journal of Physiology Heart and Circulatory Physiology

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31 January 2017

290

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31 January 2017

2

1 reference

31 January 2017

H709–715

1 reference

31 January 2017

Transforming growth factor beta in tissue fibrosis

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00186.2005

21 January 2018

Cardiac remodeling--concepts and clinical implications: a consensus paper from an international forum on cardiac remodeling. Behalf of an International Forum on Cardiac Remodeling

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00186.2005

21 January 2018

Smad-dependent and Smad-independent pathways in TGF-beta family signalling

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00186.2005

21 January 2018

TGF-beta signalling from cell membrane to nucleus through SMAD proteins

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00186.2005

21 January 2018

TGFbeta signaling: receptors, transducers, and Mad proteins.

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00186.2005

21 January 2018

A novel kinase cascade mediated by mitogen-activated protein kinase kinase 6 and MKK3

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00186.2005

21 January 2018

Inflammatory cytokines and postmyocardial infarction remodeling

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00186.2005

21 January 2018

The kinase TAK1 can activate the NIK-I kappaB as well as the MAP kinase cascade in the IL-1 signalling pathway

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00186.2005

21 January 2018

Cytokine gene expression after myocardial infarction in rat hearts: possible implication in left ventricular remodeling.

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00186.2005

21 January 2018

Peptide growth factors can provoke "fetal" contractile protein gene expression in rat cardiac myocytes

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00186.2005

21 January 2018

Ventricular remodeling after myocardial infarction. Experimental observations and clinical implications

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00186.2005

21 January 2018

TGF-beta1 and angiotensin networking in cardiac remodeling

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00186.2005

21 January 2018

Alterations of beta-adrenergic signaling and cardiac hypertrophy in transgenic mice overexpressing TGF-beta(1).

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00186.2005

21 January 2018

The cellular and molecular response of cardiac myocytes to mechanical stress.

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00186.2005

21 January 2018

Functional interactions of transforming growth factor beta-activated kinase 1 with IkappaB kinases to stimulate NF-kappaB activation

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00186.2005

21 January 2018

TGF-beta signal transduction and mesangial cell fibrogenesis

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00186.2005

21 January 2018

Left ventricular remodeling after myocardial infarction: pathophysiology and therapy

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00186.2005

21 January 2018

Hypertrophic stimuli induce transforming growth factor-beta 1 expression in rat ventricular myocytes

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00186.2005

21 January 2018

Augmented expression of neuronal nitric oxide synthase in the atria parasympathetically decreases heart rate during acute myocardial infarction in rats

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00186.2005

21 January 2018

Transforming growth factor beta-1 in acute myocardial infarction in rats

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00186.2005

21 January 2018

Cardiac muscle cell hypertrophy and apoptosis induced by distinct members of the p38 mitogen-activated protein kinase family

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00186.2005

21 January 2018

Identification of a member of the MAPKKK family as a potential mediator of TGF-beta signal transduction

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00186.2005

21 January 2018

A role for the p38 mitogen-activated protein kinase pathway in myocardial cell growth, sarcomeric organization, and cardiac-specific gene expression

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00186.2005

21 January 2018

Elevation of Expression of Smads 2, 3, and 4, Decorin and TGF-βin the Chronic Phase of Myocardial Infarct Scar Healing

1 reference

https://api.crossref.org/works/10.1152%2FAJPHEART.00186.2005

21 January 2018

Identifiers

10.1152/AJPHEART.00186.2005

1 reference

Consolidated OpenCitations Corpus – April 2017

OpenCitations bibliographic resource ID

OpenCitations bibliographic resource ID

1 reference

Consolidated OpenCitations Corpus – April 2017

OpenCitations bibliographic resource ID

1 reference

Consolidated OpenCitations Corpus – April 2017

OpenCitations bibliographic resource ID

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