(Q28591079)

31 January 2017

title

c-fos regulates neuronal excitability and survival (English)

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FBJ osteosarcoma oncogene

31 January 2017

main subject

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GOA release 2020-03-11

author name string

Jianhua Zhang

1

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31 January 2017

Dongsheng Zhang

2

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31 January 2017

Jill Slane McQuade

3

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31 January 2017

Michael Behbehani

4

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31 January 2017

Joe Z. Tsien

5

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31 January 2017

Ming Xu

6

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31 January 2017

language of work or name

English

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publication date

1 April 2002

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31 January 2017

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31 January 2017

volume

30

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31 January 2017

issue

4

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31 January 2017

page(s)

416–420

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Glutamate neurotoxicity and diseases of the nervous system

31 January 2017

cites work

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7 January 2021

Oxidative stress, glutamate, and neurodegenerative disorders

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7 January 2021

The changing landscape of ischaemic brain injury mechanisms.

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7 January 2021

Emerging insights into the genesis of epilepsy

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7 January 2021

Tissue and cell type-specific expression of two human c-onc genes.

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7 January 2021

Stimulation of 3T3 cells induces transcription of the c-fos proto-oncogene

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7 January 2021

The c-Fos protein interacts with c-Jun/AP-1 to stimulate transcription of AP-1 responsive genes

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7 January 2021

c-Jun dimerizes with itself and with c-Fos, forming complexes of different DNA binding affinities

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7 January 2021

The role of the leucine zipper in the fos-jun interaction

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7 January 2021

Continuous c-fos expression precedes programmed cell death in vivo

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7 January 2021

Effects of kainic acid-induced seizures and ischemia on c-fos-like proteins in rat brain.

1 reference

7 January 2021

fos-lacZ transgenic mice: mapping sites of gene induction in the central nervous system

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7 January 2021

The mechanism of kainic acid neurotoxicity

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7 January 2021

Limbic seizure and brain damage produced by kainic acid: mechanisms and relevance to human temporal lobe epilepsy

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7 January 2021

Subregion- and cell type-restricted gene knockout in mouse brain

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7 January 2021

Absence of excitotoxicity-induced apoptosis in the hippocampus of mice lacking the Jnk3 gene

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7 January 2021

Kainic acid induces distinct types of epileptiform discharge with differential involvement of hippocampus and neocortex

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7 January 2021

Intraventricular kainic acid preferentially destroys hippocampal pyramidal cells

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7 January 2021

Absence of a persistently elevated 37 kDa fos-related antigen and AP-1-like DNA-binding activity in the brains of kainic acid-treated fosB null mice.

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7 January 2021

Cloned glutamate receptors

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7 January 2021

Potentiated necrosis of cultured cortical neurons by neurotrophins

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7 January 2021

Neuroprotective effects of brain-derived neurotrophic factor in seizures during development

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7 January 2021

Seizure-induced changes in neurotrophin expression: implications for epilepsy

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7 January 2021

Increased production of the TrkB protein tyrosine kinase receptor after brain insults

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7 January 2021

Altered synaptic physiology and reduced susceptibility to kainate-induced seizures in GluR6-deficient mice

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7 January 2021

Dopamine D1 receptor mutant mice are deficient in striatal expression of dynorphin and in dopamine-mediated behavioral responses

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7 January 2021

Identifiers

DOI

10.1038/NG859

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Consolidated OpenCitations Corpus – April 2017

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Consolidated OpenCitations Corpus – April 2017

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Consolidated OpenCitations Corpus – April 2017