(Q28571831)

31 January 2017

title

Phosphatidylinositol 3-kinase/Akt signaling stimulates colonic mucosal cell survival during aging (English)

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31 January 2017

Adhip P. N. Majumdar

1

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31 January 2017

Jianhua Du

series ordinal

2

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31 January 2017

publication date

1 January 2006

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American Journal of Physiology - Gastrointestinal and Liver Physiology

31 January 2017

published in

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31 January 2017

volume

290

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31 January 2017

issue

1

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31 January 2017

page(s)

G49–55

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Gastrointestinal proliferation and aging

31 January 2017

cites work

1 reference

https://api.crossref.org/works/10.1152%2FAJPGI.00106.2005

21 January 2018

Phosphoinositide kinases

1 reference

https://api.crossref.org/works/10.1152%2FAJPGI.00106.2005

Akt phosphorylation of BAD couples survival signals to the cell-intrinsic death machinery

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Cellular survival: a play in three Akts

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Regulation of neuronal survival by the serine-threonine protein kinase Akt

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PI 3-kinase, Akt and cell survival

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Altered controls of proliferation in proximal small intestine of the senescent rat

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Phosphatidylinositol 3-kinase is required for integrin-stimulated AKT and Raf-1/mitogen-activated protein kinase pathway activation

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Matrix adhesion and Ras transformation both activate a phosphoinositide 3-OH kinase and protein kinase B/Akt cellular survival pathway

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Aging is associated with increased proliferation and decreased apoptosis in the colonic mucosa

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Serine phosphorylation of death agonist BAD in response to survival factor results in binding to 14-3-3 not BCL-X(L)

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