(Q28582087)

English

cAbl tyrosine kinase mediates reactive oxygen species- and caveolin-dependent AT1 receptor signaling in vascular smooth muscle: role in vascular hypertrophy

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scholarly article

1 reference

PubMed publication ID

31 January 2017

retracted paper

2 references

Dataset for "Continued use of retracted papers: Temporal trends in citations and (lack of) awareness of retractions shown in citation contexts in biomedicine"

Continued use of retracted papers: Temporal trends in citations and (lack of) awareness of retractions shown in citation contexts in biomedicine

cAbl tyrosine kinase mediates reactive oxygen species- and caveolin-dependent AT1 receptor signaling in vascular smooth muscle: role in vascular hypertrophy (English)

1 reference

PubMed publication ID

31 January 2017

reactive oxygen species

1 reference

based on heuristic

inferred from title

author name string

Masuko Ushio-Fukai

1

1 reference

PubMed publication ID

31 January 2017

Lian Zuo

2

1 reference

PubMed publication ID

31 January 2017

Satoshi Ikeda

3

1 reference

PubMed publication ID

31 January 2017

Taiki Tojo

4

1 reference

PubMed publication ID

31 January 2017

Nikolay A. Patrushev

5

1 reference

PubMed publication ID

31 January 2017

R. Wayne Alexander

6

1 reference

PubMed publication ID

31 January 2017

publication date

14 October 2005

1 reference

PubMed publication ID

31 January 2017

Circulation Research

1 reference

PubMed publication ID

31 January 2017

97

1 reference

PubMed publication ID

31 January 2017

8

1 reference

PubMed publication ID

31 January 2017

829–836

1 reference

PubMed publication ID

31 January 2017

A new link between the c-Abl tyrosine kinase and phosphoinositide signalling through PLC-gamma1

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Platelet-derived growth factor receptor transactivation mediates the trophic effects of angiotensin II in vivo.

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Imatinib attenuates diabetes-associated atherosclerosis

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Angiotensin II Type 1 Receptor

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Direct interaction of nerve growth factor receptor, TrkA, with non-receptor tyrosine kinase, c-Abl, through the activation loop

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c-Abl is an effector of Src for growth factor-induced c-myc expression and DNA synthesis

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Angiotensin II Activates pp60 c- src in Vascular Smooth Muscle Cells

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Regulation of vascular endothelial growth factor receptor-2 activity by caveolin-1 and plasma membrane cholesterol.

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Multiple signaling interactions of Abl and Arg kinases with the EphB2 receptor

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NAD(P)H oxidase: role in cardiovascular biology and disease

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Electroporation of pp60c−srcAntibodies Inhibits the Angiotensin II Activation of Phospholipase C-γ1 in Rat Aortic Smooth Muscle Cells

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A detergent-free method for purifying caveolae membrane from tissue culture cells

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Targeting of the c-Abl tyrosine kinase to mitochondria in the necrotic cell death response to oxidative stress

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Regulation of angiotensin II-induced JAK2 tyrosine phosphorylation: roles of SHP-1 and SHP-2

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Selective down-regulation of angiotensin II receptor type 1A signaling by protein tyrosine phosphatase SHP-2 in vascular smooth muscle cells

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Signal transduction of angiotensin II type 1 receptor through receptor tyrosine kinase

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Reactive oxygen species mediate the activation of Akt/protein kinase B by angiotensin II in vascular smooth muscle cells

1 reference

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Activation of the cytoplasmic c-Abl tyrosine kinase by reactive oxygen species

1 reference

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Interaction of the N-methyl-D-aspartic acid receptor NR2D subunit with the c-Abl tyrosine kinase

1 reference

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Association of the Abl tyrosine kinase with the Trk nerve growth factor receptor

1 reference

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Phosphorylation of tyrosine 319 of the angiotensin II type 1 receptor mediates angiotensin II-induced trans-activation of the epidermal growth factor receptor

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Regulation of F-actin-dependent processes by the Abl family of tyrosine kinases

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Chronic myeloid leukemia

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c-Abl is activated by growth factors and Src family kinases and has a role in the cellular response to PDGF

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Epidermal growth factor receptor is indispensable for c-Fos expression and protein synthesis by angiotensin II

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Novel role of ARF6 in vascular endothelial growth factor-induced signaling and angiogenesis

1 reference

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Constitutive and growth factor-regulated phosphorylation of caveolin-1 occurs at the same site (Tyr-14) in vivo: identification of a c-Src/Cav-1/Grb7 signaling cassette

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Neonatal lethality and lymphopenia in mice with a homozygous disruption of the c-abl proto-oncogene

1 reference

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Integrin regulation of c-Abl tyrosine kinase activity and cytoplasmic-nuclear transport

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p38 Mitogen-activated protein kinase is a critical component of the redox-sensitive signaling pathways activated by angiotensin II. Role in vascular smooth muscle cell hypertrophy

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Epidermal growth factor receptor transactivation by angiotensin II requires reactive oxygen species in vascular smooth muscle cells.

2 references

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Reversible oxidation and inactivation of protein tyrosine phosphatases in vivo

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Cholesterol depletion inhibits epidermal growth factor receptor transactivation by angiotensin II in vascular smooth muscle cells: role of cholesterol-rich microdomains and focal adhesions in angiotensin II signaling.

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Epidermal Growth Factor-stimulated Tyrosine Phosphorylation of Caveolin-1

1 reference

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Irbesartan but not amlodipine suppresses diabetes-associated atherosclerosis

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c-Abl is required for oxidative stress-induced phosphorylation of caveolin-1 on tyrosine 14.

1 reference

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A phosphotyrosine-dependent protein interaction screen reveals a role for phosphorylation of caveolin-1 on tyrosine 14: recruitment of C-terminal Src kinase

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Caveolin-1 is essential for activation of Rac1 and NAD(P)H oxidase after angiotensin II type 1 receptor stimulation in vascular smooth muscle cells: role in redox signaling and vascular hypertrophy

1 reference

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Integrin signaling

1 reference

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Cycling, stressed-out and nervous: cellular functions of c-Abl

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Identifiers

10.1161/01.RES.0000185322.46009.F5

1 reference

PubMed publication ID

31 January 2017

release_qil4eibaafd5tgfqqzppi5e5my

1 reference

https://api.fatcat.wiki/v0/release/qil4eibaafd5tgfqqzppi5e5my

24 November 2022

based on heuristic

mapped directly with Wikidata item

PubMed publication ID

1 reference

PubMed publication ID

31 January 2017

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