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Increased in vivo apoptosis in cells lacking mitochondrial DNA gene expression
scientific journal article
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instance of
scholarly article
1 reference
stated in
PubMed
PubMed ID
11259653
retrieved
31 January 2017
title
Increased in vivo apoptosis in cells lacking mitochondrial DNA gene expression
(English)
1 reference
stated in
PubMed
PubMed ID
11259653
retrieved
31 January 2017
main subject
apoptotic process
0 references
mitochondrial DNA
0 references
Transcription factor A, mitochondrial
1 reference
stated in
GOA release 2020-03-11
mitochondrion
1 reference
based on heuristic
inferred from title
author name string
J. Wang
series ordinal
1
1 reference
stated in
PubMed
PubMed ID
11259653
retrieved
31 January 2017
J. P. Silva
series ordinal
2
1 reference
stated in
PubMed
PubMed ID
11259653
retrieved
31 January 2017
C. M. Gustafsson
series ordinal
3
1 reference
stated in
PubMed
PubMed ID
11259653
retrieved
31 January 2017
P. Rustin
series ordinal
4
1 reference
stated in
PubMed
PubMed ID
11259653
retrieved
31 January 2017
N. G. Larsson
series ordinal
5
1 reference
stated in
PubMed
PubMed ID
11259653
retrieved
31 January 2017
language of work or name
English
0 references
publication date
27 March 2001
1 reference
stated in
PubMed
PubMed ID
11259653
retrieved
31 January 2017
published in
Proceedings of the National Academy of Sciences of the United States of America
1 reference
stated in
PubMed
PubMed ID
11259653
retrieved
31 January 2017
volume
98
1 reference
stated in
PubMed
PubMed ID
11259653
retrieved
31 January 2017
page(s)
4038–4043
1 reference
stated in
PubMed
PubMed ID
11259653
retrieved
31 January 2017
issue
7
1 reference
stated in
PubMed
PubMed ID
11259653
retrieved
31 January 2017
cites work
Smac, a mitochondrial protein that promotes cytochrome c-dependent caspase activation by eliminating IAP inhibition
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PubMed Central
reference URL
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Cytochrome c deficiency causes embryonic lethality and attenuates stress-induced apoptosis
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PubMed Central
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Role of reactive oxygen species and p53 in chromium(VI)-induced apoptosis
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Molecular characterization of mitochondrial apoptosis-inducing factor
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PubMed Central
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17 March 2017
Mitochondrial disease in superoxide dismutase 2 mutant mice
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Bid, a Bcl2 interacting protein, mediates cytochrome c release from mitochondria in response to activation of cell surface death receptors
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17 March 2017
A caspase-activated DNase that degrades DNA during apoptosis, and its inhibitor ICAD
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PubMed Central
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17 March 2017
Cytochrome c and dATP-dependent formation of Apaf-1/caspase-9 complex initiates an apoptotic protease cascade
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PubMed Central
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17 March 2017
Aconitase and mitochondrial iron-sulphur protein deficiency in Friedreich ataxia
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PubMed Central
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17 March 2017
DFF, a heterodimeric protein that functions downstream of caspase-3 to trigger DNA fragmentation during apoptosis
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PubMed Central
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17 March 2017
Apoptosis by death factor
1 reference
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PubMed Central
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17 March 2017
Mitochondria and apoptosis
1 reference
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PubMed Central
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7 April 2017
Human cells lacking mtDNA: repopulation with exogenous mitochondria by complementation
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7 April 2017
Mitochondrial transcription factor A is necessary for mtDNA maintenance and embryogenesis in mice
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7 April 2017
Metabolic depletion of ATP by fructose inversely controls CD95- and tumor necrosis factor receptor 1-mediated hepatic apoptosis
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29 September 2017
Significance of myocytes with positive DNA in situ nick end-labeling (TUNEL) in hearts with dilated cardiomyopathy: not apoptosis but DNA repair
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Suppression of a mitochondrial tRNA gene mutation phenotype associated with changes in the nuclear background
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29 September 2017
Cleavage of CAD inhibitor in CAD activation and DNA degradation during apoptosis
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29 September 2017
Intracellular adenosine triphosphate (ATP) concentration: a switch in the decision between apoptosis and necrosis
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PubMed Central
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29 September 2017
Apoptosis-associated derangement of mitochondrial function in cells lacking mitochondrial DNA.
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PubMed Central
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Calcium transport proteins in the nonfailing and failing heart: gene expression and function
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PubMed Central
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Sarcoplasmic reticulum gene expression in cardiac hypertrophy and heart failure.
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PubMed Central
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29 September 2017
Bcl-2 blocks apoptosis in cells lacking mitochondrial DNA.
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PubMed Central
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3 June 2018
Lack of oxidative phosphorylation and low mitochondrial membrane potential decrease susceptibility to apoptosis and do not modulate the protective effect of Bcl-x(L) in osteosarcoma cells.
1 reference
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Crossref
reference URL
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21 January 2018
Dilated cardiomyopathy and atrioventricular conduction blocks induced by heart-specific inactivation of mitochondrial DNA gene expression.
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Crossref
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21 January 2018
Impaired insulin secretion and beta-cell loss in tissue-specific knockout mice with mitochondrial diabetes.
1 reference
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Crossref
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21 January 2018
Murine hepatocyte apoptosis induced in vitro and in vivo by TNF-alpha requires transcriptional arrest
1 reference
stated in
PubMed
reference URL
https://pubmed.ncbi.nlm.nih.gov/11259653
retrieved
12 December 2020
based on heuristic
inferred from PubMed ID database lookup
Biochemical and molecular investigations in respiratory chain deficiencies
1 reference
stated in
PubMed
reference URL
https://pubmed.ncbi.nlm.nih.gov/11259653
retrieved
12 December 2020
based on heuristic
inferred from PubMed ID database lookup
Cytochrome c-mediated apoptosis in cells lacking mitochondrial DNA. Signaling pathway involving release and caspase 3 activation is conserved
1 reference
stated in
PubMed
reference URL
https://pubmed.ncbi.nlm.nih.gov/11259653
retrieved
12 December 2020
based on heuristic
inferred from PubMed ID database lookup
Apoptosis in mitochondrial encephalomyopathies with mitochondrial DNA mutations: a potential pathogenic mechanism
1 reference
stated in
PubMed
reference URL
https://pubmed.ncbi.nlm.nih.gov/11259653
retrieved
12 December 2020
based on heuristic
inferred from PubMed ID database lookup
Identifiers
DOI
10.1073/PNAS.061038798
1 reference
stated in
Consolidated OpenCitations Corpus – April 2017
OpenCitations bibliographic resource ID
4888902
ADS bibcode
2001PNAS...98.4038W
0 references
OpenCitations bibliographic resource ID
4888902
1 reference
stated in
Consolidated OpenCitations Corpus – April 2017
OpenCitations bibliographic resource ID
4888902
PMCID
31175
1 reference
stated in
Consolidated OpenCitations Corpus – April 2017
OpenCitations bibliographic resource ID
4888902
PubMed ID
11259653
1 reference
stated in
Consolidated OpenCitations Corpus – April 2017
OpenCitations bibliographic resource ID
4888902
ResearchGate publication ID
12072418
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